diclofenac has been researched along with Hyperplasia in 5 studies
Diclofenac: A non-steroidal anti-inflammatory agent (NSAID) with antipyretic and analgesic actions. It is primarily available as the sodium salt.
diclofenac : A monocarboxylic acid consisting of phenylacetic acid having a (2,6-dichlorophenyl)amino group at the 2-position.
Hyperplasia: An increase in the number of cells in a tissue or organ without tumor formation. It differs from HYPERTROPHY, which is an increase in bulk without an increase in the number of cells.
| Excerpt | Relevance | Reference |
|---|---|---|
| "Creation of an autologous arteriovenous fistula (AVF) for vascular access in haemodialysis is the modality of choice." | 5.42 | Improving arteriovenous fistula patency: Transdermal delivery of diclofenac reduces cannulation-dependent neointimal hyperplasia via AMPK activation. ( Aitken, E; Coats, P; Currie, S; Ewart, MA; Jackson, A; Kingsmore, D; MacAskill, MG; MacKenzie, G; Wadsworth, R; Watson, DG, 2015) |
| "Routine rectal administration of 100 mg of diclofenac or indomethacin was demonstrated to be an effective prevention method to prevent post-endoscopic retrograde cholangiopancreatography (ERCP) pancreatitis." | 5.22 | The Incidence and Severity of Post-ERCP Pancreatitis in Patients Receiving Standard Administration of NSAIDs: a Systematic Review and Meta-analysis. ( Chen, Z; Guo, X; Kang, X; Lou, L; Lu, Y; Luo, H; Pan, Y; Zhou, Z, 2022) |
| "Creation of an autologous arteriovenous fistula (AVF) for vascular access in haemodialysis is the modality of choice." | 1.42 | Improving arteriovenous fistula patency: Transdermal delivery of diclofenac reduces cannulation-dependent neointimal hyperplasia via AMPK activation. ( Aitken, E; Coats, P; Currie, S; Ewart, MA; Jackson, A; Kingsmore, D; MacAskill, MG; MacKenzie, G; Wadsworth, R; Watson, DG, 2015) |
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 2 (40.00) | 18.2507 |
| 2000's | 1 (20.00) | 29.6817 |
| 2010's | 1 (20.00) | 24.3611 |
| 2020's | 1 (20.00) | 2.80 |
| Authors | Studies |
|---|---|
| Kang, X | 1 |
| Guo, X | 2 |
| Chen, Z | 1 |
| Zhou, Z | 1 |
| Luo, H | 1 |
| Lu, Y | 1 |
| Lou, L | 1 |
| Pan, Y | 1 |
| MacAskill, MG | 1 |
| Watson, DG | 1 |
| Ewart, MA | 1 |
| Wadsworth, R | 1 |
| Jackson, A | 1 |
| Aitken, E | 1 |
| MacKenzie, G | 1 |
| Kingsmore, D | 1 |
| Currie, S | 1 |
| Coats, P | 1 |
| Kumar, VS | 1 |
| Kumar, DA | 1 |
| Kalaivani, K | 1 |
| Gangadharan, AC | 1 |
| Raju, KV | 1 |
| Thejomoorthy, P | 1 |
| Manohar, BM | 1 |
| Puvanakrishnan, R | 1 |
| Pospísil, M | 1 |
| Vacek, A | 1 |
| Hofer, M | 1 |
| Viklická, S | 1 |
| Pipalová, I | 1 |
| Sandula, J | 1 |
| Sánchez-Pernaute, O | 1 |
| López-Armada, MJ | 1 |
| Hernández, P | 1 |
| Palacios, I | 1 |
| Navarro, F | 1 |
| Martínez, J | 1 |
| Egido, J | 1 |
| Herrero-Beaumont, G | 1 |
| Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
|---|---|---|---|---|---|---|---|
| Rectal Disclofenac Versus Indomethacin for Prevention of Post-ERCP Pancreatitis (DIPPP): A Multicentre, Double-blind, Randomised, Controlled Trial[NCT05947461] | 3,612 participants (Anticipated) | Interventional | 2023-06-01 | Recruiting | |||
| [information is prepared from clinicaltrials.gov, extracted Sep-2024] | |||||||
1 review available for diclofenac and Hyperplasia
| Article | Year |
|---|---|
The Incidence and Severity of Post-ERCP Pancreatitis in Patients Receiving Standard Administration of NSAIDs: a Systematic Review and Meta-analysis.
Topics: Anti-Inflammatory Agents, Non-Steroidal; Cholangiopancreatography, Endoscopic Retrograde; Diclofenac | 2022 |
4 other studies available for diclofenac and Hyperplasia
| Article | Year |
|---|---|
Improving arteriovenous fistula patency: Transdermal delivery of diclofenac reduces cannulation-dependent neointimal hyperplasia via AMPK activation.
Topics: Administration, Cutaneous; AMP-Activated Protein Kinases; Animals; Arteriovenous Fistula; Catheteriz | 2015 |
Optimization of pulsed electromagnetic field therapy for management of arthritis in rats.
Topics: Acid Phosphatase; Animals; Anti-Inflammatory Agents, Non-Steroidal; Arthritis, Experimental; Arthrit | 2005 |
Hyporesponsiveness of murine myeloid progenitor cells to glucan following its repeated administration.
Topics: Adjuvants, Immunologic; Animals; beta-Glucans; Bone Marrow; Colony-Forming Units Assay; Colony-Stimu | 1993 |
Antifibroproliferative effect of tenidap in chronic antigen-induced arthritis.
Topics: Animals; Anti-Inflammatory Agents, Non-Steroidal; Arthritis, Experimental; Cell Division; Cells, Cul | 1997 |