erythrosine has been researched along with Allergic Encephalomyelitis in 8 studies
Fluoresceins: A family of spiro(isobenzofuran-1(3H),9'-(9H)xanthen)-3-one derivatives. These are used as dyes, as indicators for various metals, and as fluorescent labels in immunoassays.
Excerpt | Relevance | Reference |
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"The transport of the hydrophilic model compound sodium fluorescein into the cerebrospinal fluid (CSF) of rats was studied during experimental allergic encephalomyelitis (EAE), as a model for local central nervous system (CNS) inflammatory disease, and after a single injection of a pyrogenic dose of lipopolysaccharide (LPS), as a model for a general inflammation." | 3.69 | Transport of a hydrophilic compound into the cerebrospinal fluid during experimental allergic encephalomyelitis and after lipopolysaccharide administration. ( Breimer, DD; de Boer, AG; de Vries, HE; Eppens, EF; Kuiper, J; Prins, M; van Berkel, TJ, 1995) |
"Using experimental allergic encephalomyelitis (EAE) as a model for MS, Apo A-I deficient mice exhibited worse clinical disease and more neurodegeneration concurrent with increased levels of pro-inflammatory cytokines compared to wild-type animals." | 1.40 | A role for Apolipoprotein A-I in the pathogenesis of multiple sclerosis. ( Brand, D; Douglas, J; Gardner, LA; Groover, CJ; Lee, S; Levin, MC; Meyers, L, 2014) |
"Murine models of experimental autoimmune encephalomyelitis (EAE) are important vehicles for studying the effects of genetic manipulation on disease processes related to multiple sclerosis (MS)." | 1.34 | Time course and distribution of inflammatory and neurodegenerative events suggest structural bases for the pathogenesis of experimental autoimmune encephalomyelitis. ( Brown, DA; Sawchenko, PE, 2007) |
"Amelioration of experimental autoimmune encephalomyelitis was associated with a selective inhibition of proliferation response and cytokine production by Ag-stimulated lymph node T cells and a drastic reduction in the number of encephalitogenic and recruited inflammatory cells infiltrating the CNS." | 1.31 | Regulation of encephalitogenic T cells with recombinant TCR ligands. ( Adlard, KL; Bebo, BF; Burrows, GG; Chang, JW; Offner, H; Tenditnyy, K; Vandenbark, AA, 2000) |
"In a study of experimental autoimmune encephalomyelitis (EAE) in rabbits, a freeze-dried, paraffin-embedded tissue technique was exploited to enable (1) the immobilization of intravenously injected sodium fluorescein tracer, as an index of vascular permeability; and (2) an effective labeling by monoclonal antibodies of both T-lymphocytes and mononuclear phagocytes in "unfixed" neural tissue." | 1.27 | Simultaneous visualization of vascular permeability change and leukocyte egress in the central nervous system during autoimmune encephalomyelitis. ( Buzbee, TM; Chen, G; Linthicum, DS; Mandy, WJ; Simmons, RD; Wang, C, 1987) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 1 (12.50) | 18.7374 |
1990's | 2 (25.00) | 18.2507 |
2000's | 2 (25.00) | 29.6817 |
2010's | 3 (37.50) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
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Meyers, L | 1 |
Groover, CJ | 1 |
Douglas, J | 1 |
Lee, S | 1 |
Brand, D | 1 |
Levin, MC | 1 |
Gardner, LA | 1 |
Moriguchi, K | 1 |
Miyamoto, K | 1 |
Tanaka, N | 1 |
Ueno, R | 1 |
Nakayama, T | 1 |
Yoshie, O | 1 |
Kusunoki, S | 1 |
Zhao, Z | 1 |
Ciric, B | 1 |
Yu, S | 1 |
Li, H | 1 |
Yang, J | 1 |
Kamoun, M | 1 |
Zhang, GX | 1 |
Rostami, A | 1 |
Brown, DA | 1 |
Sawchenko, PE | 1 |
Gold, R | 1 |
Schmied, M | 1 |
Rothe, G | 1 |
Zischler, H | 1 |
Breitschopf, H | 1 |
Wekerle, H | 1 |
Lassmann, H | 1 |
de Vries, HE | 1 |
Eppens, EF | 1 |
Prins, M | 1 |
Kuiper, J | 1 |
van Berkel, TJ | 1 |
de Boer, AG | 1 |
Breimer, DD | 1 |
Burrows, GG | 1 |
Adlard, KL | 1 |
Bebo, BF | 1 |
Chang, JW | 1 |
Tenditnyy, K | 1 |
Vandenbark, AA | 1 |
Offner, H | 1 |
Simmons, RD | 1 |
Buzbee, TM | 1 |
Linthicum, DS | 1 |
Mandy, WJ | 1 |
Chen, G | 1 |
Wang, C | 1 |
8 other studies available for erythrosine and Allergic Encephalomyelitis
Article | Year |
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A role for Apolipoprotein A-I in the pathogenesis of multiple sclerosis.
Topics: Adult; Animals; Apolipoprotein A-I; Case-Control Studies; Cytokines; Disease Models, Animal; Electro | 2014 |
C-C chemokine receptor type 4 antagonist Compound 22 ameliorates experimental autoimmune encephalomyelitis.
Topics: Animals; Cell Proliferation; Cytokines; Dose-Response Relationship, Drug; Encephalomyelitis, Autoimm | 2016 |
Expression of 3G11 epitope defines subpopulations of regulatory T cells with different suppressive potency.
Topics: Animals; Antigens, CD; Antigens, Surface; Cell Proliferation; Coculture Techniques; Cytokines; Disea | 2010 |
Time course and distribution of inflammatory and neurodegenerative events suggest structural bases for the pathogenesis of experimental autoimmune encephalomyelitis.
Topics: Animals; Brain; Encephalomyelitis, Autoimmune, Experimental; Female; Fluoresceins; Freund's Adjuvant | 2007 |
Detection of DNA fragmentation in apoptosis: application of in situ nick translation to cell culture systems and tissue sections.
Topics: Animals; Apoptosis; Azides; Cell Division; Cells, Cultured; Deoxyuracil Nucleotides; Digoxigenin; DN | 1993 |
Transport of a hydrophilic compound into the cerebrospinal fluid during experimental allergic encephalomyelitis and after lipopolysaccharide administration.
Topics: Animals; Cerebrospinal Fluid; Encephalomyelitis, Autoimmune, Experimental; Fluorescein; Fluoresceins | 1995 |
Regulation of encephalitogenic T cells with recombinant TCR ligands.
Topics: Adoptive Transfer; Amino Acid Sequence; Animals; Cell Line; Encephalomyelitis, Autoimmune, Experimen | 2000 |
Simultaneous visualization of vascular permeability change and leukocyte egress in the central nervous system during autoimmune encephalomyelitis.
Topics: Animals; Autoimmune Diseases; Blood Vessels; Capillary Permeability; Cell Movement; Encephalomyeliti | 1987 |