diphenylamine has been researched along with Leukemia, Lymphoblastic, Acute, T Cell in 2 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 2 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Akagi, K; Aster, JC; Chen, SC; Dail, M; Downing, JR; Lawrence, J; Lee, LB; Li, Q; Nakitandwe, J; O'Connor, D; Pear, WS; Sampath, D; Shannon, K; Wong, J; Xu, J | 1 |
Akagi, K; Braun, BS; Dail, M; Huang, B; Kang, HC; Kogan, SC; Li, Q; McDaniel, A; Shannon, K; Shokat, K; Wolff, L; Wong, J | 1 |
2 other study(ies) available for diphenylamine and Leukemia, Lymphoblastic, Acute, T Cell
Article | Year |
---|---|
Loss of oncogenic Notch1 with resistance to a PI3K inhibitor in T-cell leukaemia.
Topics: Animals; Benzamides; Clone Cells; Diphenylamine; Down-Regulation; Drug Resistance, Neoplasm; Drug Synergism; Genes, ras; Indazoles; Male; Mice; Mice, Inbred C57BL; Mitogen-Activated Protein Kinase Kinases; Phosphoinositide-3 Kinase Inhibitors; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma; Protein Kinase Inhibitors; Protein Structure, Tertiary; Proto-Oncogene Proteins c-akt; Receptor, Notch1; Signal Transduction; Sulfonamides | 2014 |
Mutant Ikzf1, KrasG12D, and Notch1 cooperate in T lineage leukemogenesis and modulate responses to targeted agents.
Topics: Amino Acid Substitution; Amyloid Precursor Protein Secretases; Animals; Antineoplastic Agents; Benzamides; Cell Line, Tumor; Cell Lineage; Clone Cells; Diphenylamine; Enzyme Inhibitors; Genetic Loci; Humans; Ikaros Transcription Factor; Integrases; Mice; Models, Immunological; Mutant Proteins; Mutation; Precursor T-Cell Lymphoblastic Leukemia-Lymphoma; Proto-Oncogene Proteins p21(ras); Receptor, Notch1; Retroviridae; Signal Transduction | 2010 |