erythrosine has been researched along with Brain Infarction in 10 studies
Fluoresceins: A family of spiro(isobenzofuran-1(3H),9'-(9H)xanthen)-3-one derivatives. These are used as dyes, as indicators for various metals, and as fluorescent labels in immunoassays.
Brain Infarction: Tissue NECROSIS in any area of the brain, including the CEREBRAL HEMISPHERES, the CEREBELLUM, and the BRAIN STEM. Brain infarction is the result of a cascade of events initiated by inadequate blood flow through the brain that is followed by HYPOXIA and HYPOGLYCEMIA in brain tissue. Damage may be temporary, permanent, selective or pan-necrosis.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The study conducted on HT22 cells exposed to glutamate or hypoxia and mice subjected to 60-min focal cerebral ischemia revealed that selenium (100 nM) pretreatment (24 h) significantly attenuated cell death induced by either glutamate toxicity or hypoxia." | 7.78 | Selenium preserves mitochondrial function, stimulates mitochondrial biogenesis, and reduces infarct volume after focal cerebral ischemia. ( Kumari, S; Li, PA; Mehta, SL; Mendelev, N, 2012) |
| "The study conducted on HT22 cells exposed to glutamate or hypoxia and mice subjected to 60-min focal cerebral ischemia revealed that selenium (100 nM) pretreatment (24 h) significantly attenuated cell death induced by either glutamate toxicity or hypoxia." | 3.78 | Selenium preserves mitochondrial function, stimulates mitochondrial biogenesis, and reduces infarct volume after focal cerebral ischemia. ( Kumari, S; Li, PA; Mehta, SL; Mendelev, N, 2012) |
| "A brief 3-min middle cerebral artery occlusion (MCAO) was employed to induce IPC in male mice 72 h before 90-min MCAO." | 1.38 | Preconditioning induces sustained neuroprotection by downregulation of adenosine 5'-monophosphate-activated protein kinase. ( Benashski, SE; Li, J; McCullough, LD; Tarabishy, S; Venna, VR, 2012) |
| "When we performed middle cerebral artery occlusion (MCAO) on adult mice, up-regulation of the receptor uncoordinated gene 5H2 (UNC5H2) but not its ligand netrin-1 was detected with RT-PCR and immunohistochemistry." | 1.35 | Netrin-1 attenuates ischemic stroke-induced apoptosis. ( Li, H; Li, WW; Wu, TW, 2008) |
| " In a separate study, brain damage 3 days after stroke was determined histologically in mice receiving no treatment, DMSO, or NGP1-01 (dosages and dosage schedule same as above)." | 1.35 | Neuroprotection in mice by NGP1-01 after transient focal brain ischemia. ( Abbruscato, TJ; Bickel, U; Geldenhuys, WJ; Hao, J; Klein, J; Mdzinarishvili, A; Van der Schyf, CJ, 2008) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 7 (70.00) | 29.6817 |
| 2010's | 3 (30.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Xu, N | 1 |
| Zhang, Y | 2 |
| Doycheva, DM | 1 |
| Ding, Y | 1 |
| Tang, J | 1 |
| Guo, H | 1 |
| Zhang, JH | 1 |
| Wu, TW | 1 |
| Li, WW | 1 |
| Li, H | 1 |
| Glendenning, ML | 1 |
| Lovekamp-Swan, T | 1 |
| Schreihofer, DA | 1 |
| Liu, F | 1 |
| Schafer, DP | 1 |
| McCullough, LD | 2 |
| Venna, VR | 1 |
| Li, J | 1 |
| Benashski, SE | 1 |
| Tarabishy, S | 1 |
| Mehta, SL | 1 |
| Kumari, S | 1 |
| Mendelev, N | 1 |
| Li, PA | 1 |
| Kharlamov, EA | 1 |
| Kharlamov, A | 1 |
| Kelly, KM | 1 |
| Popa-Wagner, A | 1 |
| Badan, I | 1 |
| Walker, L | 1 |
| Groppa, S | 1 |
| Patrana, N | 1 |
| Kessler, C | 1 |
| Pulgar, VM | 1 |
| Zhang, J | 1 |
| Massmann, GA | 1 |
| Figueroa, JP | 1 |
| Hao, J | 1 |
| Mdzinarishvili, A | 1 |
| Abbruscato, TJ | 1 |
| Klein, J | 1 |
| Geldenhuys, WJ | 1 |
| Van der Schyf, CJ | 1 |
| Bickel, U | 1 |
10 other studies available for erythrosine and Brain Infarction
| Article | Year |
|---|---|
Adiponectin attenuates neuronal apoptosis induced by hypoxia-ischemia via the activation of AdipoR1/APPL1/LKB1/AMPK pathway in neonatal rats.
Topics: Adaptor Proteins, Signal Transducing; Adiponectin; AMP-Activated Protein Kinase Kinases; Animals; An | 2018 |
Netrin-1 attenuates ischemic stroke-induced apoptosis.
Topics: Animals; Apoptosis; Brain Infarction; Cell Movement; Cells, Cultured; Cerebral Cortex; DCC Receptor; | 2008 |
Protective effect of estrogen in endothelin-induced middle cerebral artery occlusion in female rats.
Topics: Animals; Behavior, Animal; Brain Infarction; Disease Models, Animal; Endothelin-1; Estrogens; Female | 2008 |
TTC, fluoro-Jade B and NeuN staining confirm evolving phases of infarction induced by middle cerebral artery occlusion.
Topics: Analysis of Variance; Animals; Brain; Brain Chemistry; Brain Infarction; DNA-Binding Proteins; Fluor | 2009 |
Preconditioning induces sustained neuroprotection by downregulation of adenosine 5'-monophosphate-activated protein kinase.
Topics: Animals; Brain; Brain Infarction; Disease Models, Animal; Down-Regulation; Fluoresceins; HSP72 Heat- | 2012 |
Selenium preserves mitochondrial function, stimulates mitochondrial biogenesis, and reduces infarct volume after focal cerebral ischemia.
Topics: Analysis of Variance; Animals; Antioxidants; Apoptosis Regulatory Proteins; Autophagy; Beclin-1; Bra | 2012 |
Changes in neuropeptide Y protein expression following photothrombotic brain infarction and epileptogenesis.
Topics: Animals; Astrocytes; Brain; Brain Infarction; Dentate Gyrus; Disease Models, Animal; DNA-Binding Pro | 2007 |
Accelerated infarct development, cytogenesis and apoptosis following transient cerebral ischemia in aged rats.
Topics: Aging; Analysis of Variance; Animals; Apoptosis; Blood Vessels; Brain Infarction; Bromodeoxyuridine; | 2007 |
Mild chronic hypoxia modifies the fetal sheep neural and cardiovascular responses to repeated umbilical cord occlusion.
Topics: Alpha Rhythm; Animals; Brain Infarction; Cardiovascular Physiological Phenomena; Cell Death; Chronic | 2007 |
Neuroprotection in mice by NGP1-01 after transient focal brain ischemia.
Topics: Analysis of Variance; Animals; Brain Infarction; Brain Ischemia; Bridged-Ring Compounds; Cerebrovasc | 2008 |