emd 53998 has been researched along with Asymmetric Septal Hypertrophy, Familial in 2 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (100.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Arteaga, GM; Burkart, EM; Prabhakar, R; Solaro, RJ; Sumandea, MP; Wieczorek, DF | 1 |
| Knollmann, BC; Potter, JD; Sirenko, SG | 1 |
2 other study(ies) available for emd 53998 and Asymmetric Septal Hypertrophy, Familial
| Article | Year |
|---|---|
Altered signaling surrounding the C-lobe of cardiac troponin C in myofilaments containing an alpha-tropomyosin mutation linked to familial hypertrophic cardiomyopathy.
Topics: Animals; Blotting, Western; Calcium; Cardiomyopathy, Hypertrophic, Familial; Dose-Response Relationship, Drug; Electrophoresis, Polyacrylamide Gel; Glutamic Acid; Mice; Mice, Transgenic; Muscle Fibers, Skeletal; Mutation; Myocardium; Phosphorylation; Protein Binding; Protein Kinase C; Protein Structure, Tertiary; Quinolines; Serine; Signal Transduction; Thiadiazines; Time Factors; Tropomyosin; Troponin C | 2003 |
Differential effect of troponin T mutations on the inotropic responsiveness of mouse hearts--role of myofilament Ca2+ sensitivity increase.
Topics: Actin Cytoskeleton; Adrenergic beta-Antagonists; Animals; Calcium; Cardiomyopathy, Hypertrophic, Familial; Cardiotonic Agents; Exercise Tolerance; Heart; Isoproterenol; Mice; Mice, Transgenic; Mutation; Myocardial Contraction; Myocardium; Propranolol; Quinolines; Thiadiazines; Troponin T | 2006 |