erlotinib hydrochloride has been researched along with Diabetic Nephropathies in 3 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 3 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chung, S; Fan, X; Harris, RC; Li, Y; Li, Z; Niu, A; Overstreet, JM; Wang, S; Wang, Y; Zhang, MZ | 1 |
| Harris, RC; Paueksakon, P; Wang, Y; Zhang, MZ | 1 |
| Altintas, MM; Baltimore, D; Bitzer, M; Cimbaluk, DJ; Faridi, MH; Geraghty, T; Grahammer, F; Gupta, V; Huber, TB; Khaliqdina, S; Khan, SQ; Koh, KH; Kretzler, M; Lee, HW; Moita, LF; Reiser, J; Susztak, K; Tardi, NJ; Tharaux, PL; Zhao, JL | 1 |
3 other study(ies) available for erlotinib hydrochloride and Diabetic Nephropathies
| Article | Year |
|---|---|
Inhibition of Epidermal Growth Factor Receptor Activation Is Associated With Improved Diabetic Nephropathy and Insulin Resistance in Type 2 Diabetes.
Topics: Albuminuria; Animals; Biomarkers; Crosses, Genetic; Cytokines; Diabetes Mellitus, Type 2; Diabetic Nephropathies; ErbB Receptors; Erlotinib Hydrochloride; Fibrosis; Glomerulonephritis; Hypoglycemic Agents; Insulin Resistance; Kidney; Macrophages; Membrane Transport Modulators; Mice, Knockout; Mice, Mutant Strains; Nitric Oxide Synthase Type III; Oxidative Stress; Protein Kinase Inhibitors; T-Lymphocytes; Transforming Growth Factor alpha | 2018 |
Epidermal growth factor receptor inhibition slows progression of diabetic nephropathy in association with a decrease in endoplasmic reticulum stress and an increase in autophagy.
Topics: Albuminuria; AMP-Activated Protein Kinases; Animals; Apoptosis; Autophagy; Blood Glucose; Blood Pressure; Cells, Cultured; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Disease Progression; Endoplasmic Reticulum Stress; ErbB Receptors; Erlotinib Hydrochloride; Immunohistochemistry; Kidney; Mice; Mice, Inbred NOD; Phosphorylation; Protein Kinase Inhibitors; Quinazolines; Signal Transduction | 2014 |
Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.
Topics: Animals; Chemokine CCL2; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Erlotinib Hydrochloride; Mice; Mice, Knockout; MicroRNAs; Podocytes; Receptor, ErbB-4; Receptor, Notch1; Ribonucleases; Risk Factors; Signal Transduction; Transforming Growth Factor beta1; Up-Regulation | 2017 |