epigallocatechin gallate has been researched along with Granulocytic Leukemia, Chronic in 7 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (28.57) | 29.6817 |
| 2010's | 5 (71.43) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Baksiene, S; Borutinskaite, V; Navakauskiene, R; Vitkeviciene, A | 1 |
| Jiang, K; Liu, S; Peng, H; Wang, Z; Xiao, X; Xu, Y; Zhang, G | 1 |
| Adams, E; Chen, H; Van Schepdael, A | 1 |
| Choo, EJ; Jeong, MS; Jung, DB; Jung, JH; Kaihatsu, K; Kato, N; Kim, B; Kim, EO; Kim, SH; Lee, H; Srivastava, SK; Yun, M | 1 |
| Bae, J; Hidaka, S; Huang, Y; Kim, Y; Kumazoe, M; Murata, M; Tachibana, H; Takai, M; Tsukamoto, S; Won, Y; Yamada, S; Yamashita, S | 1 |
| Adachi, S; Hamanoue, M; Ishida, T; Ito, K; Iwasaki, R; Sato, Y; Watanabe, T | 1 |
| Fujimura, Y; Tachibana, H; Umeda, D; Yamada, K | 1 |
1 review(s) available for epigallocatechin gallate and Granulocytic Leukemia, Chronic
| Article | Year |
|---|---|
(-)-Epigallocatechin-3-gallate induces cell apoptosis in chronic myeloid leukaemia by regulating Bcr/Abl-mediated p38-MAPK/JNK and JAK2/STAT3/AKT signalling pathways.
Topics: Animals; Apoptosis; Catechin; Humans; Janus Kinase 2; JNK Mitogen-Activated Protein Kinases; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; p38 Mitogen-Activated Protein Kinases; Proto-Oncogene Proteins c-abl; Proto-Oncogene Proteins c-akt; Proto-Oncogene Proteins c-bcr; Signal Transduction; STAT3 Transcription Factor | 2019 |
6 other study(ies) available for epigallocatechin gallate and Granulocytic Leukemia, Chronic
| Article | Year |
|---|---|
Epigallocatechin-3-gallate and BIX-01294 have different impact on epigenetics and senescence modulation in acute and chronic myeloid leukemia cells.
Topics: Antineoplastic Agents; Azepines; Catechin; Cellular Senescence; Chromobox Protein Homolog 5; Drug Screening Assays, Antitumor; Epigenesis, Genetic; G1 Phase Cell Cycle Checkpoints; Gene Expression Regulation, Neoplastic; Humans; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Leukemia, Promyelocytic, Acute; Quinazolines | 2018 |
Study of Abl1 tyrosine kinase inhibitors by liquid chromatography-electrospray ionization-mass spectrometry.
Topics: Amino Acid Sequence; Benzamides; Biological Products; Caffeic Acids; Catechin; Chromatography, Liquid; Drug Discovery; Fusion Proteins, bcr-abl; Humans; Imatinib Mesylate; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Limit of Detection; Peptides; Piperazines; Protein Kinase Inhibitors; Pyrimidines; Spectrometry, Mass, Electrospray Ionization | 2013 |
A derivative of epigallocatechin-3-gallate induces apoptosis via SHP-1-mediated suppression of BCR-ABL and STAT3 signalling in chronic myelogenous leukaemia.
Topics: Animals; Antineoplastic Agents; Apoptosis; Catechin; Cell Cycle; Dose-Response Relationship, Drug; Drug Screening Assays, Antitumor; Fusion Proteins, bcr-abl; Humans; K562 Cells; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Male; Mice; Mice, Inbred BALB C; Mice, Nude; Molecular Structure; Neoplasms, Experimental; Protein Tyrosine Phosphatase, Non-Receptor Type 6; Signal Transduction; STAT3 Transcription Factor; Structure-Activity Relationship; Tumor Cells, Cultured | 2015 |
Green tea polyphenol epigallocatechin-O-gallate induces cell death by acid sphingomyelinase activation in chronic myeloid leukemia cells.
Topics: Anticarcinogenic Agents; Blotting, Western; Catechin; Cell Line, Tumor; Enzyme Activation; Humans; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Membrane Microdomains; Sphingomyelin Phosphodiesterase; Tea | 2015 |
Catechin, green tea component, causes caspase-independent necrosis-like cell death in chronic myelogenous leukemia.
Topics: Anticarcinogenic Agents; Apoptosis; Blotting, Western; Caspases; Catechin; Cell Line, Tumor; Cell Proliferation; Humans; Immunoenzyme Techniques; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Membrane Potential, Mitochondrial; Necrosis; Subcellular Fractions; Tea | 2009 |
The impact of the 67kDa laminin receptor on both cell-surface binding and anti-allergic action of tea catechins.
Topics: Anti-Allergic Agents; Basophils; Catechin; Cell Line, Tumor; Glycosylation; Histamine Release; Humans; Leukemia, Myelogenous, Chronic, BCR-ABL Positive; Molecular Structure; Receptors, Laminin; Structure-Activity Relationship; Tea | 2008 |