emodin has been researched along with Thrombophilia in 1 studies
Emodin: Purgative anthraquinone found in several plants, especially RHAMNUS PURSHIANA. It was formerly used as a laxative, but is now used mainly as a tool in toxicity studies.
emodin : A trihydroxyanthraquinone that is 9,10-anthraquinone which is substituted by hydroxy groups at positions 1, 3, and 8 and by a methyl group at position 6. It is present in the roots and barks of numerous plants (particularly rhubarb and buckthorn), moulds, and lichens. It is an active ingredient of various Chinese herbs.
Thrombophilia: A disorder of HEMOSTASIS in which there is a tendency for the occurrence of THROMBOSIS.
Excerpt | Relevance | Reference |
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"The aim of this experiment was to evaluate the efficacy of emodin on LPS-provoked alveolar hypercoagulation and excessive pulmonary inflammation in ARDS, and its potential mechanism." | 7.96 | Emodin improves alveolar hypercoagulation and inhibits pulmonary inflammation in LPS-provoked ARDS in mice via NF-κB inactivation. ( Cheng, Y; He, T; Li, S; Li, X; Liu, B; Shen, F; Wu, Y; Yang, G; Zheng, X, 2020) |
"The aim of this experiment was to evaluate the efficacy of emodin on LPS-provoked alveolar hypercoagulation and excessive pulmonary inflammation in ARDS, and its potential mechanism." | 3.96 | Emodin improves alveolar hypercoagulation and inhibits pulmonary inflammation in LPS-provoked ARDS in mice via NF-κB inactivation. ( Cheng, Y; He, T; Li, S; Li, X; Liu, B; Shen, F; Wu, Y; Yang, G; Zheng, X, 2020) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 0 (0.00) | 24.3611 |
2020's | 1 (100.00) | 2.80 |
Authors | Studies |
---|---|
Liu, B | 1 |
Cheng, Y | 1 |
Wu, Y | 1 |
Zheng, X | 1 |
Li, X | 1 |
Yang, G | 1 |
He, T | 1 |
Li, S | 1 |
Shen, F | 1 |
1 other study available for emodin and Thrombophilia
Article | Year |
---|---|
Emodin improves alveolar hypercoagulation and inhibits pulmonary inflammation in LPS-provoked ARDS in mice via NF-κB inactivation.
Topics: Animals; Emodin; Gene Expression Regulation; Inflammation; Lipopolysaccharides; Mice; NF-kappa B; Pu | 2020 |