dihydrocytochalasin b has been researched along with Adenocarcinoma in 1 studies
dihydrocytochalasin B: inhibits cell motility in many eucaryotic cells
Adenocarcinoma: A malignant epithelial tumor with a glandular organization.
Excerpt | Relevance | Reference |
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"Although tolerance to tetraploidization, an intermediate state that further exacerbates CIN, is frequently mediated by TP53 dysfunction, we find that some genome-doubled tumours retain wild-type TP53." | 1.46 | Cyclin D mediates tolerance of genome-doubling in cancers with functional p53. ( Begum, J; Cerveira, J; Crockford, A; Cuomo, ME; Dewhurst, SM; Encheva, V; Grönroos, E; McGranahan, N; Patel, H; Purewal, S; Renshaw, MJ; Snijders, AP; Swanton, C; Zalmas, LP, 2017) |
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Crockford, A | 1 |
Zalmas, LP | 1 |
Grönroos, E | 1 |
Dewhurst, SM | 1 |
McGranahan, N | 1 |
Cuomo, ME | 1 |
Encheva, V | 1 |
Snijders, AP | 1 |
Begum, J | 1 |
Purewal, S | 1 |
Cerveira, J | 1 |
Patel, H | 1 |
Renshaw, MJ | 1 |
Swanton, C | 1 |
1 other study available for dihydrocytochalasin b and Adenocarcinoma
Article | Year |
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Cyclin D mediates tolerance of genome-doubling in cancers with functional p53.
Topics: Adenocarcinoma; Aminopyridines; Benzimidazoles; Cell Line, Tumor; Colorectal Neoplasms; Cyclin C; Cy | 2017 |