Page last updated: 2024-09-03

dihydroethidium and Reperfusion Injury

dihydroethidium has been researched along with Reperfusion Injury in 8 studies

Research

Studies (8)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's6 (75.00)29.6817
2010's2 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chan, PH; Kim, GW; Noshita, N; Sugawara, T1
Chan, PH; Kim, GW; Kondo, T; Noshita, N1
Reymann, KG; Röhnert, P; Schröder, UH; Striggow, F; Täger, M; Ziabreva, I1
Berne, JP; Lauzier, B; Rochette, L; Vergely, C1
Gottlieb, RA; Gustafsson, AB; Molkentin, JD; Sayen, MR; Sussman, MA1
Chan, PH; Sugawara, T; Yu, F1
Busseuil, D; Demaison, L; Martin, C; Oudot, A; Rochette, L; Vergely, C1
Baudry, N; Laemmel, E; Vicaut, E1

Other Studies

8 other study(ies) available for dihydroethidium and Reperfusion Injury

ArticleYear
Early decrease in dna repair proteins, Ku70 and Ku86, and subsequent DNA fragmentation after transient focal cerebral ischemia in mice.
    Stroke, 2001, Volume: 32, Issue:6

    Topics: Animals; Antigens, Nuclear; Blotting, Western; Brain; Cerebral Infarction; Disease Models, Animal; DNA; DNA Fragmentation; DNA Helicases; DNA Repair; DNA-Binding Proteins; Electrophoresis, Agar Gel; Immunohistochemistry; In Situ Nick-End Labeling; Ischemic Attack, Transient; Ku Autoantigen; Male; Mice; Nuclear Proteins; Oxidation-Reduction; Oxidative Stress; Phenanthridines; Reperfusion Injury

2001
Manganese superoxide dismutase deficiency exacerbates cerebral infarction after focal cerebral ischemia/reperfusion in mice: implications for the production and role of superoxide radicals.
    Stroke, 2002, Volume: 33, Issue:3

    Topics: Animals; Cerebral Infarction; Cytoprotection; Disease Models, Animal; Ethidium; Heterozygote; Homozygote; Male; Mice; Mice, Knockout; Mice, Mutant Strains; Mitochondria; Oxidative Stress; Phenanthridines; Reperfusion Injury; Superoxide Dismutase; Superoxide Dismutase-1; Superoxides; Survival Rate

2002
Insufficient endogenous redox buffer capacity may underlie neuronal vulnerability to cerebral ischemia and reperfusion.
    Journal of neuroscience research, 2012, Volume: 90, Issue:1

    Topics: Animals; Brain Ischemia; Cell Death; Disease Models, Animal; Ethidium; Fluoresceins; Gerbillinae; Glial Fibrillary Acidic Protein; Glucose; Glycoproteins; Hippocampus; Hypoxia; Lectins; Neurons; Neuroprotective Agents; Organ Culture Techniques; Oxidation-Reduction; Rats; Rats, Wistar; Reactive Oxygen Species; Reperfusion Injury; Rhodamines; Sulfhydryl Compounds; Thioctic Acid; Versicans

2012
Carbon monoxide protects against ischemia-reperfusion injury in vitro via antioxidant properties.
    Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 2012, Volume: 29, Issue:3-4

    Topics: Animals; Antioxidants; Carbon Monoxide; Cardiotonic Agents; Ethidium; Heart; In Vitro Techniques; L-Lactate Dehydrogenase; Male; Myocardial Contraction; Organometallic Compounds; Oxidative Stress; Rats; Rats, Wistar; Reperfusion Injury; Superoxides; Time Factors; Ventricular Function, Left

2012
Calcineurin transgenic mice have mitochondrial dysfunction and elevated superoxide production.
    American journal of physiology. Cell physiology, 2003, Volume: 284, Issue:2

    Topics: Adenosine Diphosphate; Animals; Animals, Newborn; Calcineurin; Cardiomegaly; Cell Respiration; Cells, Cultured; Electron Transport; Ethidium; Genetic Vectors; Heart Failure; Mice; Mice, Transgenic; Mitochondria; Mitochondrial Diseases; Myocardium; Myocytes, Cardiac; Oxidative Stress; Reperfusion Injury; Superoxides

2003
Treatment with dihydroethidium reduces infarct size after transient focal cerebral ischemia in mice.
    Brain research, 2003, Jul-18, Volume: 978, Issue:1-2

    Topics: Animals; Body Temperature; Brain Edema; Brain Mapping; Cerebral Infarction; Disease Models, Animal; Dose-Response Relationship, Drug; Ethidium; Infarction, Middle Cerebral Artery; Ischemic Attack, Transient; Male; Mice; Mice, Inbred C57BL; Neuroprotective Agents; Reperfusion Injury; Time Factors

2003
NADPH oxidases are in part responsible for increased cardiovascular superoxide production during aging.
    Free radical biology & medicine, 2006, Jun-15, Volume: 40, Issue:12

    Topics: Adaptation, Physiological; Age Factors; Aging; Animals; Aorta; Cardiovascular System; Electron Spin Resonance Spectroscopy; Endothelial Cells; Ethidium; Heart; Hemodynamics; In Vitro Techniques; Male; Myocardial Reperfusion Injury; Myocardium; NADPH Oxidases; Oxidative Stress; Pyrrolidines; Rats; Rats, Wistar; Receptor, Angiotensin, Type 1; Recovery of Function; Reperfusion Injury; Superoxides

2006
In vivo reactive oxygen species production induced by ischemia in muscle arterioles of mice: involvement of xanthine oxidase and mitochondria.
    American journal of physiology. Heart and circulatory physiology, 2008, Volume: 294, Issue:2

    Topics: Animals; Arterioles; Blood Pressure; Ethidium; Fluorescent Dyes; Ischemia; Mice; Mice, Inbred BALB C; Microcirculation; Mitochondria, Muscle; Muscle, Skeletal; Reactive Oxygen Species; Regional Blood Flow; Reperfusion Injury; Superoxide Dismutase; Xanthine Oxidase

2008