Compounds > dihydrotachysterol

dihydrotachysterol and Myocardial-Infarction

dihydrotachysterol has been researched along with Myocardial-Infarction* in 7 studies

Reviews

1 review(s) available for dihydrotachysterol and Myocardial-Infarction

Article	Year
EXPERIMENTAL CARDIAC NECROSIS. Biochemical clinics, 1963, Volume: 2 Topics: Chlorides; Desoxycorticosterone; Dihydrotachysterol; Heart; Humans; Hypokalemia; Isoproterenol; Magnesium; Metabolism; Myocardial Infarction; Necrosis; Pathology; Potassium; Potassium Deficiency; Research; Sodium	1963

Other Studies

6 other study(ies) available for dihydrotachysterol and Myocardial-Infarction

Article	Year
[Key-role of intracellular calcium overload in acute necrosis of the myocardium. Cardioprotection with verapamil]. Minerva medica, 1975, May-23, Volume: 66, Issue:38 Ca ions are highly cardiotoxic if their influx into the myocardial fibres becomes abundant. The intracellular Ca overload initiates a deleterious high-energy phosphate deficiency by excessive activation of Ca-dependent intracellular ATPases and by impairing the phosphorylating capacity of mitochondria. This Ca-induced high-energy phosphate exhaustion is a crucial point in the etiology of the myocardial fibre necroses produced in rats by large doses of beta-adrenergic catecholamines, particularly isoproterenol, or by a number of other cardiotoxic agents. Accordinly, the myocardium is sensitized to necrotization by factors which favour Ca overload (dihydrotachysterol, 9alpha-flourocortisol acetate, NaH2PO4). Conversely, the structural integrity of the hearts can be protected by any substance or procedure which prevents an excessive intracellular Ca accumulation, particularly by inhibitors of the transmembrane Ca influx, such as verapamil, D 600 or prenylamine. Topics: Adenosine Triphosphate; Animals; Calcium; Catecholamines; Dihydrotachysterol; Fludrocortisone; Heart; Isoproterenol; Mitochondria; Myocardial Infarction; Myocardium; Necrosis; Phosphates; Prenylamine; Rats; Verapamil	1975
[Production of cardiac necrosis without coronary obstruction in the rat by a thrombogenic diet]. Pathologie-biologie, 1972, Volume: 20, Issue:3 Topics: Animals; Bile Acids and Salts; Cholesterol; Diet, Atherogenic; Dietary Fats; Dihydrotachysterol; Fludrocortisone; Heart Diseases; Myocardial Infarction; Necrosis; Nephrocalcinosis; Phosphates; Propylthiouracil; Rats; Stress, Physiological; Thrombosis	1972
Electrocardiogram of the month: the significance of QT, QTc, QaT, and QaTc. Connecticut medicine, 1972, Volume: 36, Issue:5 Topics: Calcium; Dihydrotachysterol; Electrocardiography; Heart; Humans; Hypocalcemia; Myocardial Infarction; Quinidine	1972
Stress and aging. Journal of the American Geriatrics Society, 1970, Volume: 18, Issue:9 Topics: Adaptation, Physiological; Aging; Anabolic Agents; Animals; Calcium Metabolism Disorders; Chlorides; Dihydrotachysterol; Disease Models, Animal; Female; Humans; Magnesium; Myocardial Infarction; Necrosis; Potassium Chloride; Progeria; Rats; Sodium; Steroids; Stress, Physiological	1970
Histochemically demonstrable phosphorylase as an early index of anoxic myocardial damage. Experientia, 1964, Jul-15, Volume: 20, Issue:7 Topics: Animals; Antimetabolites; Dihydrotachysterol; Glucosyltransferases; Histocytochemistry; In Vitro Techniques; Methoxamine; Myocardial Infarction; Rats; Steroids; Vitamin K Deficiency	1964
[Induction and prevention of cardiac necroses in the dihydrotachysterin-sensitized rat]. Virchows Archiv fur pathologische Anatomie und Physiologie und fur klinische Medizin, 1959, Volume: 332, Issue:2 Topics: Animals; Dihydrotachysterol; Myocardial Infarction; Necrosis; Rats	1959