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ebselen and Bone Loss, Osteoclastic

ebselen has been researched along with Bone Loss, Osteoclastic in 1 studies

ebselen : A benzoselenazole that is 1,2-benzoselenazol-3-one carrying an additional phenyl substituent at position 2. Acts as a mimic of glutathione peroxidase.

Research Excerpts

ExcerptRelevanceReference
"Ebselen is a non-toxic seleno-organic drug with anti-inflammatory and antioxidant properties that is currently being examined in clinical trials to prevent and treat various diseases, including atherosclerosis, stroke, and cancer."5.43Ebselen Is a Potential Anti-Osteoporosis Agent by Suppressing Receptor Activator of Nuclear Factor Kappa-B Ligand-Induced Osteoclast Differentiation In vitro and Lipopolysaccharide-Induced Inflammatory Bone Destruction In vivo. ( Baek, JM; Kim, JY; Lee, MS; Oh, J; Yoon, KH, 2016)
"Ebselen is a non-toxic seleno-organic drug with anti-inflammatory and antioxidant properties that is currently being examined in clinical trials to prevent and treat various diseases, including atherosclerosis, stroke, and cancer."1.43Ebselen Is a Potential Anti-Osteoporosis Agent by Suppressing Receptor Activator of Nuclear Factor Kappa-B Ligand-Induced Osteoclast Differentiation In vitro and Lipopolysaccharide-Induced Inflammatory Bone Destruction In vivo. ( Baek, JM; Kim, JY; Lee, MS; Oh, J; Yoon, KH, 2016)

Research

Studies (1)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Baek, JM1
Kim, JY1
Yoon, KH1
Oh, J1
Lee, MS1

Other Studies

1 other study available for ebselen and Bone Loss, Osteoclastic

ArticleYear
Ebselen Is a Potential Anti-Osteoporosis Agent by Suppressing Receptor Activator of Nuclear Factor Kappa-B Ligand-Induced Osteoclast Differentiation In vitro and Lipopolysaccharide-Induced Inflammatory Bone Destruction In vivo.
    International journal of biological sciences, 2016, Volume: 12, Issue:5

    Topics: Animals; Azoles; Bone Resorption; Cell Differentiation; Inflammation; Isoindoles; Lipopolysaccharide

2016