emodin has been researched along with Pulmonary Edema in 2 studies
Emodin: Purgative anthraquinone found in several plants, especially RHAMNUS PURSHIANA. It was formerly used as a laxative, but is now used mainly as a tool in toxicity studies.
emodin : A trihydroxyanthraquinone that is 9,10-anthraquinone which is substituted by hydroxy groups at positions 1, 3, and 8 and by a methyl group at position 6. It is present in the roots and barks of numerous plants (particularly rhubarb and buckthorn), moulds, and lichens. It is an active ingredient of various Chinese herbs.
Pulmonary Edema: Excessive accumulation of extravascular fluid in the lung, an indication of a serious underlying disease or disorder. Pulmonary edema prevents efficient PULMONARY GAS EXCHANGE in the PULMONARY ALVEOLI, and can be life-threatening.
| Excerpt | Relevance | Reference |
|---|---|---|
| "To investigate the effect of emodin on expression of claudin-4, claudin-5 and occludin, as well as the alveolar epithelial barrier in rats with pancreatitis induced by sodium taurocholate." | 7.76 | Emodin enhances alveolar epithelial barrier function in rats with experimental acute pancreatitis. ( Lu, H; Wan, HJ; Wang, FY; Wang, ZK; Xia, XM; Xu, WA, 2010) |
| "Uncontrolled and self-amplified pulmonary inflammation lies at the center of the pathology of this disease." | 5.40 | Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice. ( Shen, YC; Wan, QF; Wang, T; Wen, FQ; Xiao, M; Zhang, W; Zhu, T, 2014) |
| "To investigate the effect of emodin on expression of claudin-4, claudin-5 and occludin, as well as the alveolar epithelial barrier in rats with pancreatitis induced by sodium taurocholate." | 3.76 | Emodin enhances alveolar epithelial barrier function in rats with experimental acute pancreatitis. ( Lu, H; Wan, HJ; Wang, FY; Wang, ZK; Xia, XM; Xu, WA, 2010) |
| "Uncontrolled and self-amplified pulmonary inflammation lies at the center of the pathology of this disease." | 1.40 | Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice. ( Shen, YC; Wan, QF; Wang, T; Wen, FQ; Xiao, M; Zhang, W; Zhu, T, 2014) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 2 (100.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Xiao, M | 1 |
| Zhu, T | 1 |
| Zhang, W | 1 |
| Wang, T | 1 |
| Shen, YC | 1 |
| Wan, QF | 1 |
| Wen, FQ | 1 |
| Xia, XM | 1 |
| Wang, FY | 1 |
| Wang, ZK | 1 |
| Wan, HJ | 1 |
| Xu, WA | 1 |
| Lu, H | 1 |
2 other studies available for emodin and Pulmonary Edema
| Article | Year |
|---|---|
Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice.
Topics: Acute Lung Injury; Animals; Bronchoalveolar Lavage Fluid; Chemokine CCL2; E-Selectin; Emodin; Enzyme | 2014 |
Emodin enhances alveolar epithelial barrier function in rats with experimental acute pancreatitis.
Topics: Animals; Blood-Air Barrier; Claudin-4; Claudin-5; Emodin; Lung; Male; Membrane Proteins; Occludin; P | 2010 |