dinoprost and Polyuria

dinoprost has been researched along with Polyuria* in 2 studies

Other Studies

2 other study(ies) available for dinoprost and Polyuria

Article	Year
Thenogenology question of the month. Pyometra. Journal of the American Veterinary Medical Association, 2002, Jan-15, Volume: 220, Issue:2 Topics: Animals; Anti-Bacterial Agents; Dinoprost; Dog Diseases; Dogs; Escherichia coli Infections; Female; Leukocyte Count; Polyuria; Suppuration; Urinalysis; Uterine Diseases	2002
Post-obstructive urinary concentrating defect. A case study in the role of prostaglandins. Acta paediatrica Scandinavica, 1987, Volume: 76, Issue:6 A child with post-obstructive urinary concentrating defect was studied for the possible pathophysiological role of prostaglandins and an eventual therapeutic approach. Increased urinary excretion of prostaglandins was corrected by indomethacin, with resultant increased nephrogenous cyclic AMP and partial improvement in the concentrating defect. The addition of a thiazide restored urinary concentration. These results add clinical support to the conception of the important role of prostaglandins in the mechanism of post-obstructive hyposthenuria. This therapeutic regimen is advocated for prolonged post-obstructive concentrating defect. Topics: Child, Preschool; Dinoprost; Dinoprostone; Humans; Hydrochlorothiazide; Indomethacin; Kidney Concentrating Ability; Male; Polyuria; Postoperative Complications; Prostaglandins E; Prostaglandins F; Urethral Obstruction	1987

Article

Year

Thenogenology question of the month. Pyometra.

Journal of the American Veterinary Medical Association, 2002, Jan-15, Volume: 220, Issue:2

Topics: Animals; Anti-Bacterial Agents; Dinoprost; Dog Diseases; Dogs; Escherichia coli Infections; Female; Leukocyte Count; Polyuria; Suppuration; Urinalysis; Uterine Diseases

2002

Post-obstructive urinary concentrating defect. A case study in the role of prostaglandins.

Acta paediatrica Scandinavica, 1987, Volume: 76, Issue:6

A child with post-obstructive urinary concentrating defect was studied for the possible pathophysiological role of prostaglandins and an eventual therapeutic approach. Increased urinary excretion of prostaglandins was corrected by indomethacin, with resultant increased nephrogenous cyclic AMP and partial improvement in the concentrating defect. The addition of a thiazide restored urinary concentration. These results add clinical support to the conception of the important role of prostaglandins in the mechanism of post-obstructive hyposthenuria. This therapeutic regimen is advocated for prolonged post-obstructive concentrating defect.

Topics: Child, Preschool; Dinoprost; Dinoprostone; Humans; Hydrochlorothiazide; Indomethacin; Kidney Concentrating Ability; Male; Polyuria; Postoperative Complications; Prostaglandins E; Prostaglandins F; Urethral Obstruction

1987