entinostat has been researched along with Stomach Neoplasms in 2 studies
Stomach Neoplasms: Tumors or cancer of the STOMACH.
| Excerpt | Relevance | Reference |
|---|---|---|
| "The effects of entinostat on gastric cancer cell lines were measured by the CCK-8 assay, clone formation assay, wound healing assay and transwell assay." | 1.91 | Targeting SALL4 by Entinostat Inhibits the Malignant Phenotype of Gastric Cancer Cells by Reducing EMT Signaling. ( DU, L; Han, H; Xie, F; Zhang, L, 2023) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (50.00) | 24.3611 |
| 2020's | 1 (50.00) | 2.80 |
| Authors | Studies |
|---|---|
| DU, L | 1 |
| Xie, F | 1 |
| Han, H | 1 |
| Zhang, L | 1 |
| Alvarez, MJ | 1 |
| Subramaniam, PS | 1 |
| Tang, LH | 1 |
| Grunn, A | 1 |
| Aburi, M | 1 |
| Rieckhof, G | 1 |
| Komissarova, EV | 1 |
| Hagan, EA | 1 |
| Bodei, L | 1 |
| Clemons, PA | 1 |
| Dela Cruz, FS | 1 |
| Dhall, D | 1 |
| Diolaiti, D | 1 |
| Fraker, DA | 1 |
| Ghavami, A | 1 |
| Kaemmerer, D | 1 |
| Karan, C | 1 |
| Kidd, M | 1 |
| Kim, KM | 1 |
| Kim, HC | 1 |
| Kunju, LP | 1 |
| Langel, Ü | 1 |
| Li, Z | 1 |
| Lee, J | 1 |
| Li, H | 1 |
| LiVolsi, V | 1 |
| Pfragner, R | 1 |
| Rainey, AR | 1 |
| Realubit, RB | 1 |
| Remotti, H | 1 |
| Regberg, J | 1 |
| Roses, R | 1 |
| Rustgi, A | 1 |
| Sepulveda, AR | 1 |
| Serra, S | 1 |
| Shi, C | 1 |
| Yuan, X | 1 |
| Barberis, M | 1 |
| Bergamaschi, R | 1 |
| Chinnaiyan, AM | 1 |
| Detre, T | 1 |
| Ezzat, S | 1 |
| Frilling, A | 1 |
| Hommann, M | 1 |
| Jaeger, D | 1 |
| Kim, MK | 1 |
| Knudsen, BS | 1 |
| Kung, AL | 1 |
| Leahy, E | 1 |
| Metz, DC | 1 |
| Milsom, JW | 1 |
| Park, YS | 1 |
| Reidy-Lagunes, D | 1 |
| Schreiber, S | 1 |
| Washington, K | 1 |
| Wiedenmann, B | 1 |
| Modlin, I | 1 |
| Califano, A | 1 |
2 other studies available for entinostat and Stomach Neoplasms
| Article | Year |
|---|---|
Targeting SALL4 by Entinostat Inhibits the Malignant Phenotype of Gastric Cancer Cells by Reducing EMT Signaling.
Topics: Cell Line, Tumor; Cell Movement; Cell Proliferation; Epithelial-Mesenchymal Transition; Gene Express | 2023 |
A precision oncology approach to the pharmacological targeting of mechanistic dependencies in neuroendocrine tumors.
Topics: Antineoplastic Agents; Benzamides; Cell Line, Tumor; Cohort Studies; Gastrointestinal Tract; Histone | 2018 |