Page last updated: 2024-08-21

diphenylamine and Insulin Sensitivity

diphenylamine has been researched along with Insulin Sensitivity in 1 studies

Research

Studies (1)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	1 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Awazu, M; Harada, A; Iwasaki, Y; Kohno, M; Kugisaki, S; Ozaki, KI; Tamiya, M; Tanimura, S	1

Other Studies

1 other study(ies) available for diphenylamine and Insulin Sensitivity

Article	Year
Targeting the ERK signaling pathway as a potential treatment for insulin resistance and type 2 diabetes. American journal of physiology. Endocrinology and metabolism, 2016, Apr-15, Volume: 310, Issue:8 Topics: 3T3-L1 Cells; Adipocytes; Adipokines; Adiponectin; Animals; Benzamides; Blood Glucose; Chemokine CCL2; Diabetes Mellitus, Type 2; Diet, High-Fat; Diphenylamine; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Fatty Acids, Nonesterified; Glucose Tolerance Test; Hyperlipidemias; Immunoblotting; In Vitro Techniques; Insulin; Insulin Resistance; Interleukin-6; Lipolysis; Male; MAP Kinase Signaling System; Mice; Protein Kinase Inhibitors; Real-Time Polymerase Chain Reaction; Receptors, Leptin; Reverse Transcriptase Polymerase Chain Reaction; Signal Transduction; Triglycerides; Tumor Necrosis Factor-alpha	2016

Article

Year

Targeting the ERK signaling pathway as a potential treatment for insulin resistance and type 2 diabetes.

American journal of physiology. Endocrinology and metabolism, 2016, Apr-15, Volume: 310, Issue:8

Topics: 3T3-L1 Cells; Adipocytes; Adipokines; Adiponectin; Animals; Benzamides; Blood Glucose; Chemokine CCL2; Diabetes Mellitus, Type 2; Diet, High-Fat; Diphenylamine; Disease Models, Animal; Extracellular Signal-Regulated MAP Kinases; Fatty Acids, Nonesterified; Glucose Tolerance Test; Hyperlipidemias; Immunoblotting; In Vitro Techniques; Insulin; Insulin Resistance; Interleukin-6; Lipolysis; Male; MAP Kinase Signaling System; Mice; Protein Kinase Inhibitors; Real-Time Polymerase Chain Reaction; Receptors, Leptin; Reverse Transcriptase Polymerase Chain Reaction; Signal Transduction; Triglycerides; Tumor Necrosis Factor-alpha

2016