Page last updated: 2024-08-17

dinitrofluorobenzene and Dermatitis

dinitrofluorobenzene has been researched along with Dermatitis in 10 studies

Research

Studies (10)

TimeframeStudies, this research(%)All Research%
pre-19902 (20.00)18.7374
1990's1 (10.00)18.2507
2000's4 (40.00)29.6817
2010's2 (20.00)24.3611
2020's1 (10.00)2.80

Authors

AuthorsStudies
Niu, C; Qu, Y; Sun, X; Wang, K; Wu, H1
Chen, CM; Chiang, MF; Hong, JB; Lin, IY; Lin, KI; Lin, SJ; Liu, FT; Wu, SY; Yang, SY; Ying, HY1
Choi, IY; Hong, SH; Jeon, YD; Jung, HJ; Kee, JY; Kim, BS; Kim, DS; Kim, HM; Kim, MC; Kim, SG; Kim, SJ; Um, JY1
BLAND, H; TEMPS, W1
BAER, H; BOWSER, RT; KIRSCHSTEIN, RL1
Engeman, T; Fairchild, RL; Gorbachev, AV; Kish, DD1
Bae, SJ; Katayama, I; Lee, JB; Shimizu, K; Takenaka, M; Tanaka, Y1
Dunham, A; HogenEsch, H; Renninger, M; Seymour, R; Sundberg, JP1
Ando, T; Goto, H; Ishiguro, K; Maeda, O; Niwa, Y; Ohmiya, N1
Goebeler, M; Henseleit, U; Roth, J; Sorg, C; Steinbrink, K; Sunderkötter, C1

Other Studies

10 other study(ies) available for dinitrofluorobenzene and Dermatitis

ArticleYear
Selective activation of TRPA1 ion channels by nitrobenzene skin sensitizers DNFB and DNCB.
    The Journal of biological chemistry, 2022, Volume: 298, Issue:2

    Topics: Dermatitis; Dinitrochlorobenzene; Dinitrofluorobenzene; Humans; Molecular Docking Simulation; Skin; TRPA1 Cation Channel

2022
Inducible deletion of the Blimp-1 gene in adult epidermis causes granulocyte-dominated chronic skin inflammation in mice.
    Proceedings of the National Academy of Sciences of the United States of America, 2013, Apr-16, Volume: 110, Issue:16

    Topics: Animals; Cytokines; Dermatitis; Dinitrofluorobenzene; Epidermis; Flow Cytometry; Fluorescent Antibody Technique; Gene Deletion; Granulocyte Colony-Stimulating Factor; Immunoblotting; Keratinocytes; Macrophages; Mice; Neutrophil Infiltration; Positive Regulatory Domain I-Binding Factor 1; Real-Time Polymerase Chain Reaction; Transcription Factors

2013
Insamhodo-tang, a traditional Korean medicine, regulates mast cell-mediated allergic inflammation in vivo and in vitro.
    Journal of ethnopharmacology, 2011, Mar-24, Volume: 134, Issue:2

    Topics: Anaphylaxis; Animals; Anti-Inflammatory Agents; Calcimycin; Dermatitis; Dinitrofluorobenzene; Edema; Humans; Hypersensitivity; Inflammation; Inflammation Mediators; Ionophores; Male; Mast Cells; Medicine, Korean Traditional; Mice; Mice, Inbred BALB C; Mice, Inbred ICR; p-Methoxy-N-methylphenethylamine; Phytotherapy; Plant Extracts

2011
[A case of severe damage of the skin and deeper tissue, subsequent hepatopathy, caused by a single local action of dinitrofluorobenzene].
    Zeitschrift fur Haut- und Geschlechtskrankheiten, 1953, Nov-01, Volume: 15, Issue:9

    Topics: Dermatitis; Dermatitis, Contact; Dinitrofluorobenzene; Humans; Liver; Liver Diseases; Nitrobenzenes

1953
CONTACT SENSITIVITY IN RHESUS MONKEYS TO POISON IVY EXTRACTS AND FLUORODINITROBENZENE.
    Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.), 1964, Volume: 117

    Topics: Animals; Blood Cell Count; Dermatitis; Dermatitis, Contact; Dermatitis, Toxicodendron; Desensitization, Immunologic; Dinitrofluorobenzene; Haplorhini; Injections, Intramuscular; Macaca mulatta; Nitrobenzenes; Pathology; Research; Skin Tests; Toxicodendron; Toxicology

1964
The intensity of neutrophil infiltration controls the number of antigen-primed CD8 T cells recruited into cutaneous antigen challenge sites.
    Journal of leukocyte biology, 2004, Volume: 76, Issue:5

    Topics: Adaptation, Physiological; Animals; Antigen Presentation; Antigens; CD8-Positive T-Lymphocytes; Chemokine CXCL1; Chemokines, CXC; Dermatitis; Dinitrofluorobenzene; Dose-Response Relationship, Drug; Female; Haptens; Immunity, Innate; Intercellular Signaling Peptides and Proteins; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Neutrophil Infiltration; Neutrophils; Oxazolone; Skin

2004
Topical glucocorticoid augments scratching behaviour in dinitrofluorobenzene-sensitized mice by the induction of substance P.
    Experimental dermatology, 2004, Volume: 13, Issue:12

    Topics: Administration, Topical; Animals; Brain; Cytokines; Dermatitis; Dinitrofluorobenzene; Female; Glucocorticoids; Interleukin-1; Interleukin-2; Interleukin-3; Mice; Mice, Inbred BALB C; Nitric Oxide Synthase; Nitric Oxide Synthase Type II; Protein Precursors; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Substance P; Tachykinins; Tumor Necrosis Factor-alpha

2004
Expression of chitinase-like proteins in the skin of chronic proliferative dermatitis (cpdm/cpdm) mice.
    Experimental dermatology, 2006, Volume: 15, Issue:10

    Topics: Animals; beta-N-Acetylhexosaminidases; Chronic Disease; Dermatitis; Dermatitis, Contact; Dinitrofluorobenzene; Disease Models, Animal; Female; Gene Expression Regulation, Enzymologic; Interleukin-4; Lectins; Macrophages; Male; Mast Cells; Mice; Mice, Inbred C57BL; Mice, Mutant Strains; RNA, Messenger; Skin

2006
Acetate inhibits NFAT activation in T cells via importin beta1 interference.
    European journal of immunology, 2007, Volume: 37, Issue:8

    Topics: Acetic Acid; Animals; Anti-Inflammatory Agents; beta Karyopherins; Colitis; Dermatitis; Dinitrofluorobenzene; Enzyme Activation; Fatty Acids, Volatile; Female; Humans; Interleukin-2; Jurkat Cells; Mice; Mice, Inbred BALB C; NF-kappa B; NFATC Transcription Factors; Protein Transport; T-Lymphocytes; Trinitrobenzenesulfonic Acid

2007
Expression of murine VCAM-1 in vitro and in different models of inflammation in vivo: correlation with immigration of monocytes.
    Experimental dermatology, 1995, Volume: 4, Issue:5

    Topics: Animals; Cell Adhesion; Cell Adhesion Molecules; Cell Line; Cell Movement; Dermatitis; Dermatitis, Allergic Contact; Dinitrofluorobenzene; Endothelium, Vascular; Eye Burns; Gene Expression Regulation; Genetics; Inflammation; Interleukin-4; Leishmania major; Leishmaniasis, Cutaneous; Lipopolysaccharides; Mice; Mice, Inbred BALB C; Monocytes; Recombinant Proteins; Tumor Cells, Cultured; Tumor Necrosis Factor-alpha; Vascular Cell Adhesion Molecule-1

1995