Page last updated: 2024-10-26

diphenyleneiodonium and Carcinoma, Ductal, Pancreatic

diphenyleneiodonium has been researched along with Carcinoma, Ductal, Pancreatic in 2 studies

diphenyleneiodonium: structure in first source; NADPH oxidase inhibitor
dibenziodolium : An organic cation that is fluorene in which the methylene group is replaced by a positively charged iodine.

Research Excerpts

ExcerptRelevanceReference
"Treatment of pancreatic cancer cells with TGF-β induced Nox4 expression, indicating that Nox4 represents a major source for ROS production."1.39Nox4-derived ROS signaling contributes to TGF-β-induced epithelial-mesenchymal transition in pancreatic cancer cells. ( Hiraga, R; Kamata, T; Kato, M; Miyagawa, S, 2013)

Research

Studies (2)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's1 (50.00)24.3611
2020's1 (50.00)2.80

Authors

AuthorsStudies
Liu, M1
Wang, D1
Luo, Y1
Hu, L1
Bi, Y1
Ji, J1
Huang, H1
Wang, G1
Zhu, L1
Ma, J1
Kim, E1
Luo, CK1
Abbruzzese, JL1
Li, X1
Yang, VW1
Li, Z1
Lu, W1
Hiraga, R1
Kato, M1
Miyagawa, S1
Kamata, T1

Other Studies

2 other studies available for diphenyleneiodonium and Carcinoma, Ductal, Pancreatic

ArticleYear
Selective killing of cancer cells harboring mutant RAS by concomitant inhibition of NADPH oxidase and glutathione biosynthesis.
    Cell death & disease, 2021, 02-16, Volume: 12, Issue:2

    Topics: Animals; Antineoplastic Combined Chemotherapy Protocols; Carcinoma, Pancreatic Ductal; Cell Death; C

2021
Nox4-derived ROS signaling contributes to TGF-β-induced epithelial-mesenchymal transition in pancreatic cancer cells.
    Anticancer research, 2013, Volume: 33, Issue:10

    Topics: Acetylcysteine; Antigens, CD; Cadherins; Carcinoma, Pancreatic Ductal; Cell Line, Tumor; Enzyme Indu

2013