entinostat has been researched along with Leucocythaemia in 11 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 7 (63.64) | 29.6817 |
2010's | 3 (27.27) | 24.3611 |
2020's | 1 (9.09) | 2.80 |
Authors | Studies |
---|---|
Li, X | 1 |
Zhang, Y | 1 |
Jiang, Y | 1 |
Wu, J | 1 |
Inks, ES | 1 |
Chou, CJ | 1 |
Gao, S | 2 |
Hou, J | 1 |
Ding, Q | 1 |
Li, J | 1 |
Wang, X | 1 |
Huang, Y | 1 |
Xu, W | 1 |
Alves Avelar, LA | 1 |
Schrenk, C | 1 |
Sönnichsen, M | 1 |
Hamacher, A | 1 |
Hansen, FK | 1 |
Schliehe-Diecks, J | 1 |
Borkhardt, A | 1 |
Bhatia, S | 1 |
Kassack, MU | 1 |
Kurz, T | 1 |
Kettyle, LM | 1 |
Liberante, FG | 1 |
Thompson, A | 1 |
Noack, K | 1 |
Mahendrarajah, N | 1 |
Hennig, D | 1 |
Schmidt, L | 1 |
Grebien, F | 1 |
Hildebrand, D | 1 |
Christmann, M | 1 |
Kaina, B | 1 |
Sellmer, A | 1 |
Mahboobi, S | 1 |
Kubatzky, K | 1 |
Heinzel, T | 1 |
Krämer, OH | 1 |
Vire, B | 1 |
de Walque, S | 1 |
Restouin, A | 1 |
Olive, D | 1 |
Van Lint, C | 1 |
Collette, Y | 1 |
Rosato, RR | 2 |
Almenara, JA | 1 |
Grant, S | 3 |
Maggio, SC | 1 |
Kramer, LB | 1 |
Dai, Y | 2 |
Rahmani, M | 2 |
Paik, DS | 1 |
Czarnik, AC | 1 |
Payne, SG | 1 |
Spiegel, S | 1 |
Acharya, MR | 1 |
Figg, WD | 2 |
Dent, P | 1 |
Gojo, I | 1 |
Jiemjit, A | 1 |
Trepel, JB | 1 |
Sparreboom, A | 1 |
Rollins, S | 1 |
Tidwell, ML | 1 |
Greer, J | 1 |
Chung, EJ | 1 |
Lee, MJ | 1 |
Gore, SD | 1 |
Sausville, EA | 1 |
Zwiebel, J | 1 |
Karp, JE | 1 |
Mobley, A | 1 |
Miller, C | 1 |
Boklan, J | 1 |
Chandra, J | 1 |
1 trial available for entinostat and Leucocythaemia
Article | Year |
---|---|
Phase 1 and pharmacologic study of MS-275, a histone deacetylase inhibitor, in adults with refractory and relapsed acute leukemias.
Topics: Acetylation; Adult; Aged; Aged, 80 and over; Antineoplastic Agents; Benzamides; Enzyme Inhibitors; F | 2007 |
10 other studies available for entinostat and Leucocythaemia
Article | Year |
---|---|
Selective HDAC inhibitors with potent oral activity against leukemia and colorectal cancer: Design, structure-activity relationship and anti-tumor activity study.
Topics: Animals; Antineoplastic Agents; Benzamides; Cell Proliferation; Colon; Colorectal Neoplasms; HCT116 | 2017 |
Synergistic induction of apoptosis in resistant head and neck carcinoma and leukemia by alkoxyamide-based histone deacetylase inhibitors.
Topics: Antineoplastic Agents; Apoptosis; Drug Synergism; Epigenomics; Head and Neck Neoplasms; Histone Deac | 2021 |
Rational drug repurposing using sscMap analysis in a HOX-TALE model of leukemia.
Topics: Animals; Antineoplastic Agents; Benzamides; Cell Line, Tumor; Databases, Genetic; Disease Models, An | 2014 |
Analysis of the interplay between all-trans retinoic acid and histone deacetylase inhibitors in leukemic cells.
Topics: Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Benzamides; CCAAT-Enhancer-Binding Protei | 2017 |
Anti-leukemia activity of MS-275 histone deacetylase inhibitor implicates 4-1BBL/4-1BB immunomodulatory functions.
Topics: 4-1BB Ligand; Antineoplastic Agents; Benzamides; Cell Proliferation; Gene Expression Regulation, Leu | 2009 |
The histone deacetylase inhibitor MS-275 promotes differentiation or apoptosis in human leukemia cells through a process regulated by generation of reactive oxygen species and induction of p21CIP1/WAF1 1.
Topics: Apoptosis; Benzamides; Cell Cycle; Cell Differentiation; Cell Division; Cyclin-Dependent Kinase Inhi | 2003 |
The histone deacetylase inhibitor MS-275 interacts synergistically with fludarabine to induce apoptosis in human leukemia cells.
Topics: Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Benzamides; Caspases; Cell Cycle; Cell Cy | 2004 |
Histone deacetylase inhibitor enhances the anti-leukemic activity of an established nucleoside analogue.
Topics: Antineoplastic Agents; Apoptosis; Benzamides; Cell Cycle; Cell Cycle Proteins; DNA (Cytosine-5-)-Met | 2004 |
Blockade of histone deacetylase inhibitor-induced RelA/p65 acetylation and NF-kappaB activation potentiates apoptosis in leukemia cells through a process mediated by oxidative damage, XIAP downregulation, and c-Jun N-terminal kinase 1 activation.
Topics: Acetylation; Apoptosis; Benzamides; Down-Regulation; Enzyme Activation; Enzyme Inhibitors; Gene Expr | 2005 |
Potentiation of reactive oxygen species is a marker for synergistic cytotoxicity of MS-275 and 5-azacytidine in leukemic cells.
Topics: Acetylation; Antimetabolites, Antineoplastic; Apoptosis; Azacitidine; Benzamides; Biomarkers; Caspas | 2008 |