dinitrofluorobenzene has been researched along with Acute Disease in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 1 (16.67) | 18.2507 |
| 2000's | 3 (50.00) | 29.6817 |
| 2010's | 1 (16.67) | 24.3611 |
| 2020's | 1 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Chen, D; Jia, J; Li, H; Lin, Y; Liu, J; Mo, X; Yan, F; Ye, S; Zhang, Y | 1 |
| Christensen, AD; Haase, C; Kvist, PH; Skov, S | 1 |
| Fox-Robichaud, AE; Scott, JR | 1 |
| Asano, M; Homma, I; Iwakura, Y; Iwase, M; Komiyama, Y; Nakae, S; Nambu, A; Sekikawa, K; Sudo, K | 1 |
| Brumbaugh, K; Flint, MS; Salmen, RR; Tinkle, SS | 1 |
| Dhabhar, FS; McEwen, BS | 1 |
6 other study(ies) available for dinitrofluorobenzene and Acute Disease
| Article | Year |
|---|---|
Role of YAP-related T cell imbalance and epidermal keratinocyte dysfunction in the pathogenesis of atopic dermatitis.
Topics: Acute Disease; Adaptor Proteins, Signal Transducing; Adolescent; Adult; Animals; Apoptosis; Cell Differentiation; Child; Chronic Disease; Dermatitis, Atopic; Dinitrofluorobenzene; Disease Models, Animal; Down-Regulation; Epidermis; Female; HaCaT Cells; Humans; Keratinocytes; Male; Mice; T-Lymphocytes, Regulatory; Th1 Cells; Th17 Cells; Th2 Cells; Transcription Factors; Up-Regulation; YAP-Signaling Proteins; Young Adult | 2021 |
Depletion of regulatory T cells in a hapten-induced inflammation model results in prolonged and increased inflammation driven by T cells.
Topics: Abatacept; Acute Disease; Animals; Antibodies, Monoclonal; CD8-Positive T-Lymphocytes; Chronic Disease; Cytokines; Dermatitis, Contact; Dinitrofluorobenzene; Disease Models, Animal; Female; Haptoglobins; Humans; Immunoconjugates; Inflammation; Interleukin-2 Receptor alpha Subunit; Lymph Nodes; Lymphocyte Depletion; Macrophages; Mice; Mice, Inbred BALB C; Neutrophils; Serum Amyloid A Protein; T-Lymphocytes, Regulatory | 2015 |
Hepatic leukocyte recruitment in a model of acute colitis.
Topics: Acute Disease; Administration, Rectal; Animals; Antigens, CD; Bile Ducts; Cell Adhesion; Cell Movement; Colitis; Dinitrofluorobenzene; Integrin alpha4; Leukocytes; Liver; Male; Mice; Mice, Inbred C57BL; P-Selectin; Time Factors; Venules | 2002 |
Antigen-specific T cell sensitization is impaired in IL-17-deficient mice, causing suppression of allergic cellular and humoral responses.
Topics: Acute Disease; Animals; Antibody Formation; B-Lymphocytes; Bronchial Hyperreactivity; CD4-Positive T-Lymphocytes; Cells, Cultured; Coculture Techniques; Dendritic Cells; Dermatitis, Allergic Contact; Dinitrofluorobenzene; Female; Graft vs Host Reaction; Haptens; Hypersensitivity, Delayed; Immunity, Cellular; Interleukin-17; Lymphocyte Activation; Lymphocyte Cooperation; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Mitogens; Models, Animal; Nickel; Picryl Chloride; Specific Pathogen-Free Organisms; Spleen; T-Lymphocyte Subsets; Tumor Necrosis Factor-alpha | 2002 |
Acute stress modulates the irritant component of sensitizers in allergic contact dermatitis: implications for exposure assessment.
Topics: Acute Disease; Administration, Topical; Animals; Dermatitis, Allergic Contact; Dicyclohexylcarbodiimide; Dinitrofluorobenzene; Disease Models, Animal; Dose-Response Relationship, Immunologic; Ear, External; Edema; Irritants; Male; Mice; Mice, Inbred BALB C; Oxazolone; Restraint, Physical; Stress, Physiological | 2003 |
Stress-induced enhancement of antigen-specific cell-mediated immunity.
Topics: Acute Disease; Animals; Antigens; Cell Count; Dermatitis, Allergic Contact; Dinitrofluorobenzene; Hypersensitivity, Delayed; Immunity, Cellular; Leukocytes; Male; Models, Biological; Rats; Rats, Sprague-Dawley; Stress, Physiological | 1996 |