dihydroxyphenylalanine has been researched along with Anterior Circulation Transient Ischemic Attack in 4 studies
Dihydroxyphenylalanine: A beta-hydroxylated derivative of phenylalanine. The D-form of dihydroxyphenylalanine has less physiologic activity than the L-form and is commonly used experimentally to determine whether the pharmacological effects of LEVODOPA are stereospecific.
dopa : A hydroxyphenylalanine carrying hydroxy substituents at positions 3 and 4 of the benzene ring.
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 2 (50.00) | 18.7374 |
| 1990's | 1 (25.00) | 18.2507 |
| 2000's | 1 (25.00) | 29.6817 |
| 2010's | 0 (0.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Misu, Y | 2 |
| Furukawa, N | 2 |
| Arai, N | 2 |
| Miyamae, T | 2 |
| Goshima, Y | 2 |
| Ohshima, E | 1 |
| Suzuki, F | 1 |
| Fujita, K | 1 |
| Calderini, G | 1 |
| Carlsson, A | 1 |
| Nordström, CH | 1 |
| Miyauchi, Y | 1 |
| Wieloch, T | 1 |
| Lindvall, O | 1 |
4 other studies available for dihydroxyphenylalanine and Anterior Circulation Transient Ischemic Attack
| Article | Year |
|---|---|
[Is endogenously released DOPA itself an upstream factor for increase in glutamate release and delayed neuronal cell death induced by transient ischemia in rats?].
Topics: Animals; Aromatic Amino Acid Decarboxylase Inhibitors; Cell Death; Corpus Striatum; Dihydroxyphenyla | 1998 |
Endogenously released DOPA is a causal factor for glutamate release and resultant delayed neuronal cell death by transient ischemia in rat striata.
Topics: Animals; Cell Death; Corpus Striatum; Dihydroxyphenylalanine; Dopamine; Enzyme Inhibitors; Glutamic | 2001 |
Influence of transient ischemia on monoamine metabolism in the rat brain during nitrous oxide and phenobarbitone anaesthesia.
Topics: 5-Hydroxytryptophan; Anesthesia, Inhalation; Anesthesia, Intravenous; Animals; Brain; Catecholamines | 1978 |
Noradrenaline metabolism in neocortex and hippocampus following transient forebrain ischemia in rats: relation to development of selective neuronal necrosis.
Topics: Animals; Cerebral Cortex; Diencephalon; Dihydroxyphenylalanine; Hippocampus; Ischemic Attack, Transi | 1989 |