entinostat has been researched along with Central Nervous System Neoplasms in 2 studies
Central Nervous System Neoplasms: Benign and malignant neoplastic processes that arise from or secondarily involve the brain, spinal cord, or meninges.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Glioblastoma is the most common primary brain tumor with a dismal prognosis, highlighting the need for novel treatment strategies." | 1.37 | Chemosensitization of glioblastoma cells by the histone deacetylase inhibitor MS275. ( Bangert, A; Cristofanon, S; Debatin, KM; Fulda, S; Häcker, S, 2011) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Bangert, A | 1 |
| Häcker, S | 1 |
| Cristofanon, S | 1 |
| Debatin, KM | 1 |
| Fulda, S | 1 |
| Knipstein, JA | 1 |
| Birks, DK | 1 |
| Donson, AM | 1 |
| Alimova, I | 1 |
| Foreman, NK | 1 |
| Vibhakar, R | 1 |
2 other studies available for entinostat and Central Nervous System Neoplasms
| Article | Year |
|---|---|
Chemosensitization of glioblastoma cells by the histone deacetylase inhibitor MS275.
Topics: Antibiotics, Antineoplastic; Antineoplastic Agents; Antineoplastic Agents, Alkylating; Antineoplasti | 2011 |
Histone deacetylase inhibition decreases proliferation and potentiates the effect of ionizing radiation in atypical teratoid/rhabdoid tumor cells.
Topics: Acetylation; Apoptosis; Benzamides; Cell Line, Tumor; Central Nervous System Neoplasms; Combined Mod | 2012 |