equol has been researched along with Stomach Neoplasms in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 4 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chang, SH; Cho, LY; Choi, BY; Han, DS; Kang, D; Kim, HJ; Kim, YS; Ko, KP; Ma, SH; Park, SK; Shin, A; Shin, HR; Song, KS; Yang, JJ; Yoo, KY | 1 |
| Chen, J; Huang, F; Li, J; Wu, XN; Yang, ZP; Yao, YH; Zhao, Y | 1 |
| Li, J; Wang, W; Wu, X; Yang, Z; Yao, Y; Zhao, Y | 1 |
| Chang, SH; Cho, LY; Choi, BY; Han, DS; Kang, D; Kim, YS; Ko, KP; Ma, SH; Park, SK; Shin, A; Shin, HR; Song, KS; Yang, JJ; Yoo, KY | 1 |
1 review(s) available for equol and Stomach Neoplasms
| Article | Year |
|---|---|
Gene polymorphisms in the ornithine decarboxylase-polyamine pathway modify gastric cancer risk by interaction with isoflavone concentrations.
Topics: 4-Butyrolactone; Adenosylmethionine Decarboxylase; Asian People; Case-Control Studies; Equol; Gene-Environment Interaction; Genistein; Humans; Isoflavones; Lignans; Multicenter Studies as Topic; NAD(P)H Dehydrogenase (Quinone); Nitric Oxide Synthase Type II; Ornithine Decarboxylase; Phytoestrogens; Polyamines; Polymorphism, Single Nucleotide; Stomach Neoplasms | 2015 |
3 other study(ies) available for equol and Stomach Neoplasms
| Article | Year |
|---|---|
Equol inhibits proliferation of human gastric carcinoma cells via modulating Akt pathway.
Topics: Antineoplastic Agents, Phytogenic; Apoptosis; Carcinoma; Caspase 3; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Proliferation; Cell Survival; Cyclin E; Cyclin-Dependent Kinase Inhibitor p21; Dose-Response Relationship, Drug; Enzyme Activation; Equol; Gene Expression Regulation, Neoplastic; Humans; Ki-67 Antigen; Oncogene Proteins; Phosphorylation; Poly(ADP-ribose) Polymerases; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Signal Transduction; Stomach Neoplasms; Time Factors | 2015 |
Equol Induces Mitochondria-Dependent Apoptosis in Human Gastric Cancer Cells via the Sustained Activation of ERK1/2 Pathway.
Topics: Apoptosis; Cell Line, Tumor; Enzyme Activation; Equol; Humans; MAP Kinase Signaling System; Mitochondria; Phytoestrogens; Stomach Neoplasms | 2016 |
Interaction effects between genes involved in the AKT signaling pathway and phytoestrogens in gastric carcinogenesis: a nested case-control study from the Korean Multi-Center Cancer Cohort.
Topics: 4-Butyrolactone; Aged; Anticarcinogenic Agents; Asian People; Case-Control Studies; CDC2 Protein Kinase; Equol; fas Receptor; Female; Gene-Environment Interaction; Genetic Predisposition to Disease; Genistein; Humans; Isoflavones; Lignans; Male; MAP Kinase Kinase Kinase 1; Middle Aged; Odds Ratio; Phytoestrogens; Polymorphism, Single Nucleotide; Proto-Oncogene Proteins c-akt; Republic of Korea; Signal Transduction; Stomach Neoplasms | 2012 |