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erlotinib hydrochloride and Diabetic Glomerulosclerosis

erlotinib hydrochloride has been researched along with Diabetic Glomerulosclerosis in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chung, S; Fan, X; Harris, RC; Li, Y; Li, Z; Niu, A; Overstreet, JM; Wang, S; Wang, Y; Zhang, MZ1
Harris, RC; Paueksakon, P; Wang, Y; Zhang, MZ1
Altintas, MM; Baltimore, D; Bitzer, M; Cimbaluk, DJ; Faridi, MH; Geraghty, T; Grahammer, F; Gupta, V; Huber, TB; Khaliqdina, S; Khan, SQ; Koh, KH; Kretzler, M; Lee, HW; Moita, LF; Reiser, J; Susztak, K; Tardi, NJ; Tharaux, PL; Zhao, JL1

Other Studies

3 other study(ies) available for erlotinib hydrochloride and Diabetic Glomerulosclerosis

ArticleYear
Inhibition of Epidermal Growth Factor Receptor Activation Is Associated With Improved Diabetic Nephropathy and Insulin Resistance in Type 2 Diabetes.
    Diabetes, 2018, Volume: 67, Issue:9

    Topics: Albuminuria; Animals; Biomarkers; Crosses, Genetic; Cytokines; Diabetes Mellitus, Type 2; Diabetic Nephropathies; ErbB Receptors; Erlotinib Hydrochloride; Fibrosis; Glomerulonephritis; Hypoglycemic Agents; Insulin Resistance; Kidney; Macrophages; Membrane Transport Modulators; Mice, Knockout; Mice, Mutant Strains; Nitric Oxide Synthase Type III; Oxidative Stress; Protein Kinase Inhibitors; T-Lymphocytes; Transforming Growth Factor alpha

2018
Epidermal growth factor receptor inhibition slows progression of diabetic nephropathy in association with a decrease in endoplasmic reticulum stress and an increase in autophagy.
    Diabetes, 2014, Volume: 63, Issue:6

    Topics: Albuminuria; AMP-Activated Protein Kinases; Animals; Apoptosis; Autophagy; Blood Glucose; Blood Pressure; Cells, Cultured; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Disease Progression; Endoplasmic Reticulum Stress; ErbB Receptors; Erlotinib Hydrochloride; Immunohistochemistry; Kidney; Mice; Mice, Inbred NOD; Phosphorylation; Protein Kinase Inhibitors; Quinazolines; Signal Transduction

2014
Absence of miR-146a in Podocytes Increases Risk of Diabetic Glomerulopathy via Up-regulation of ErbB4 and Notch-1.
    The Journal of biological chemistry, 2017, Jan-13, Volume: 292, Issue:2

    Topics: Animals; Chemokine CCL2; Diabetes Mellitus, Experimental; Diabetic Nephropathies; Erlotinib Hydrochloride; Mice; Mice, Knockout; MicroRNAs; Podocytes; Receptor, ErbB-4; Receptor, Notch1; Ribonucleases; Risk Factors; Signal Transduction; Transforming Growth Factor beta1; Up-Regulation

2017