emodin has been researched along with Pneumonia in 5 studies
Emodin: Purgative anthraquinone found in several plants, especially RHAMNUS PURSHIANA. It was formerly used as a laxative, but is now used mainly as a tool in toxicity studies.
emodin : A trihydroxyanthraquinone that is 9,10-anthraquinone which is substituted by hydroxy groups at positions 1, 3, and 8 and by a methyl group at position 6. It is present in the roots and barks of numerous plants (particularly rhubarb and buckthorn), moulds, and lichens. It is an active ingredient of various Chinese herbs.
Pneumonia: Infection of the lung often accompanied by inflammation.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Uncontrolled and self-amplified pulmonary inflammation lies at the center of the pathology of this disease." | 5.40 | Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice. ( Shen, YC; Wan, QF; Wang, T; Wen, FQ; Xiao, M; Zhang, W; Zhu, T, 2014) |
| "Silicosis is a fatal pulmonary disease caused by the inhalation of silica dust, and characterized by inflammation and fibrosis of the lung, with no effective treatment to date." | 1.62 | Emodin attenuates silica-induced lung injury by inhibition of inflammation, apoptosis and epithelial-mesenchymal transition. ( Jia, Q; Lavin, MF; Li, C; Nie, X; Pang, X; Peng, C; Shao, H; Shao, L; Xia, Q; Yan, H; Yeo, AJ; Yu, G, 2021) |
| "Treatment with emodin led to significant decreases in the levels of IL-4, IL-5, IL-13 and eotaxin in BALF and total IgE level in serum." | 1.42 | Protective effect of emodin against airway inflammation in the ovalbumin-induced mouse model. ( Gao, YS; Jia, X; Li, YH; Liu, M; Wang, T; Wu, RH; Zhang, SJ; Zhong, XG, 2015) |
| "Emodin treatment abated the DEP-induced increase in airway resistance, and prevented the influx of neutrophils in bronchoalveolar lavage fluid." | 1.42 | Emodin mitigates diesel exhaust particles-induced increase in airway resistance, inflammation and oxidative stress in mice. ( Al-Salam, S; Ali, BH; Beegam, S; Nemmar, A; Yuvaraju, P, 2015) |
| "Uncontrolled and self-amplified pulmonary inflammation lies at the center of the pathology of this disease." | 1.40 | Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice. ( Shen, YC; Wan, QF; Wang, T; Wen, FQ; Xiao, M; Zhang, W; Zhu, T, 2014) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 4 (80.00) | 24.3611 |
| 2020's | 1 (20.00) | 2.80 |
| Authors | Studies |
|---|---|
| Pang, X | 1 |
| Shao, L | 1 |
| Nie, X | 1 |
| Yan, H | 1 |
| Li, C | 1 |
| Yeo, AJ | 1 |
| Lavin, MF | 1 |
| Xia, Q | 1 |
| Shao, H | 1 |
| Yu, G | 1 |
| Jia, Q | 1 |
| Peng, C | 1 |
| Dai, JP | 1 |
| Wang, QW | 1 |
| Su, Y | 1 |
| Gu, LM | 1 |
| Zhao, Y | 1 |
| Chen, XX | 1 |
| Chen, C | 1 |
| Li, WZ | 1 |
| Wang, GF | 1 |
| Li, KS | 1 |
| Xiao, M | 1 |
| Zhu, T | 1 |
| Zhang, W | 1 |
| Wang, T | 2 |
| Shen, YC | 1 |
| Wan, QF | 1 |
| Wen, FQ | 1 |
| Zhong, XG | 1 |
| Li, YH | 1 |
| Jia, X | 1 |
| Zhang, SJ | 1 |
| Gao, YS | 1 |
| Liu, M | 1 |
| Wu, RH | 1 |
| Nemmar, A | 1 |
| Al-Salam, S | 1 |
| Yuvaraju, P | 1 |
| Beegam, S | 1 |
| Ali, BH | 1 |
5 other studies available for emodin and Pneumonia
| Article | Year |
|---|---|
Emodin attenuates silica-induced lung injury by inhibition of inflammation, apoptosis and epithelial-mesenchymal transition.
Topics: A549 Cells; Acute Lung Injury; Animals; Anti-Inflammatory Agents; Apoptosis; Apoptosis Regulatory Pr | 2021 |
Emodin Inhibition of Influenza A Virus Replication and Influenza Viral Pneumonia via the Nrf2, TLR4, p38/JNK and NF-kappaB Pathways.
Topics: Animals; Disease Models, Animal; Emodin; Humans; Influenza A virus; Influenza, Human; Mice; Myeloid | 2017 |
Emodin ameliorates LPS-induced acute lung injury, involving the inactivation of NF-κB in mice.
Topics: Acute Lung Injury; Animals; Bronchoalveolar Lavage Fluid; Chemokine CCL2; E-Selectin; Emodin; Enzyme | 2014 |
Protective effect of emodin against airway inflammation in the ovalbumin-induced mouse model.
Topics: Animals; Bronchoalveolar Lavage Fluid; Chemokines; Disease Models, Animal; Emodin; Female; Gene Expr | 2015 |
Emodin mitigates diesel exhaust particles-induced increase in airway resistance, inflammation and oxidative stress in mice.
Topics: Airway Resistance; Animals; Cytokines; Disease Models, Animal; Dose-Response Relationship, Drug; Emo | 2015 |