diphenylamine has been researched along with Neurofibroma, Plexiform in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 1 (50.00) | 2.80 |
Authors | Studies |
---|---|
Allen, JC; Blakeley, J; Cutter, G; Dombi, E; Edwards, L; Emoto, C; Fisher, MJ; Fukuda, T; Goldman, S; Korf, B; Packer, R; Plotkin, SR; Ratner, N; Robinson, CT; Robison, NJ; Schorry, E; Tonsgard, JH; Vinks, AA; Weiss, BD; Widemann, BC; Wolters, PL | 1 |
Chapuis, N; Hivelin, M; Hubas, A; Lantieri, L; Laurendeau, I; Nusbaum, P; Parfait, B; Pasmant, E; Poulain, L; Varin, J; Vidaud, M; Wolkenstein, P | 1 |
1 trial(s) available for diphenylamine and Neurofibroma, Plexiform
Article | Year |
---|---|
NF106: A Neurofibromatosis Clinical Trials Consortium Phase II Trial of the MEK Inhibitor Mirdametinib (PD-0325901) in Adolescents and Adults With NF1-Related Plexiform Neurofibromas.
Topics: Adolescent; Adult; Benzamides; Diphenylamine; Female; Humans; Magnetic Resonance Imaging; Male; Mitogen-Activated Protein Kinase Kinases; Neurofibroma, Plexiform; Neurofibromatosis 1; Protein Kinase Inhibitors; Time Factors; Treatment Outcome; United States; Young Adult | 2021 |
1 other study(ies) available for diphenylamine and Neurofibroma, Plexiform
Article | Year |
---|---|
Dual mTORC1/2 inhibition induces anti-proliferative effect in NF1-associated plexiform neurofibroma and malignant peripheral nerve sheath tumor cells.
Topics: Antineoplastic Agents; Benzamides; Cell Cycle Proteins; Cell Line, Tumor; Cell Movement; Cell Proliferation; Diphenylamine; Drug Synergism; G1 Phase Cell Cycle Checkpoints; Humans; Inhibitory Concentration 50; MAP Kinase Kinase 1; MAP Kinase Kinase 2; Mechanistic Target of Rapamycin Complex 1; Mechanistic Target of Rapamycin Complex 2; Morpholines; Nerve Sheath Neoplasms; Neurofibroma, Plexiform; Neurofibromatosis 1; Neurofibromin 1; Nuclear Proteins; Primary Cell Culture; Protein Kinase Inhibitors; Proto-Oncogene Proteins c-akt; ras Proteins; Schwann Cells; Signal Transduction; Sirolimus; TOR Serine-Threonine Kinases; Transcription Factors | 2016 |