Page last updated: 2024-08-24

epigallocatechin gallate and EBV Infections

epigallocatechin gallate has been researched along with EBV Infections in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (33.33)24.3611
2020's2 (66.67)2.80

Authors

AuthorsStudies
Jha, HC; Mishra, A; Pandey, RK; Patra, P; Prashar, T; Rani, A; Saini, V1
Bode, AM; Cao, Y; Dong, Z; Hu, J; Li, H; Li, Y; Liao, W; Liu, S; Liu, X; Luo, X; Tang, M1
Bode, AM; Cao, Y; Chen, L; Dong, Z; Li, H; Li, L; Li, W; Li, Z; Liu, H; Liu, S; Tang, M; Tao, Y; Yang, L1

Reviews

1 review(s) available for epigallocatechin gallate and EBV Infections

ArticleYear
Epigallocatechin Gallate: A Multifaceted Molecule for Neurological Disorders and Neurotropic Viral Infections.
    ACS chemical neuroscience, 2023, 09-06, Volume: 14, Issue:17

    Topics: Alzheimer Disease; Epstein-Barr Virus Infections; Glycogen Synthase Kinase 3; Herpesvirus 4, Human; Humans; Nervous System Diseases; Phosphatidylinositol 3-Kinases; Zika Virus; Zika Virus Infection

2023

Other Studies

2 other study(ies) available for epigallocatechin gallate and EBV Infections

ArticleYear
()-Epigallocatechin-3-Gallate Inhibits EBV Lytic Replication via Targeting LMP1-Mediated MAPK Signal Axes.
    Oncology research, 2021, Sep-07, Volume: 28, Issue:7

    Topics: Antiviral Agents; Catechin; Cell Line, Tumor; Epstein-Barr Virus Infections; Herpesvirus 4, Human; Humans; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Mutation; Phosphorylation; Promoter Regions, Genetic; Tumor Suppressor Protein p53; Viral Matrix Proteins; Virus Replication

2021
(-)-Epigallocatechin-3-gallate inhibition of Epstein-Barr virus spontaneous lytic infection involves ERK1/2 and PI3-K/Akt signaling in EBV-positive cells.
    Carcinogenesis, 2013, Volume: 34, Issue:3

    Topics: Antiviral Agents; Catechin; Cell Line, Tumor; Cell Survival; Chromones; DNA, Viral; Enzyme Activation; Epstein-Barr Virus Infections; Flavonoids; Herpesvirus 4, Human; Humans; Lymphoma; MAP Kinase Signaling System; Mitogen-Activated Protein Kinases; Morpholines; Nasopharyngeal Neoplasms; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Phosphorylation; Protein Processing, Post-Translational; Proto-Oncogene Proteins c-akt; Viral Proteins; Virus Activation

2013