Page last updated: 2024-09-03

dihydroethidium and Injury, Ischemia-Reperfusion

dihydroethidium has been researched along with Injury, Ischemia-Reperfusion in 8 studies

Research

Studies (8)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's6 (75.00)29.6817
2010's2 (25.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
Chan, PH; Kim, GW; Noshita, N; Sugawara, T1
Chan, PH; Kim, GW; Kondo, T; Noshita, N1
Reymann, KG; Röhnert, P; Schröder, UH; Striggow, F; Täger, M; Ziabreva, I1
Berne, JP; Lauzier, B; Rochette, L; Vergely, C1
Gottlieb, RA; Gustafsson, AB; Molkentin, JD; Sayen, MR; Sussman, MA1
Chan, PH; Sugawara, T; Yu, F1
Busseuil, D; Demaison, L; Martin, C; Oudot, A; Rochette, L; Vergely, C1
Baudry, N; Laemmel, E; Vicaut, E1

Other Studies

8 other study(ies) available for dihydroethidium and Injury, Ischemia-Reperfusion

ArticleYear
Early decrease in dna repair proteins, Ku70 and Ku86, and subsequent DNA fragmentation after transient focal cerebral ischemia in mice.
    Stroke, 2001, Volume: 32, Issue:6

    Topics: Animals; Antigens, Nuclear; Blotting, Western; Brain; Cerebral Infarction; Disease Models, Animal; DNA; DNA Fragmentation; DNA Helicases; DNA Repair; DNA-Binding Proteins; Electrophoresis, Agar Gel; Immunohistochemistry; In Situ Nick-End Labeling; Ischemic Attack, Transient; Ku Autoantigen; Male; Mice; Nuclear Proteins; Oxidation-Reduction; Oxidative Stress; Phenanthridines; Reperfusion Injury

2001
Manganese superoxide dismutase deficiency exacerbates cerebral infarction after focal cerebral ischemia/reperfusion in mice: implications for the production and role of superoxide radicals.
    Stroke, 2002, Volume: 33, Issue:3

    Topics: Animals; Cerebral Infarction; Cytoprotection; Disease Models, Animal; Ethidium; Heterozygote; Homozygote; Male; Mice; Mice, Knockout; Mice, Mutant Strains; Mitochondria; Oxidative Stress; Phenanthridines; Reperfusion Injury; Superoxide Dismutase; Superoxide Dismutase-1; Superoxides; Survival Rate

2002
Insufficient endogenous redox buffer capacity may underlie neuronal vulnerability to cerebral ischemia and reperfusion.
    Journal of neuroscience research, 2012, Volume: 90, Issue:1

    Topics: Animals; Brain Ischemia; Cell Death; Disease Models, Animal; Ethidium; Fluoresceins; Gerbillinae; Glial Fibrillary Acidic Protein; Glucose; Glycoproteins; Hippocampus; Hypoxia; Lectins; Neurons; Neuroprotective Agents; Organ Culture Techniques; Oxidation-Reduction; Rats; Rats, Wistar; Reactive Oxygen Species; Reperfusion Injury; Rhodamines; Sulfhydryl Compounds; Thioctic Acid; Versicans

2012
Carbon monoxide protects against ischemia-reperfusion injury in vitro via antioxidant properties.
    Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 2012, Volume: 29, Issue:3-4

    Topics: Animals; Antioxidants; Carbon Monoxide; Cardiotonic Agents; Ethidium; Heart; In Vitro Techniques; L-Lactate Dehydrogenase; Male; Myocardial Contraction; Organometallic Compounds; Oxidative Stress; Rats; Rats, Wistar; Reperfusion Injury; Superoxides; Time Factors; Ventricular Function, Left

2012
Calcineurin transgenic mice have mitochondrial dysfunction and elevated superoxide production.
    American journal of physiology. Cell physiology, 2003, Volume: 284, Issue:2

    Topics: Adenosine Diphosphate; Animals; Animals, Newborn; Calcineurin; Cardiomegaly; Cell Respiration; Cells, Cultured; Electron Transport; Ethidium; Genetic Vectors; Heart Failure; Mice; Mice, Transgenic; Mitochondria; Mitochondrial Diseases; Myocardium; Myocytes, Cardiac; Oxidative Stress; Reperfusion Injury; Superoxides

2003
Treatment with dihydroethidium reduces infarct size after transient focal cerebral ischemia in mice.
    Brain research, 2003, Jul-18, Volume: 978, Issue:1-2

    Topics: Animals; Body Temperature; Brain Edema; Brain Mapping; Cerebral Infarction; Disease Models, Animal; Dose-Response Relationship, Drug; Ethidium; Infarction, Middle Cerebral Artery; Ischemic Attack, Transient; Male; Mice; Mice, Inbred C57BL; Neuroprotective Agents; Reperfusion Injury; Time Factors

2003
NADPH oxidases are in part responsible for increased cardiovascular superoxide production during aging.
    Free radical biology & medicine, 2006, Jun-15, Volume: 40, Issue:12

    Topics: Adaptation, Physiological; Age Factors; Aging; Animals; Aorta; Cardiovascular System; Electron Spin Resonance Spectroscopy; Endothelial Cells; Ethidium; Heart; Hemodynamics; In Vitro Techniques; Male; Myocardial Reperfusion Injury; Myocardium; NADPH Oxidases; Oxidative Stress; Pyrrolidines; Rats; Rats, Wistar; Receptor, Angiotensin, Type 1; Recovery of Function; Reperfusion Injury; Superoxides

2006
In vivo reactive oxygen species production induced by ischemia in muscle arterioles of mice: involvement of xanthine oxidase and mitochondria.
    American journal of physiology. Heart and circulatory physiology, 2008, Volume: 294, Issue:2

    Topics: Animals; Arterioles; Blood Pressure; Ethidium; Fluorescent Dyes; Ischemia; Mice; Mice, Inbred BALB C; Microcirculation; Mitochondria, Muscle; Muscle, Skeletal; Reactive Oxygen Species; Regional Blood Flow; Reperfusion Injury; Superoxide Dismutase; Xanthine Oxidase

2008