egtazic acid has been researched along with Encephalopathy, Toxic in 4 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (50.00) | 29.6817 |
| 2010's | 2 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Chang, CY; Chou, SS; Huang, SK; Lin, TY; Lu, CW; Wang, SJ; Wang, YC | 1 |
| Costa, LG; Pellacani, C; Roqué, PJ; Tagliaferri, S | 1 |
| Blom, P; Kox, WJ; Müller, J; Petzelt, C; Schmehl, W | 1 |
| Kasaba, T; Kashiwada, M; Onizuka, S; Takasaki, M | 1 |
4 other study(ies) available for egtazic acid and Encephalopathy, Toxic
| Article | Year |
|---|---|
Hesperidin inhibits glutamate release and exerts neuroprotection against excitotoxicity induced by kainic acid in the hippocampus of rats.
Topics: 4-Aminopyridine; Animals; Calcium; Disease Models, Animal; Egtazic Acid; Enzyme Inhibitors; Excitatory Amino Acid Agonists; Excitatory Postsynaptic Potentials; Glutamic Acid; Hesperidin; Hippocampus; Kainic Acid; Male; Membrane Potentials; Neuroprotective Agents; Neurotoxicity Syndromes; Potassium Channel Blockers; Rats; Rats, Sprague-Dawley; Sodium Channel Blockers; Synaptosomes; Tetrodotoxin | 2015 |
Role of glutamate receptors in tetrabrominated diphenyl ether (BDE-47) neurotoxicity in mouse cerebellar granule neurons.
Topics: Animals; Calcium; Cell Death; Cerebellum; Chelating Agents; Cytoplasmic Granules; Egtazic Acid; Excitatory Amino Acid Antagonists; Flame Retardants; Halogenated Diphenyl Ethers; Mice; Mice, Inbred C57BL; Neurons; Neurotoxicity Syndromes; Oxidative Stress; Receptors, AMPA; Receptors, Glutamate; Receptors, Ionotropic Glutamate; Receptors, Metabotropic Glutamate; Receptors, N-Methyl-D-Aspartate | 2016 |
Prevention of neurotoxicity in hypoxic cortical neurons by the noble gas xenon.
Topics: Anesthetics, Inhalation; Animals; Benzylamines; Calcium Signaling; Calcium-Calmodulin-Dependent Protein Kinase Type 2; Calcium-Calmodulin-Dependent Protein Kinases; Cell Hypoxia; Cell Survival; Cells, Cultured; Cerebral Cortex; Chelating Agents; Dizocilpine Maleate; Egtazic Acid; Glutamic Acid; Helium; Hypoxia, Brain; L-Lactate Dehydrogenase; Neurons; Neuroprotective Agents; Neurotoxicity Syndromes; Nitrogen; Rats; Sulfonamides; Xenon | 2003 |
Increase in intracellular Ca2+ concentration is not the only cause of lidocaine-induced cell damage in the cultured neurons of Lymnaea stagnalis.
Topics: Anesthetics, Local; Animals; Calcimycin; Calcium; Cell Culture Techniques; Chelating Agents; Coloring Agents; Dose-Response Relationship, Drug; Egtazic Acid; Intracellular Fluid; Ion Transport; Ionophores; Lidocaine; Lymnaea; Neurons; Neurotoxicity Syndromes; Time Factors; Trypan Blue | 2006 |