Page last updated: 2024-08-21

diphenylamine and Lung Adenocarcinoma

diphenylamine has been researched along with Lung Adenocarcinoma in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Cha, HJ; Go, YH; Hong, SK; Kwon, OS; Kwon, SJ; Oh, E1
Juan, J; McMahon, M; Sweet-Cordero, A; Trejo, CL; Vicent, S1
Christensen, JG; Eswaraka, J; Giddabasappa, A; Kan, JL; Lalwani, K; Lappin, PB; Lee, JH; Shojaei, F; Simmons, BH; Snider, BA; Wong, A1

Other Studies

3 other study(ies) available for diphenylamine and Lung Adenocarcinoma

ArticleYear
BCL2 induced by LAMTOR3/MAPK is a druggable target of chemoradioresistance in mesenchymal lung cancer.
    Cancer letters, 2017, 09-10, Volume: 403

    Topics: A549 Cells; Adaptor Proteins, Signal Transducing; Adenocarcinoma; Adenocarcinoma of Lung; Aniline Compounds; Antineoplastic Combined Chemotherapy Protocols; Benzamides; Biphenyl Compounds; Cell Proliferation; Cell Survival; Chemoradiotherapy; Diphenylamine; Dose-Response Relationship, Drug; Drug Resistance, Neoplasm; Etoposide; Gene Expression Regulation, Neoplastic; Humans; Lung Neoplasms; MAP Kinase Kinase 1; Mitogen-Activated Protein Kinase 3; Mitogen-Activated Protein Kinases; Molecular Mimicry; Nitrophenols; Peptide Fragments; Piperazines; Protein Kinase Inhibitors; Proto-Oncogene Proteins; Proto-Oncogene Proteins c-bcl-2; Radiation Tolerance; RNA Interference; Signal Transduction; Sulfonamides; Transfection; Up-Regulation

2017
MEK1/2 inhibition elicits regression of autochthonous lung tumors induced by KRASG12D or BRAFV600E.
    Cancer research, 2012, Jun-15, Volume: 72, Issue:12

    Topics: Adenocarcinoma; Adenocarcinoma of Lung; Animals; Antineoplastic Agents; Benzamides; Carcinoma, Non-Small-Cell Lung; Cell Proliferation; Diphenylamine; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Lung Neoplasms; MAP Kinase Kinase 1; MAP Kinase Kinase 2; MAP Kinase Signaling System; Mice; Mice, Transgenic; Proto-Oncogene Proteins B-raf; Proto-Oncogene Proteins p21(ras); Respiratory Mucosa; Tumor Cells, Cultured

2012
Combination of a MEK inhibitor at sub-MTD with a PI3K/mTOR inhibitor significantly suppresses growth of lung adenocarcinoma tumors in Kras(G12D-LSL) mice.
    Cancer chemotherapy and pharmacology, 2012, Volume: 70, Issue:2

    Topics: Adenocarcinoma; Adenocarcinoma of Lung; Animals; Antineoplastic Combined Chemotherapy Protocols; Benzamides; Cell Line, Tumor; Diphenylamine; Dose-Response Relationship, Drug; Heterozygote; Humans; Lung Neoplasms; Maximum Tolerated Dose; Mice; Mice, Mutant Strains; Mitogen-Activated Protein Kinase Kinases; Neoplasm Transplantation; Phosphoinositide-3 Kinase Inhibitors; Proto-Oncogene Proteins p21(ras); Pyridones; Pyrimidines; TOR Serine-Threonine Kinases

2012