Page last updated: 2024-09-05

emd 53998 and Muscle Contraction

emd 53998 has been researched along with Muscle Contraction in 7 studies

Research

Studies (7)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's3 (42.86)29.6817
2010's4 (57.14)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Han, SJ; Regnier, M; Rodriguez, AG; Rodriguez, ML; Sniadecki, NJ1
Copeland, O; Ferenczi, MA; Marston, SB; Messer, AE; Song, W; Vikhorev, PG; Wilkinson, R1
Marston, SB; Messer, AE; Papadaki, M; Vikhorev, PG1
Eriksson, LI; Larsson, L; Ochala, J; Radell, PJ1
Georgakopoulos, D; Kass, DA; Murphy, AM; Soergel, DG; Stull, LB1
Arteaga, GM; Leiden, JM; Palmiter, KA; Solaro, RJ1
Hongo, K; Kawai, M; Konishi, M; Kurihara, S; Kusakari, Y; Mochizuki, S1

Other Studies

7 other study(ies) available for emd 53998 and Muscle Contraction

ArticleYear
Enhanced contractility with 2-deoxy-ATP and EMD 57033 is correlated with reduced myofibril structure and twitch power in neonatal cardiomyocytes.
    Integrative biology : quantitative biosciences from nano to macro, 2013, Volume: 5, Issue:11

    Topics: Actinin; Animals; Animals, Newborn; Cardiotonic Agents; Cells, Cultured; Deoxyadenine Nucleotides; Green Fluorescent Proteins; Heterocyclic Compounds, 4 or More Rings; Homeostasis; Microscopy, Fluorescence; Muscle Contraction; Myocytes, Cardiac; Myofibrils; Optics and Photonics; Quinolines; Rats; Rats, Inbred F344; Sarcomeres; Signal Transduction; Stress, Mechanical; Thiadiazines

2013
The dilated cardiomyopathy-causing mutation ACTC E361G in cardiac muscle myofibrils specifically abolishes modulation of Ca(2+) regulation by phosphorylation of troponin I.
    Biophysical journal, 2014, Nov-18, Volume: 107, Issue:10

    Topics: Actins; Animals; Calcium; Cardiomyopathy, Dilated; Carrier Proteins; Cyclic AMP-Dependent Protein Kinases; Female; Male; Mice; Mice, Transgenic; Muscle Contraction; Mutation; Myofibrils; Phosphorylation; Propranolol; Quinolines; Sarcomeres; Thiadiazines; Troponin I

2014
Uncoupling of myofilament Ca2+ sensitivity from troponin I phosphorylation by mutations can be reversed by epigallocatechin-3-gallate.
    Cardiovascular research, 2015, Oct-01, Volume: 108, Issue:1

    Topics: Animals; Bepridil; Calcium; Catechin; Humans; Mice; Muscle Contraction; Mutation; Myofibrils; Phosphorylation; Quinolines; Rabbits; Thiadiazines; Troponin I

2015
EMD 57033 partially reverses ventilator-induced diaphragm muscle fibre calcium desensitisation.
    Pflugers Archiv : European journal of physiology, 2010, Volume: 459, Issue:3

    Topics: Actins; Animals; Calcium; Diaphragm; Female; Humans; Muscle Contraction; Muscle Fibers, Skeletal; Myosins; Phosphodiesterase Inhibitors; Quinolines; Random Allocation; Swine; Thiadiazines; Ventilators, Mechanical

2010
Augmented systolic response to the calcium sensitizer EMD-57033 in a transgenic model with troponin I truncation.
    American journal of physiology. Heart and circulatory physiology, 2004, Volume: 286, Issue:5

    Topics: Animals; Ca(2+) Mg(2+)-ATPase; Cardiotonic Agents; Hemodynamics; In Vitro Techniques; Mice; Mice, Transgenic; Muscle Contraction; Muscles; Mutation; Myocardial Contraction; Myocardium; Myofibrils; Quinolines; Systole; Thiadiazines; Troponin I

2004
Attenuation of length dependence of calcium activation in myofilaments of transgenic mouse hearts expressing slow skeletal troponin I.
    The Journal of physiology, 2000, Aug-01, Volume: 526 Pt 3

    Topics: Actin Cytoskeleton; Actins; Animals; Azocines; Calcium; Cardiotonic Agents; Dose-Response Relationship, Drug; Enzyme Inhibitors; Imidazoles; In Vitro Techniques; Mice; Mice, Transgenic; Muscle Contraction; Myocardium; Phosphodiesterase Inhibitors; Protein Isoforms; Quinolines; Sarcomeres; Thiadiazines; Troponin I

2000
Use of tetanus to investigate myofibrillar responsiveness to Ca(2+) in isolated mouse ventricular myocytes.
    The Japanese journal of physiology, 2002, Volume: 52, Issue:1

    Topics: 1-Methyl-3-isobutylxanthine; Adrenergic beta-Agonists; Animals; Calcium; Calcium-Transporting ATPases; Cardiotonic Agents; Cell Separation; Enzyme Inhibitors; Fluorescence; Isoproterenol; Mice; Muscle Contraction; Myocardium; Myofibrils; Phosphodiesterase Inhibitors; Quinolines; Thapsigargin; Thiadiazines; Ventricular Function

2002