emodin has been researched along with Hyperglycemia, Postprandial in 4 studies
Emodin: Purgative anthraquinone found in several plants, especially RHAMNUS PURSHIANA. It was formerly used as a laxative, but is now used mainly as a tool in toxicity studies.
emodin : A trihydroxyanthraquinone that is 9,10-anthraquinone which is substituted by hydroxy groups at positions 1, 3, and 8 and by a methyl group at position 6. It is present in the roots and barks of numerous plants (particularly rhubarb and buckthorn), moulds, and lichens. It is an active ingredient of various Chinese herbs.
Hyperglycemia, Postprandial: Abnormally high BLOOD GLUCOSE level after a meal.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Treatment with emodin significantly turns down the accelerated cell cycle and proliferation of MCs cultured in high glucose (HG) via inhibiting cFLIP." | 5.40 | Emodin suppresses hyperglycemia-induced proliferation and fibronectin expression in mesangial cells via inhibiting cFLIP. ( Cao, S; Gao, J; Guo, C; Su, Z; Wang, F; Wang, W, 2014) |
| "Emodin ameliorated hyperglycemia and dyslipidemia in T2DM rats, and glucose metabolism in a concentration- and time-dependent manner." | 3.91 | Emodin improves glucose metabolism by targeting microRNA-20b in insulin-resistant skeletal muscle. ( Fu, Y; Hu, Y; Li, M; Li, X; Li, Z; Wang, R; Xiao, D; Xu, C; Xuan, L; Yang, B; Zhang, H; Zhang, Y, 2019) |
| "Treatment with emodin significantly turns down the accelerated cell cycle and proliferation of MCs cultured in high glucose (HG) via inhibiting cFLIP." | 1.40 | Emodin suppresses hyperglycemia-induced proliferation and fibronectin expression in mesangial cells via inhibiting cFLIP. ( Cao, S; Gao, J; Guo, C; Su, Z; Wang, F; Wang, W, 2014) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 3 (75.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Suresh Babu, K | 1 |
| Tiwari, AK | 1 |
| Srinivas, PV | 1 |
| Ali, AZ | 1 |
| China Raju, B | 1 |
| Rao, JM | 1 |
| Fan, L | 1 |
| Zhang, H | 2 |
| Li, X | 2 |
| Yang, G | 1 |
| Ru, J | 1 |
| Liu, T | 1 |
| Xiao, D | 1 |
| Hu, Y | 1 |
| Fu, Y | 1 |
| Wang, R | 1 |
| Li, M | 1 |
| Li, Z | 1 |
| Zhang, Y | 2 |
| Xuan, L | 1 |
| Xu, C | 1 |
| Yang, B | 1 |
| Gao, J | 1 |
| Wang, F | 1 |
| Wang, W | 1 |
| Su, Z | 1 |
| Guo, C | 1 |
| Cao, S | 1 |
4 other studies available for emodin and Hyperglycemia, Postprandial
| Article | Year |
|---|---|
Yeast and mammalian alpha-glucosidase inhibitory constituents from Himalayan rhubarb Rheum emodi Wall.ex Meisson.
Topics: alpha-Glucosidases; Animals; Binding, Competitive; Diabetes Mellitus; Enzyme Inhibitors; Glycoside H | 2004 |
Emodin protects hyperglycemia-induced injury in PC-12 cells by up-regulation of miR-9.
Topics: Animals; Apoptosis; Autophagy; Emodin; Glucose; Hyperglycemia; MicroRNAs; Neuroprotection; NF-kappa | 2018 |
Emodin improves glucose metabolism by targeting microRNA-20b in insulin-resistant skeletal muscle.
Topics: Animals; Diabetes Mellitus, Type 2; Down-Regulation; Emodin; Glucose; Glucose Transporter Type 4; Hy | 2019 |
Emodin suppresses hyperglycemia-induced proliferation and fibronectin expression in mesangial cells via inhibiting cFLIP.
Topics: Animals; Apoptosis; CASP8 and FADD-Like Apoptosis Regulating Protein; Cell Cycle Checkpoints; Cell P | 2014 |