emodin has been researched along with Cognitive Dysfunction in 3 studies
Emodin: Purgative anthraquinone found in several plants, especially RHAMNUS PURSHIANA. It was formerly used as a laxative, but is now used mainly as a tool in toxicity studies.
emodin : A trihydroxyanthraquinone that is 9,10-anthraquinone which is substituted by hydroxy groups at positions 1, 3, and 8 and by a methyl group at position 6. It is present in the roots and barks of numerous plants (particularly rhubarb and buckthorn), moulds, and lichens. It is an active ingredient of various Chinese herbs.
Cognitive Dysfunction: Diminished or impaired mental and/or intellectual function.
| Excerpt | Relevance | Reference |
|---|
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 0 (0.00) | 29.6817 |
| 2010's | 1 (33.33) | 24.3611 |
| 2020's | 2 (66.67) | 2.80 |
| Authors | Studies |
|---|---|
| Xie, Y | 1 |
| Liu, L | 1 |
| Zheng, J | 1 |
| Shi, K | 1 |
| Ai, W | 1 |
| Zhang, X | 1 |
| Wang, P | 1 |
| Lan, Z | 1 |
| Chen, L | 2 |
| Wang, L | 1 |
| Liu, S | 1 |
| Xu, J | 1 |
| Watanabe, N | 1 |
| Mayo, KH | 1 |
| Li, J | 1 |
| Li, X | 1 |
| Zhong, J | 1 |
| Wang, F | 1 |
| Wang, Z | 1 |
| Shen, C | 1 |
| Zheng, Y | 1 |
| Ma, F | 1 |
| Zhu, T | 1 |
| Tang, Q | 1 |
| Zhu, J | 1 |
3 other studies available for emodin and Cognitive Dysfunction
| Article | Year |
|---|---|
Polygoni Multiflori Radix Praeparata and Acori Tatarinowii Rhizoma ameliorate scopolamine-induced cognitive impairment by regulating the cholinergic and synaptic associated proteins.
Topics: Acetylcholinesterase; Animals; Cholinergic Agents; Cognitive Dysfunction; Emodin; GAP-43 Protein; Ma | 2023 |
Emodin inhibits aggregation of amyloid-β peptide 1-42 and improves cognitive deficits in Alzheimer's disease transgenic mice.
Topics: Alzheimer Disease; Amyloid beta-Peptides; Animals; Cognitive Dysfunction; Emodin; Female; Male; Mice | 2021 |
Aloin attenuates cognitive impairment and inflammation induced by d-galactose via down-regulating ERK, p38 and NF-κB signaling pathway.
Topics: Animals; Brain; Cognitive Dysfunction; Cytokines; Down-Regulation; Emodin; Galactose; Glutathione; M | 2019 |