enkephalin, leucine has been researched along with Autosomal Dominant Cerebellar Ataxia, Type II in 1 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Abe, K; Akechi, Y; Habu, T; Hitomi, T; Ikeda, Y; Kobayashi, H; Koizumi, A; Liu, W; Matsuura, T; Okuda, H | 1 |
1 other study(ies) available for enkephalin, leucine and Autosomal Dominant Cerebellar Ataxia, Type II
Article | Year |
---|---|
Expansion of intronic GGCCTG hexanucleotide repeat in NOP56 causes SCA36, a type of spinocerebellar ataxia accompanied by motor neuron involvement.
Topics: Adult; Age of Onset; Aged; Alzheimer Disease; Amyotrophic Lateral Sclerosis; Cohort Studies; Enkephalins; Female; Haplotypes; Humans; In Situ Hybridization, Fluorescence; Introns; Lod Score; Male; MicroRNAs; Middle Aged; Motor Neurons; Mutation, Missense; Nuclear Proteins; Pedigree; Protein Precursors; Spinocerebellar Ataxias | 2011 |