Page last updated: 2024-10-26

emodin and Glioblastoma

emodin has been researched along with Glioblastoma in 2 studies

Emodin: Purgative anthraquinone found in several plants, especially RHAMNUS PURSHIANA. It was formerly used as a laxative, but is now used mainly as a tool in toxicity studies.
emodin : A trihydroxyanthraquinone that is 9,10-anthraquinone which is substituted by hydroxy groups at positions 1, 3, and 8 and by a methyl group at position 6. It is present in the roots and barks of numerous plants (particularly rhubarb and buckthorn), moulds, and lichens. It is an active ingredient of various Chinese herbs.

Glioblastoma: A malignant form of astrocytoma histologically characterized by pleomorphism of cells, nuclear atypia, microhemorrhage, and necrosis. They may arise in any region of the central nervous system, with a predilection for the cerebral hemispheres, basal ganglia, and commissural pathways. Clinical presentation most frequently occurs in the fifth or sixth decade of life with focal neurologic signs or seizures.

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (50.00)24.3611
2020's1 (50.00)2.80

Authors

AuthorsStudies
Staffieri, S1
Russo, V1
Oliva, MA1
Alborghetti, M1
Russo, M1
Arcella, A1
Li, W1
Li, F1
Zhu, Y1
Song, D1

Other Studies

2 other studies available for emodin and Glioblastoma

ArticleYear
Aloe-Emodin Overcomes Anti-Cancer Drug Resistance to Temozolomide and Prevents Colony Formation and Migration in Primary Human Glioblastoma Cell Lines NULU and ZAR.
    Molecules (Basel, Switzerland), 2023, Aug-11, Volume: 28, Issue:16

    Topics: Aloe; Cell Line; Emodin; Glioblastoma; Humans; Temozolomide

2023
RETRACTED: Physcion 8-O-β-glucopyranosideregulates cell cycle, apoptosis, and invasion in glioblastoma cells through modulating Skp2.
    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie, 2017, Volume: 95

    Topics: Acetylcysteine; Adenylate Kinase; Antineoplastic Agents; Apoptosis; Brain Neoplasms; Cell Cycle; Cel

2017