Page last updated: 2024-08-21

diphenylamine and Failure to Thrive

diphenylamine has been researched along with Failure to Thrive in 2 studies

Research

Studies (2)

Timeframe	Studies, this research(%)	All Research%
pre-1990	0 (0.00)	18.7374
1990's	0 (0.00)	18.2507
2000's	0 (0.00)	29.6817
2010's	2 (100.00)	24.3611
2020's	0 (0.00)	2.80

Authors

Authors	Studies
Aoki, Y; Inoue, S; Kure, S; Matsubara, Y; Miyagawa-Tomita, S; Moriya, M; Niihori, T; Oba, D; Ogura, T; Ono, M; Watanabe, Y	1
Anastasaki, C; Patton, EE; Rauen, KA	1

Other Studies

2 other study(ies) available for diphenylamine and Failure to Thrive

Article	Year
New BRAF knockin mice provide a pathogenetic mechanism of developmental defects and a therapeutic approach in cardio-facio-cutaneous syndrome. Human molecular genetics, 2014, Dec-15, Volume: 23, Issue:24 Topics: Animals; Benzamides; Benzazepines; Diphenylamine; Disease Models, Animal; Drug Synergism; Ectodermal Dysplasia; Embryo, Mammalian; Facies; Failure to Thrive; Female; Gene Expression Regulation; Gene Knock-In Techniques; Genes, Lethal; Heart Defects, Congenital; Histone Deacetylase Inhibitors; Histone Demethylases; Humans; Liver; Male; MAP Kinase Kinase Kinases; Mice; Mice, Transgenic; Mutation; Myocardium; Protein Kinase Inhibitors; Proto-Oncogene Proteins B-raf; Pyrimidines; Signal Transduction; Skull	2014
Continual low-level MEK inhibition ameliorates cardio-facio-cutaneous phenotypes in zebrafish. Disease models & mechanisms, 2012, Volume: 5, Issue:4 Topics: Animals; Benzamides; Diphenylamine; Ectodermal Dysplasia; Embryo, Nonmammalian; Facies; Failure to Thrive; Heart Defects, Congenital; Mitogen-Activated Protein Kinase Kinases; Phenotype; Protein Kinase Inhibitors; Proto-Oncogene Proteins B-raf; Small Molecule Libraries; Zebrafish	2012

Article

Year

New BRAF knockin mice provide a pathogenetic mechanism of developmental defects and a therapeutic approach in cardio-facio-cutaneous syndrome.

Human molecular genetics, 2014, Dec-15, Volume: 23, Issue:24

Topics: Animals; Benzamides; Benzazepines; Diphenylamine; Disease Models, Animal; Drug Synergism; Ectodermal Dysplasia; Embryo, Mammalian; Facies; Failure to Thrive; Female; Gene Expression Regulation; Gene Knock-In Techniques; Genes, Lethal; Heart Defects, Congenital; Histone Deacetylase Inhibitors; Histone Demethylases; Humans; Liver; Male; MAP Kinase Kinase Kinases; Mice; Mice, Transgenic; Mutation; Myocardium; Protein Kinase Inhibitors; Proto-Oncogene Proteins B-raf; Pyrimidines; Signal Transduction; Skull

2014

Continual low-level MEK inhibition ameliorates cardio-facio-cutaneous phenotypes in zebrafish.

Disease models & mechanisms, 2012, Volume: 5, Issue:4

Topics: Animals; Benzamides; Diphenylamine; Ectodermal Dysplasia; Embryo, Nonmammalian; Facies; Failure to Thrive; Heart Defects, Congenital; Mitogen-Activated Protein Kinase Kinases; Phenotype; Protein Kinase Inhibitors; Proto-Oncogene Proteins B-raf; Small Molecule Libraries; Zebrafish

2012