dihydroergotoxine and Shock--Cardiogenic

dihydroergotoxine has been researched along with Shock--Cardiogenic* in 2 studies

Other Studies

2 other study(ies) available for dihydroergotoxine and Shock--Cardiogenic

Article	Year
[Drug therapy of shock]. Fortschritte der Medizin, 1978, Mar-30, Volume: 96, Issue:12 Shock is to define by clinical symptoms, measurable haemodynamic and metabolic changes, clotting failure and disorders of fluid, electrolyte and acid-base balance. It is essential to remove hypovolemia, acidosis, cardiovascular failure, pulmonary and renal insufficiency. Quite a lot of proved and controversial pharmacological substances are placed at physician's disposal. However, the mortality of cardiogenic shock and gram-negative septic shock is more than 80% respectively 30%, often caused by shock lung or irreversible injury to the kidneys. Shock due to depletion of extracellular fluid, poisoning and acute pancreatitis require specific treatment. Topics: Bicarbonates; Dehydration; Dihydroergotoxine; Furosemide; Glucagon; Humans; Methylprednisolone; Peritoneal Dialysis; Plasma Substitutes; Shock; Shock, Cardiogenic; Shock, Septic; Spironolactone; Sympathomimetics; Theophylline	1978
[The effects of dihydroergo-corine,-cryptine and-cristine on Haemodynamics in experimental cardiogenic shock]. Praktische Anasthesie, Wiederbelebung und Intensivtherapie, 1976, Volume: 11, Issue:2 Experimental results demonstrate, that serial ligatures of the side brances of the anterior descending and circumflex artery in the openchest dog produce a hemodynamic and electrocardiographic criteria fullfilling cardiogrenic shock. The application of the alpha-receptor-blocking agens Hydergin is followed by a statistically significant increase of the mean aortic pressure, the left ventricular systolic pressure and the mean blood flow in the femoral artery. The left ventricular enddiastolic pressure in the left ventricular enddiastolic pressure in the left ventricle decreases statistically not significant, dp/dtmax is unchanged. Hydergin effects a slope of the increased peripheral vascular ristance; if the peripheral vascular risistance is lowered during cardiogenic shock, there will not be a further lowering after treatment. The results show, that Hydergin causes a dilation of peripheral vasoconstricted vessels without unwanted hemodynamic side effects. Topics: Animals; Aorta, Thoracic; Blood Pressure; Blood Volume; Dihydroergotoxine; Dogs; Ergoloid Mesylates; Femoral Artery; Shock, Cardiogenic; Vasodilator Agents	1976

Article

Year

Fortschritte der Medizin, 1978, Mar-30, Volume: 96, Issue:12

Shock is to define by clinical symptoms, measurable haemodynamic and metabolic changes, clotting failure and disorders of fluid, electrolyte and acid-base balance. It is essential to remove hypovolemia, acidosis, cardiovascular failure, pulmonary and renal insufficiency. Quite a lot of proved and controversial pharmacological substances are placed at physician's disposal. However, the mortality of cardiogenic shock and gram-negative septic shock is more than 80% respectively 30%, often caused by shock lung or irreversible injury to the kidneys. Shock due to depletion of extracellular fluid, poisoning and acute pancreatitis require specific treatment.

Topics: Bicarbonates; Dehydration; Dihydroergotoxine; Furosemide; Glucagon; Humans; Methylprednisolone; Peritoneal Dialysis; Plasma Substitutes; Shock; Shock, Cardiogenic; Shock, Septic; Spironolactone; Sympathomimetics; Theophylline

1978

[The effects of dihydroergo-corine,-cryptine and-cristine on Haemodynamics in experimental cardiogenic shock].

Praktische Anasthesie, Wiederbelebung und Intensivtherapie, 1976, Volume: 11, Issue:2

Experimental results demonstrate, that serial ligatures of the side brances of the anterior descending and circumflex artery in the openchest dog produce a hemodynamic and electrocardiographic criteria fullfilling cardiogrenic shock. The application of the alpha-receptor-blocking agens Hydergin is followed by a statistically significant increase of the mean aortic pressure, the left ventricular systolic pressure and the mean blood flow in the femoral artery. The left ventricular enddiastolic pressure in the left ventricular enddiastolic pressure in the left ventricle decreases statistically not significant, dp/dtmax is unchanged. Hydergin effects a slope of the increased peripheral vascular ristance; if the peripheral vascular risistance is lowered during cardiogenic shock, there will not be a further lowering after treatment. The results show, that Hydergin causes a dilation of peripheral vasoconstricted vessels without unwanted hemodynamic side effects.

Topics: Animals; Aorta, Thoracic; Blood Pressure; Blood Volume; Dihydroergotoxine; Dogs; Ergoloid Mesylates; Femoral Artery; Shock, Cardiogenic; Vasodilator Agents

1976