Page last updated: 2024-08-23

enkephalin, methionine and Cot Death

enkephalin, methionine has been researched along with Cot Death in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19902 (40.00)18.7374
1990's1 (20.00)18.2507
2000's2 (40.00)29.6817
2010's0 (0.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Bergström, L; Lagercrantz, H; Terenius, L1
Franciosi, RA; Kuich, TE1
Mallard, C; McGregor, H; Rees, S; Tolcos, M; Walker, D1
McGregor, H; Rees, S; Tolcos, M; Walker, D1
Boudinot, E; Champagnat, J; Denavit-Saubie, M; Fournie-Zaluski, MC; Morin-Surun, MP; Roques, BP1

Other Studies

5 other study(ies) available for enkephalin, methionine and Cot Death

ArticleYear
Post-mortem analyses of neuropeptides in brains from sudden infant death victims.
    Brain research, 1984, Dec-10, Volume: 323, Issue:2

    Topics: Age Factors; Brain Chemistry; Enkephalin, Methionine; Female; Humans; Infant; Infant, Newborn; Male; Nerve Tissue Proteins; Neuropeptide Y; Postmortem Changes; Sex Factors; Substance P; Sudden Infant Death

1984
A study of the endogenous opioid system in the sudden infant death syndrome.
    Medical hypotheses, 1983, Volume: 10, Issue:4

    Topics: Adrenal Glands; Adult; beta-Endorphin; Brain Chemistry; Drug Stability; Endorphins; Enkephalin, Methionine; Female; Humans; Infant; Infant, Newborn; Lung; Male; Medulla Oblongata; Radioimmunoassay; Sudden Infant Death

1983
Exposure to prenatal carbon monoxide and postnatal hyperthermia: short and long-term effects on neurochemicals and neuroglia in the developing brain.
    Experimental neurology, 2000, Volume: 162, Issue:2

    Topics: Animals; Animals, Newborn; Body Weight; Brain; Brain Chemistry; Carbon Monoxide; Cerebral Cortex; Enkephalin, Methionine; Female; Glial Fibrillary Acidic Protein; Guinea Pigs; Hyperthermia, Induced; Immunohistochemistry; Inhalation Exposure; Maternal Exposure; Medulla Oblongata; Neuroglia; Organ Size; Pregnancy; Prenatal Exposure Delayed Effects; Serotonin; Substance P; Sudden Infant Death; Thalamus; Tyrosine 3-Monooxygenase

2000
Chronic prenatal exposure to carbon monoxide results in a reduction in tyrosine hydroxylase-immunoreactivity and an increase in choline acetyltransferase-immunoreactivity in the fetal medulla: implications for Sudden Infant Death Syndrome.
    Journal of neuropathology and experimental neurology, 2000, Volume: 59, Issue:3

    Topics: Animals; Astrocytes; Blotting, Western; Carbon Monoxide; Choline O-Acetyltransferase; Enkephalin, Methionine; Female; Glial Fibrillary Acidic Protein; Guinea Pigs; Medulla Oblongata; Neurons; Pregnancy; Prenatal Exposure Delayed Effects; Receptor, Muscarinic M2; Receptors, Muscarinic; Smoking; Substance P; Sudden Infant Death; Tyrosine 3-Monooxygenase

2000
Control of breathing by endogenous opioid peptides: possible involvement in sudden infant death syndrome.
    Neurochemistry international, 1992, Volume: 20, Issue:1

    Topics: Animals; Brain Stem; Cats; Dipeptides; Endorphins; Enkephalin, Methionine; Homeostasis; Humans; Infant; Inhalation; Iontophoresis; Models, Neurological; Naloxone; Neurons; Phrenic Nerve; Respiration; Sudden Infant Death

1992