Page last updated: 2024-09-05

erlotinib hydrochloride and Hyperplasia

erlotinib hydrochloride has been researched along with Hyperplasia in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's4 (80.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Comstock, AT; Gimenes, JA; Jing, Y; Mishra, R; Pham, D; Sajjan, U; Yu, D1
Arcidiacono, MV; Dusso, A; Fernandez, E; Yang, J1
Bhagathavula, N; Dame, MK; Dawes, SM; Elder, JT; Fu, W; Gudjonsson, JE; Johnston, A; Lambert, S; Varani, J; Ward, NL; Warner, RL1
Chung, WC; Gold, KA; Hittelman, WN; Kang, SM; Ki Hong, W; Kim, ES; Koo, JS; Lee, J; Ryu, SH1
Alvarez-Hernandez, D; Arcidiacono, MV; Cannata-Andia, J; Dusso, AS; Gonzalez-Suarez, I; Lu, Y; Sato, T; Slatopolsky, E; Tokumoto, M; Tominaga, Y; Yang, J1

Other Studies

5 other study(ies) available for erlotinib hydrochloride and Hyperplasia

ArticleYear
NOTCH3 contributes to rhinovirus-induced goblet cell hyperplasia in COPD airway epithelial cells.
    Thorax, 2019, Volume: 74, Issue:1

    Topics: Actins; Amyloid Precursor Protein Secretases; Animals; Antibodies, Neutralizing; Basic Helix-Loop-Helix Transcription Factors; Cell Cycle Proteins; Cells, Cultured; Disease Models, Animal; ErbB Receptors; Erlotinib Hydrochloride; Female; Gene Expression; Gene Silencing; Goblet Cells; Hepatocyte Nuclear Factor 3-gamma; Humans; Hyperplasia; Interleukin-13; Mice; Mucin 5AC; Mucin-5B; Pulmonary Disease, Chronic Obstructive; Receptor, Notch1; Receptor, Notch3; Respiratory Mucosa; Rhinovirus; RNA, Messenger; Signal Transduction

2019
The induction of C/EBPβ contributes to vitamin D inhibition of ADAM17 expression and parathyroid hyperplasia in kidney disease.
    Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association, 2015, Volume: 30, Issue:3

    Topics: ADAM Proteins; ADAM17 Protein; Animals; Antineoplastic Agents; Apoptosis; Blotting, Western; Calcitriol; CCAAT-Enhancer-Binding Protein-beta; Cell Proliferation; Cells, Cultured; ErbB Receptors; Erlotinib Hydrochloride; Gene Expression Regulation; Humans; Hyperparathyroidism, Secondary; Hyperplasia; Immunoenzyme Techniques; Kidney Diseases; Parathyroid Glands; Parathyroid Hormone; Rats; Real-Time Polymerase Chain Reaction; Receptors, Calcitriol; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Transforming Growth Factor alpha; Vitamin D; Vitamins

2015
Erlotinib-induced skin inflammation is IL-1 mediated in KC-Tie2 mice and human skin organ culture.
    The Journal of investigative dermatology, 2015, Volume: 135, Issue:3

    Topics: Animals; Biopsy; Dermatitis; Disease Models, Animal; Dose-Response Relationship, Drug; Erlotinib Hydrochloride; Humans; Hyperplasia; In Vitro Techniques; Interleukin-1; Mice; Mice, Transgenic; Organ Culture Techniques; Phenotype; Quinazolines; Receptor, TIE-2; Skin

2015
Prevention of bronchial hyperplasia by EGFR pathway inhibitors in an organotypic culture model.
    Cancer prevention research (Philadelphia, Pa.), 2011, Volume: 4, Issue:8

    Topics: Bronchi; Butadienes; Cell Line, Tumor; Cyclin D1; Enzyme Inhibitors; ErbB Receptors; Erlotinib Hydrochloride; Humans; Hyperplasia; Luciferases; Lung Neoplasms; MAP Kinase Kinase Kinases; Models, Biological; Nitriles; Organ Culture Techniques; Quinazolines; RNA, Small Interfering

2011
EGFR activation increases parathyroid hyperplasia and calcitriol resistance in kidney disease.
    Journal of the American Society of Nephrology : JASN, 2008, Volume: 19, Issue:2

    Topics: Animals; Calcitriol; Carcinoma, Squamous Cell; CCAAT-Enhancer-Binding Protein-beta; Cell Line, Tumor; Disease Models, Animal; Drug Resistance; ErbB Receptors; Erlotinib Hydrochloride; Female; Genes, Reporter; Humans; Hyperparathyroidism, Secondary; Hyperplasia; Protein Kinase Inhibitors; Quinazolines; Rats; Rats, Sprague-Dawley; Receptors, Calcitriol; Renal Insufficiency, Chronic; Transforming Growth Factor alpha

2008