Page last updated: 2024-08-22

diphenylarsinic acid and Liver Neoplasms

diphenylarsinic acid has been researched along with Liver Neoplasms in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (33.33)29.6817
2010's2 (66.67)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Doi, K; Fujioka, M; Gi, M; Kakehashi, A; Okuno, T; Wanibuchi, H; Yamaguchi, T1
Fujioka, M; Kakehashi, A; Kato, M; Kitano, M; Tago, Y; Wanibuchi, H; Wei, M; Yamada, T; Yamano, S1
Kita, K; Ochi, T; Sato, M; Suzuki, T1

Other Studies

3 other study(ies) available for diphenylarsinic acid and Liver Neoplasms

ArticleYear
A carcinogenicity study of diphenylarsinic acid in F344 rats in drinking water for 104 weeks.
    The Journal of toxicological sciences, 2017, Volume: 42, Issue:4

    Topics: Administration, Oral; Animals; Arsenicals; Body Weight; Chemical Warfare Agents; Dose-Response Relationship, Drug; Female; Liver Neoplasms; Male; Rats, Inbred F344; Survival Rate; Time Factors; Water; Water Pollutants, Chemical

2017
Diphenylarsinic acid, a chemical warfare-related neurotoxicant, promotes liver carcinogenesis via activation of aryl hydrocarbon receptor signaling and consequent induction of oxidative DNA damage in rats.
    Toxicology and applied pharmacology, 2013, Nov-15, Volume: 273, Issue:1

    Topics: Animals; Apoptosis; Arsenicals; Aryl Hydrocarbon Hydroxylases; bcl-2-Associated X Protein; Body Weight; Carcinogenesis; Carcinogens; Chemical Warfare Agents; Cytochrome P-450 CYP1B1; Diethylnitrosamine; DNA Damage; Japan; Liver; Liver Neoplasms; Male; Neurotoxins; Organ Size; Oxidative Stress; Rats; Rats, Inbred F344; Receptors, Aryl Hydrocarbon; Signal Transduction; Transcriptional Activation

2013
Structure-effect relationship in the down-regulation of glutaminase in cultured human cells by phenylarsenic compounds.
    Toxicology, 2009, Apr-28, Volume: 258, Issue:2-3

    Topics: Arsenicals; Carcinoma, Hepatocellular; Cell Line, Tumor; Cell Survival; Dose-Response Relationship, Drug; Down-Regulation; Glutaminase; Hepatocytes; Humans; Liver Neoplasms; Molecular Structure; Time Factors

2009