erythrosine has been researched along with Status Epilepticus in 44 studies
Fluoresceins: A family of spiro(isobenzofuran-1(3H),9'-(9H)xanthen)-3-one derivatives. These are used as dyes, as indicators for various metals, and as fluorescent labels in immunoassays.
Status Epilepticus: A prolonged seizure or seizures repeated frequently enough to prevent recovery between episodes occurring over a period of 20-30 minutes. The most common subtype is generalized tonic-clonic status epilepticus, a potentially fatal condition associated with neuronal injury and respiratory and metabolic dysfunction. Nonconvulsive forms include petit mal status and complex partial status, which may manifest as behavioral disturbances. Simple partial status epilepticus consists of persistent motor, sensory, or autonomic seizures that do not impair cognition (see also EPILEPSIA PARTIALIS CONTINUA). Subclinical status epilepticus generally refers to seizures occurring in an unresponsive or comatose individual in the absence of overt signs of seizure activity. (From N Engl J Med 1998 Apr 2;338(14):970-6; Neurologia 1997 Dec;12 Suppl 6:25-30)
| Excerpt | Relevance | Reference |
|---|---|---|
| " We used kainic acid to induce status epilepticus in an animal model and examined the resultant changes in protein expression by Western blot and changes in histology by specific staining for cell death and MFS." | 7.88 | Lacosamide modulates collapsin response mediator protein 2 and inhibits mossy fiber sprouting after kainic acid-induced status epilepticus. ( Ma, R; Meng, H; Shao, N; Wang, X; Yu, Y, 2018) |
| "Experiments were designed to evaluate changes in the histamine release, mast cell number and neuronal damage in hippocampus induced by status epilepticus." | 7.81 | The mast cell stabilizer sodium cromoglycate reduces histamine release and status epilepticus-induced neuronal damage in the rat hippocampus. ( Orozco-Suárez, SA; Rocha, L; Santana-Gómez, CE; Valle-Dorado, MG, 2015) |
| " After pilocarpine-induced status epilepticus (SE), increases in neurotrophins regulate a wide variety of cell-signaling pathways, including prosurvival and cell-death machinery in a receptor-specific manner." | 7.80 | Acute administration of the small-molecule p75(NTR) ligand does not prevent hippocampal neuron loss or development of spontaneous seizures after pilocarpine-induced status epilepticus. ( Brooks-Kayal, AR; Carlsen, J; Cruz Del Angel, Y; Gonzalez, MI; Grabenstatter, HL; Hund, D; Longo, FM; Raol, YH; Russek, SJ; White, AM; Yang, T, 2014) |
| "Levetiracetam has been reported to be well tolerated and effective in status epilepticus (SE) refractory to benzodiazepine." | 7.79 | The effect of levetiracetam on status epilepticus-induced neuronal death in the rat hippocampus. ( Choi, HC; Kang, TC; Kim, JE; Kim, YI; Lee, DS; Ryu, HJ; Song, HK, 2013) |
| "Experiments were conducted to evaluate the effects of transcranial focal electrical stimulation (TFS) applied via tripolar concentric ring electrodes, alone and associated with a sub-effective dose of diazepam (DZP) on the expression of status epilepticus (SE) induced by lithium-pilocarpine (LP) and subsequent neuronal damage in the hippocampus." | 7.79 | Effects of transcranial focal electrical stimulation alone and associated with a sub-effective dose of diazepam on pilocarpine-induced status epilepticus and subsequent neuronal damage in rats. ( Besio, W; Cuellar-Herrera, M; Luna-Munguia, H; Orozco-Suárez, S; Rocha, L, 2013) |
| "The aims of this study were to characterize the spatial distribution of neurodegeneration after status epilepticus (SE) induced by either systemic (S) or intrahippocampal (H) injection of pilocarpine (PILO), two models of temporal lobe epilepsy (TLE), using FluoroJade (FJ) histochemistry, and to evaluate the kinetics of FJ staining in the H-PILO model." | 7.77 | Comparative neuroanatomical and temporal characterization of FluoroJade-positive neurodegeneration after status epilepticus induced by systemic and intrahippocampal pilocarpine in Wistar rats. ( Castro, OW; Fernandes, A; Furtado, MA; Garcia-Cairasco, N; Pajolla, GP; Tilelli, CQ, 2011) |
| " The present study evaluated the distribution pattern of GABAergic interneurons, especially parvalbumin (PV)- and somatostatin (SS)-immunopositive neurons, and excitatory propagation pattern in the IC of rats 4-7 days and 2 months after pilocarpine-induced status epilepticus (4-7 d and 2 m post-SE rats, respectively)." | 7.76 | Pilocarpine-induced status epilepticus causes acute interneuron loss and hyper-excitatory propagation in rat insular cortex. ( Chen, S; Fujita, S; Kobayashi, M; Koshikawa, N, 2010) |
| "Pilocarpine-induced status epilepticus (SE) mimics many features of temporal lobe epilepsy and is a useful model to study neural changes that result from prolonged seizure activity." | 7.74 | Extracellular matrix protein SC1/hevin in the hippocampus following pilocarpine-induced status epilepticus. ( Brown, IR; Lively, S, 2008) |
| "Evidence for increased calpain activity has been described in the hippocampus of rodent models of temporal lobe epilepsy." | 7.74 | Calpain activation is involved in early caspase-independent neurodegeneration in the hippocampus following status epilepticus. ( Araújo, IM; Bahr, BA; Brundin, P; Carreira, BP; Carvalho, CM; Gil, JM; Mohapel, P; Petersen, A; Pinheiro, PS; Soulet, D, 2008) |
| "Kainic acid-induced status epilepticus leads to structural and functional changes in inhibitory GABAA receptors in the adult rat hippocampus, but whether similar changes occur in the developing rat is not known." | 7.73 | Kainic acid-induced status epilepticus alters GABA receptor subunit mRNA and protein expression in the developing rat hippocampus. ( Holopainen, IE; Korpi, ER; Laurén, HB; Lopez-Picon, FR, 2005) |
| " Pilocarpine-induced status epilepticus (SE) was chosen as a model to generate chronic epileptic animals." | 7.73 | Septal GABAergic neurons are selectively vulnerable to pilocarpine-induced status epilepticus and chronic spontaneous seizures. ( Banuelos, C; Castañeda, MT; Colom, LV; Garrido Sanabria, ER; Hernandez, S; Perez-Cordova, MG, 2006) |
| "Diazepam-resistant SE was induced in adult mice fed with standard or ketogenic diet or in cannabinoid receptor type 1 (CB1) receptor knock-out mice." | 5.48 | Inhibition of monoacylglycerol lipase terminates diazepam-resistant status epilepticus in mice and its effects are potentiated by a ketogenic diet. ( Butler, CR; Guilmette, E; Pauletti, A; Piro, JR; Porcu, L; Rizzi, M; Salamone, A; Samad, TA; Sheehan, MJ; Terrone, G; Vezzani, A; Villa, BR, 2018) |
| " Using the kainic acid model of status epilepticus, we have studied the effects of repetitive neonatal episodes of status epilepticus on the expression of cation chloride cotransporter KCC2 in the neonatal hippocampus." | 4.84 | Developmental patterns in the regulation of chloride homeostasis and GABA(A) receptor signaling by seizures. ( Galanopoulou, AS, 2007) |
| " We used kainic acid to induce status epilepticus in an animal model and examined the resultant changes in protein expression by Western blot and changes in histology by specific staining for cell death and MFS." | 3.88 | Lacosamide modulates collapsin response mediator protein 2 and inhibits mossy fiber sprouting after kainic acid-induced status epilepticus. ( Ma, R; Meng, H; Shao, N; Wang, X; Yu, Y, 2018) |
| " In this study, we ablated the hippocampal neurogenesis by methylazoxymethanol acetate (MAM) treatment both before and after pilocarpine induced status epilepticus (SE)." | 3.85 | Reduced abnormal integration of adult-generated granule cells does not attenuate spontaneous recurrent seizures in mice. ( Feng, GF; Hu, M; Liu, JX; Liu, Y; Yuan, B; Zhu, K, 2017) |
| " Lithium chloride- and pilocarpine-induced status epilepticus (LiCl/Pilo-SE) in rodents represents a model of severe seizures that result in development of temporal lobe epilepsy (TLE)." | 3.83 | Influence of early life status epilepticus on the developmental expression profile of the GluA2 subunit of AMPA receptors. ( Druga, R; Ergang, P; Kubová, H; Mareš, P; Salaj, M; Szczurowska, E, 2016) |
| "Adult rats underwent electrically induced temporal status epilepticus, and the eyes were studied 6 h, 1, and 7 weeks later with biochemical and immunohistochemical analyses." | 3.83 | Immune response in the eye following epileptic seizures. ( Ahl, M; Ali, I; Avdic, U; Chugh, D; Ekdahl, CT; Johansson, UE; Skoug, C, 2016) |
| "Experiments were designed to evaluate changes in the histamine release, mast cell number and neuronal damage in hippocampus induced by status epilepticus." | 3.81 | The mast cell stabilizer sodium cromoglycate reduces histamine release and status epilepticus-induced neuronal damage in the rat hippocampus. ( Orozco-Suárez, SA; Rocha, L; Santana-Gómez, CE; Valle-Dorado, MG, 2015) |
| " After pilocarpine-induced status epilepticus (SE), increases in neurotrophins regulate a wide variety of cell-signaling pathways, including prosurvival and cell-death machinery in a receptor-specific manner." | 3.80 | Acute administration of the small-molecule p75(NTR) ligand does not prevent hippocampal neuron loss or development of spontaneous seizures after pilocarpine-induced status epilepticus. ( Brooks-Kayal, AR; Carlsen, J; Cruz Del Angel, Y; Gonzalez, MI; Grabenstatter, HL; Hund, D; Longo, FM; Raol, YH; Russek, SJ; White, AM; Yang, T, 2014) |
| "Levetiracetam has been reported to be well tolerated and effective in status epilepticus (SE) refractory to benzodiazepine." | 3.79 | The effect of levetiracetam on status epilepticus-induced neuronal death in the rat hippocampus. ( Choi, HC; Kang, TC; Kim, JE; Kim, YI; Lee, DS; Ryu, HJ; Song, HK, 2013) |
| "Experiments were conducted to evaluate the effects of transcranial focal electrical stimulation (TFS) applied via tripolar concentric ring electrodes, alone and associated with a sub-effective dose of diazepam (DZP) on the expression of status epilepticus (SE) induced by lithium-pilocarpine (LP) and subsequent neuronal damage in the hippocampus." | 3.79 | Effects of transcranial focal electrical stimulation alone and associated with a sub-effective dose of diazepam on pilocarpine-induced status epilepticus and subsequent neuronal damage in rats. ( Besio, W; Cuellar-Herrera, M; Luna-Munguia, H; Orozco-Suárez, S; Rocha, L, 2013) |
| "The aims of this study were to characterize the spatial distribution of neurodegeneration after status epilepticus (SE) induced by either systemic (S) or intrahippocampal (H) injection of pilocarpine (PILO), two models of temporal lobe epilepsy (TLE), using FluoroJade (FJ) histochemistry, and to evaluate the kinetics of FJ staining in the H-PILO model." | 3.77 | Comparative neuroanatomical and temporal characterization of FluoroJade-positive neurodegeneration after status epilepticus induced by systemic and intrahippocampal pilocarpine in Wistar rats. ( Castro, OW; Fernandes, A; Furtado, MA; Garcia-Cairasco, N; Pajolla, GP; Tilelli, CQ, 2011) |
| " The present study evaluated the distribution pattern of GABAergic interneurons, especially parvalbumin (PV)- and somatostatin (SS)-immunopositive neurons, and excitatory propagation pattern in the IC of rats 4-7 days and 2 months after pilocarpine-induced status epilepticus (4-7 d and 2 m post-SE rats, respectively)." | 3.76 | Pilocarpine-induced status epilepticus causes acute interneuron loss and hyper-excitatory propagation in rat insular cortex. ( Chen, S; Fujita, S; Kobayashi, M; Koshikawa, N, 2010) |
| " To better define the role of increased homocysteine in epilepsy, we analyzed the effects of homocysteine pretreatment in the pilocarpine model of status epilepticus (SE), which is used to mimic temporal lobe epilepsy (TLE) in rodents." | 3.76 | Homocysteine potentiates seizures and cell loss induced by pilocarpine treatment. ( Agnati, LF; Andreoli, N; Baldelli, E; Biagini, G; Fuxe, K; Leo, G, 2010) |
| "Pilocarpine-induced status epilepticus (SE) mimics many features of temporal lobe epilepsy and is a useful model to study neural changes that result from prolonged seizure activity." | 3.74 | Extracellular matrix protein SC1/hevin in the hippocampus following pilocarpine-induced status epilepticus. ( Brown, IR; Lively, S, 2008) |
| "Evidence for increased calpain activity has been described in the hippocampus of rodent models of temporal lobe epilepsy." | 3.74 | Calpain activation is involved in early caspase-independent neurodegeneration in the hippocampus following status epilepticus. ( Araújo, IM; Bahr, BA; Brundin, P; Carreira, BP; Carvalho, CM; Gil, JM; Mohapel, P; Petersen, A; Pinheiro, PS; Soulet, D, 2008) |
| "Lithium-pilocarpine status epilepticus was elicited in 12- (SE12) or 25-day-old (SE25) rats." | 3.73 | Changes of cortical interhemispheric responses after status epilepticus in immature rats. ( Aleksakhina, K; Druga, R; Kubová, H; Mares, P; Tsenov, G, 2005) |
| "Kainic acid-induced status epilepticus leads to structural and functional changes in inhibitory GABAA receptors in the adult rat hippocampus, but whether similar changes occur in the developing rat is not known." | 3.73 | Kainic acid-induced status epilepticus alters GABA receptor subunit mRNA and protein expression in the developing rat hippocampus. ( Holopainen, IE; Korpi, ER; Laurén, HB; Lopez-Picon, FR, 2005) |
| " Pilocarpine-induced status epilepticus (SE) was chosen as a model to generate chronic epileptic animals." | 3.73 | Septal GABAergic neurons are selectively vulnerable to pilocarpine-induced status epilepticus and chronic spontaneous seizures. ( Banuelos, C; Castañeda, MT; Colom, LV; Garrido Sanabria, ER; Hernandez, S; Perez-Cordova, MG, 2006) |
| "Diazepam-resistant SE was induced in adult mice fed with standard or ketogenic diet or in cannabinoid receptor type 1 (CB1) receptor knock-out mice." | 1.48 | Inhibition of monoacylglycerol lipase terminates diazepam-resistant status epilepticus in mice and its effects are potentiated by a ketogenic diet. ( Butler, CR; Guilmette, E; Pauletti, A; Piro, JR; Porcu, L; Rizzi, M; Salamone, A; Samad, TA; Sheehan, MJ; Terrone, G; Vezzani, A; Villa, BR, 2018) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 21 (47.73) | 29.6817 |
| 2010's | 23 (52.27) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Zhu, K | 1 |
| Yuan, B | 1 |
| Hu, M | 1 |
| Feng, GF | 1 |
| Liu, Y | 1 |
| Liu, JX | 1 |
| Terrone, G | 1 |
| Pauletti, A | 1 |
| Salamone, A | 1 |
| Rizzi, M | 1 |
| Villa, BR | 1 |
| Porcu, L | 1 |
| Sheehan, MJ | 1 |
| Guilmette, E | 1 |
| Butler, CR | 1 |
| Piro, JR | 1 |
| Samad, TA | 1 |
| Vezzani, A | 1 |
| Wang, X | 1 |
| Yu, Y | 1 |
| Ma, R | 1 |
| Shao, N | 1 |
| Meng, H | 1 |
| Lee, DS | 1 |
| Ryu, HJ | 3 |
| Kim, JE | 3 |
| Choi, HC | 1 |
| Kim, YI | 1 |
| Song, HK | 1 |
| Kang, TC | 3 |
| Besio, W | 1 |
| Cuellar-Herrera, M | 1 |
| Luna-Munguia, H | 1 |
| Orozco-Suárez, S | 1 |
| Rocha, L | 3 |
| Prager, EM | 1 |
| Aroniadou-Anderjaska, V | 2 |
| Almeida-Suhett, CP | 1 |
| Figueiredo, TH | 2 |
| Apland, JP | 1 |
| Rossetti, F | 1 |
| Olsen, CH | 1 |
| Braga, MF | 2 |
| Grabenstatter, HL | 1 |
| Carlsen, J | 1 |
| Raol, YH | 1 |
| Yang, T | 1 |
| Hund, D | 1 |
| Cruz Del Angel, Y | 1 |
| White, AM | 1 |
| Gonzalez, MI | 1 |
| Longo, FM | 1 |
| Russek, SJ | 1 |
| Brooks-Kayal, AR | 1 |
| VonDran, MW | 1 |
| LaFrancois, J | 1 |
| Padow, VA | 1 |
| Friedman, WJ | 1 |
| Scharfman, HE | 1 |
| Milner, TA | 1 |
| Hempstead, BL | 1 |
| Akman, O | 1 |
| Moshé, SL | 1 |
| Galanopoulou, AS | 2 |
| Valle-Dorado, MG | 1 |
| Santana-Gómez, CE | 2 |
| Orozco-Suárez, SA | 2 |
| Szczurowska, E | 1 |
| Ergang, P | 1 |
| Kubová, H | 5 |
| Druga, R | 4 |
| Salaj, M | 1 |
| Mareš, P | 3 |
| Ahl, M | 1 |
| Avdic, U | 1 |
| Skoug, C | 1 |
| Ali, I | 1 |
| Chugh, D | 1 |
| Johansson, UE | 1 |
| Ekdahl, CT | 2 |
| Talevi, A | 1 |
| Bruno-Blanch, L | 1 |
| Magdaleno-Madrigal, VM | 1 |
| Fernández-Mas, R | 1 |
| Unsain, N | 1 |
| Nuñez, N | 1 |
| Anastasía, A | 1 |
| Mascó, DH | 1 |
| Avignone, E | 1 |
| Ulmann, L | 1 |
| Levavasseur, F | 1 |
| Rassendren, F | 1 |
| Audinat, E | 1 |
| Lively, S | 1 |
| Brown, IR | 1 |
| Liang, LP | 1 |
| Jarrett, SG | 1 |
| Patel, M | 1 |
| Lee, B | 2 |
| Cao, R | 1 |
| Choi, YS | 2 |
| Cho, HY | 1 |
| Rhee, AD | 1 |
| Hah, CK | 1 |
| Hoyt, KR | 1 |
| Obrietan, K | 2 |
| Fritsch, B | 1 |
| Qashu, F | 1 |
| Rogawski, MA | 1 |
| Chen, S | 1 |
| Fujita, S | 1 |
| Koshikawa, N | 1 |
| Kobayashi, M | 1 |
| Baldelli, E | 1 |
| Leo, G | 1 |
| Andreoli, N | 1 |
| Fuxe, K | 1 |
| Biagini, G | 1 |
| Agnati, LF | 1 |
| Spigolon, G | 1 |
| Veronesi, C | 1 |
| Bonny, C | 1 |
| Vercelli, A | 1 |
| Castro, OW | 2 |
| Furtado, MA | 2 |
| Tilelli, CQ | 1 |
| Fernandes, A | 1 |
| Pajolla, GP | 1 |
| Garcia-Cairasco, N | 2 |
| Del Vecchio, F | 1 |
| de Oliveira, JA | 1 |
| Yeo, SI | 1 |
| Seo, CH | 1 |
| Lee, BC | 1 |
| Choi, IG | 1 |
| Kim, DS | 1 |
| Rosim, FE | 1 |
| Persike, DS | 1 |
| Nehlig, A | 2 |
| Amorim, RP | 1 |
| de Oliveira, DM | 1 |
| Fernandes, MJ | 1 |
| Serrano, GE | 1 |
| Lelutiu, N | 1 |
| Rojas, A | 1 |
| Cochi, S | 1 |
| Shaw, R | 1 |
| Makinson, CD | 1 |
| Wang, D | 1 |
| FitzGerald, GA | 1 |
| Dingledine, R | 1 |
| Choi, SY | 1 |
| do Nascimento, AL | 1 |
| Dos Santos, NF | 1 |
| Campos Pelágio, F | 1 |
| Aparecida Teixeira, S | 1 |
| de Moraes Ferrari, EA | 1 |
| Langone, F | 1 |
| Haugvicová, R | 1 |
| Suchomelová, L | 1 |
| Pitkanen, A | 4 |
| Narkilahti, S | 1 |
| Pirttilä, TJ | 1 |
| Lukasiuk, K | 2 |
| Tuunanen, J | 1 |
| Otáhal, J | 1 |
| Tsenov, G | 1 |
| Aleksakhina, K | 1 |
| Laurén, HB | 1 |
| Lopez-Picon, FR | 1 |
| Korpi, ER | 1 |
| Holopainen, IE | 1 |
| Bonde, S | 1 |
| Lindvall, O | 1 |
| Nairismägi, J | 1 |
| Kettunen, MI | 1 |
| Kauppinen, RA | 1 |
| Voutsinos-Porche, B | 1 |
| Koning, E | 1 |
| Clément, Y | 1 |
| Kaplan, H | 1 |
| Ferrandon, A | 1 |
| Motte, J | 1 |
| Garrido Sanabria, ER | 1 |
| Castañeda, MT | 1 |
| Banuelos, C | 1 |
| Perez-Cordova, MG | 1 |
| Hernandez, S | 1 |
| Colom, LV | 1 |
| Dziema, H | 1 |
| Lee, KH | 1 |
| Lahtinen, L | 1 |
| van Vliet, EA | 1 |
| da Costa Araújo, S | 1 |
| Redeker, S | 1 |
| van Schaik, R | 1 |
| Aronica, E | 1 |
| Gorter, JA | 1 |
| Matzen, J | 1 |
| Buchheim, K | 1 |
| van Landeghem, FK | 1 |
| Meierkord, H | 1 |
| Holtkamp, M | 1 |
| Araújo, IM | 1 |
| Gil, JM | 1 |
| Carreira, BP | 1 |
| Mohapel, P | 1 |
| Petersen, A | 1 |
| Pinheiro, PS | 1 |
| Soulet, D | 1 |
| Bahr, BA | 1 |
| Brundin, P | 1 |
| Carvalho, CM | 1 |
1 review available for erythrosine and Status Epilepticus
| Article | Year |
|---|---|
Developmental patterns in the regulation of chloride homeostasis and GABA(A) receptor signaling by seizures.
Topics: Animals; Animals, Newborn; Brain; Chlorides; Disease Models, Animal; Fluoresceins; Hippocampus; Home | 2007 |
43 other studies available for erythrosine and Status Epilepticus
| Article | Year |
|---|---|
Reduced abnormal integration of adult-generated granule cells does not attenuate spontaneous recurrent seizures in mice.
Topics: Animals; Bromodeoxyuridine; Cell Count; Disease Models, Animal; Doublecortin Domain Proteins; Female | 2017 |
Inhibition of monoacylglycerol lipase terminates diazepam-resistant status epilepticus in mice and its effects are potentiated by a ketogenic diet.
Topics: Animals; Brain; Brain Waves; Carbamates; Cognition Disorders; Diazepam; Disease Models, Animal; Dose | 2018 |
Lacosamide modulates collapsin response mediator protein 2 and inhibits mossy fiber sprouting after kainic acid-induced status epilepticus.
Topics: Animals; Anticonvulsants; Disease Models, Animal; Fluoresceins; Intercellular Signaling Peptides and | 2018 |
The effect of levetiracetam on status epilepticus-induced neuronal death in the rat hippocampus.
Topics: Animals; Behavior, Animal; Cell Death; Diazepam; Disease Models, Animal; Drug Therapy, Combination; | 2013 |
Effects of transcranial focal electrical stimulation alone and associated with a sub-effective dose of diazepam on pilocarpine-induced status epilepticus and subsequent neuronal damage in rats.
Topics: Analysis of Variance; Animals; Anticonvulsants; Cell Count; Deep Brain Stimulation; Diazepam; Diseas | 2013 |
The recovery of acetylcholinesterase activity and the progression of neuropathological and pathophysiological alterations in the rat basolateral amygdala after soman-induced status epilepticus: relation to anxiety-like behavior.
Topics: Acetylcholinesterase; Animals; Anxiety; Basolateral Nuclear Complex; Cholinesterase Inhibitors; Dise | 2014 |
Acute administration of the small-molecule p75(NTR) ligand does not prevent hippocampal neuron loss or development of spontaneous seizures after pilocarpine-induced status epilepticus.
Topics: Analysis of Variance; Animals; Anticonvulsants; Brain Waves; Disease Models, Animal; Electroencephal | 2014 |
p75NTR, but not proNGF, is upregulated following status epilepticus in mice.
Topics: Animals; Brain-Derived Neurotrophic Factor; Disease Models, Animal; Excitatory Amino Acid Agonists; | 2014 |
Early life status epilepticus and stress have distinct and sex-specific effects on learning, subsequent seizure outcomes, including anticonvulsant response to phenobarbital.
Topics: Age Factors; Analysis of Variance; Animals; Animals, Newborn; Anticonvulsants; Convulsants; Disease | 2015 |
The mast cell stabilizer sodium cromoglycate reduces histamine release and status epilepticus-induced neuronal damage in the rat hippocampus.
Topics: Analysis of Variance; Animals; Anti-Asthmatic Agents; Anticonvulsants; Cell Count; Chromatography, H | 2015 |
Influence of early life status epilepticus on the developmental expression profile of the GluA2 subunit of AMPA receptors.
Topics: Age Factors; Animals; Animals, Newborn; Brain; Convulsants; Disease Models, Animal; Fluoresceins; Ge | 2016 |
Immune response in the eye following epileptic seizures.
Topics: Animals; Antibodies; Antigens, CD; Calcium-Binding Proteins; Cell Death; CX3C Chemokine Receptor 1; | 2016 |
Propylparaben applied after pilocarpine-induced status epilepticus modifies hippocampal excitability and glutamate release in rats.
Topics: Action Potentials; Animals; Anticonvulsants; Cell Count; Diazepam; Disease Models, Animal; Electric | 2017 |
Status epilepticus induces a TrkB to p75 neurotrophin receptor switch and increases brain-derived neurotrophic factor interaction with p75 neurotrophin receptor: an initial event in neuronal injury induction.
Topics: Animals; Benzoxazines; Blotting, Western; Brain-Derived Neurotrophic Factor; Coloring Agents; Electr | 2008 |
Status epilepticus induces a particular microglial activation state characterized by enhanced purinergic signaling.
Topics: Adenosine Diphosphate; Adenosine Triphosphate; Animals; Cell Movement; Cell Proliferation; CX3C Chem | 2008 |
Extracellular matrix protein SC1/hevin in the hippocampus following pilocarpine-induced status epilepticus.
Topics: Animals; Calcium-Binding Proteins; Disease Models, Animal; Extracellular Matrix Proteins; Fluorescei | 2008 |
Chelation of mitochondrial iron prevents seizure-induced mitochondrial dysfunction and neuronal injury.
Topics: Analysis of Variance; Animals; Behavior, Animal; Bleomycin; Chelating Agents; Colorimetry; Disease M | 2008 |
The CREB/CRE transcriptional pathway: protection against oxidative stress-mediated neuronal cell death.
Topics: Animals; Atropine; Brain-Derived Neurotrophic Factor; Cells, Cultured; Corpus Striatum; Cyclic AMP R | 2009 |
Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis.
Topics: Amygdala; Animals; Convulsants; Down-Regulation; Epilepsy; Fluoresceins; gamma-Aminobutyric Acid; Gl | 2009 |
Pilocarpine-induced status epilepticus causes acute interneuron loss and hyper-excitatory propagation in rat insular cortex.
Topics: Animals; Biomarkers; Cell Count; Cerebral Cortex; Convulsants; Disease Models, Animal; Electric Stim | 2010 |
Homocysteine potentiates seizures and cell loss induced by pilocarpine treatment.
Topics: Amyloid beta-Peptides; Animals; Behavior, Animal; Disease Models, Animal; Epilepsy, Temporal Lobe; F | 2010 |
c-Jun N-terminal kinase signaling pathway in excitotoxic cell death following kainic acid-induced status epilepticus.
Topics: Analysis of Variance; Animals; Cell Count; Cell Death; Disease Models, Animal; Enzyme Inhibitors; Fl | 2010 |
Comparative neuroanatomical and temporal characterization of FluoroJade-positive neurodegeneration after status epilepticus induced by systemic and intrahippocampal pilocarpine in Wistar rats.
Topics: Animals; Fluoresceins; Fluorescent Dyes; Hippocampus; Male; Microinjections; Nerve Degeneration; Org | 2011 |
Study of spontaneous recurrent seizures and morphological alterations after status epilepticus induced by intrahippocampal injection of pilocarpine.
Topics: Animals; Axons; Behavior, Animal; Brain; Chi-Square Distribution; Disease Models, Animal; Fluorescei | 2011 |
The roles of fractalkine/CX3CR1 system in neuronal death following pilocarpine-induced status epilepticus.
Topics: Animals; Cell Count; Chemokine CX3CL1; CX3C Chemokine Receptor 1; Disease Models, Animal; Fluorescei | 2011 |
Differential neuroprotection by A(1) receptor activation and A(2A) receptor inhibition following pilocarpine-induced status epilepticus.
Topics: Adenosine; Analysis of Variance; Animals; Brain; Cell Count; Disease Models, Animal; Drug Interactio | 2011 |
Ablation of cyclooxygenase-2 in forebrain neurons is neuroprotective and dampens brain inflammation after status epilepticus.
Topics: Alprostadil; Animals; Blood-Testis Barrier; Cyclooxygenase 2; Cytokines; Disease Models, Animal; Ele | 2011 |
Tumor necrosis factor-α-mediated threonine 435 phosphorylation of p65 nuclear factor-κB subunit in endothelial cells induces vasogenic edema and neutrophil infiltration in the rat piriform cortex following status epilepticus.
Topics: Animals; Antigens, Surface; Brain Edema; Cell Count; Chemokine CXCL2; Disease Models, Animal; Endoth | 2012 |
Neuronal degeneration and gliosis time-course in the mouse hippocampal formation after pilocarpine-induced status epilepticus.
Topics: Analysis of Variance; Animals; Cell Death; Disease Models, Animal; Disease Progression; Fluoresceins | 2012 |
Dynamic changes of status epilepticus-induced neuronal degeneration in the mediodorsal nucleus of the thalamus during postnatal development of the rat.
Topics: Aging; Animals; Animals, Newborn; Fluoresceins; Fluorescent Dyes; Male; Mediodorsal Thalamic Nucleus | 2002 |
Expression and activation of caspase 3 following status epilepticus in the rat.
Topics: Animals; Blotting, Western; Brain; Caspase 3; Caspases; Cell Count; Cell Death; Disease Models, Anim | 2003 |
Degenerative neuronal changes in the rat thalamus induced by status epilepticus at different developmental stages.
Topics: Age Factors; Animals; Animals, Newborn; Behavior, Animal; Cell Count; Computer-Aided Design; Fluores | 2005 |
Changes of cortical interhemispheric responses after status epilepticus in immature rats.
Topics: Animals; Cerebral Cortex; Corpus Callosum; Electric Stimulation; Fluoresceins; Fluorescent Dyes; Fun | 2005 |
Kainic acid-induced status epilepticus alters GABA receptor subunit mRNA and protein expression in the developing rat hippocampus.
Topics: Aging; Animals; Animals, Newborn; Fluoresceins; Hippocampus; Immunohistochemistry; In Situ Hybridiza | 2005 |
Long-term neuronal replacement in adult rat hippocampus after status epilepticus despite chronic inflammation.
Topics: Analysis of Variance; Animals; Bromodeoxyuridine; Calcium-Binding Proteins; Cell Count; Disease Mode | 2006 |
Status epilepticus in 12-day-old rats leads to temporal lobe neurodegeneration and volume reduction: a histologic and MRI study.
Topics: Amygdala; Animals; Animals, Newborn; Apoptosis; Disease Models, Animal; Entorhinal Cortex; Fluoresce | 2006 |
EAAC1 glutamate transporter expression in the rat lithium-pilocarpine model of temporal lobe epilepsy.
Topics: Animals; Brain; Brain Chemistry; Epilepsy, Temporal Lobe; Excitatory Amino Acid Transporter 3; Fluor | 2006 |
Septal GABAergic neurons are selectively vulnerable to pilocarpine-induced status epilepticus and chronic spontaneous seizures.
Topics: Analysis of Variance; Animals; Cell Survival; Disease Models, Animal; Fluoresceins; gamma-Aminobutyr | 2006 |
CRE-mediated transcription and COX-2 expression in the pilocarpine model of status epilepticus.
Topics: Animals; Astrocytes; Blotting, Western; Cell Line; Cyclic AMP Response Element-Binding Protein; Cycl | 2007 |
Increased expression and activity of urokinase-type plasminogen activator during epileptogenesis.
Topics: Amygdala; Animals; Antigens, Surface; Blood Vessels; Disease Models, Animal; Electric Stimulation; E | 2006 |
Blood-brain barrier leakage may lead to progression of temporal lobe epilepsy.
Topics: Acute Disease; Adolescent; Adult; Albumins; Animals; Blood-Brain Barrier; Brain; Chronic Disease; Co | 2007 |
Functional and morphological changes in the dentate gyrus after experimental status epilepticus.
Topics: Animals; Chronic Disease; Data Interpretation, Statistical; Dentate Gyrus; Electric Stimulation; Ele | 2008 |
Calpain activation is involved in early caspase-independent neurodegeneration in the hippocampus following status epilepticus.
Topics: Animals; Calpain; Caspases; Convulsants; Dipeptides; Disease Models, Animal; Enzyme Activation; Enzy | 2008 |