diclofenac has been researched along with Diabetes Insipidus in 2 studies
Diclofenac: A non-steroidal anti-inflammatory agent (NSAID) with antipyretic and analgesic actions. It is primarily available as the sodium salt.
diclofenac : A monocarboxylic acid consisting of phenylacetic acid having a (2,6-dichlorophenyl)amino group at the 2-position.
Diabetes Insipidus: A disease that is characterized by frequent urination, excretion of large amounts of dilute URINE, and excessive THIRST. Etiologies of diabetes insipidus include deficiency of antidiuretic hormone (also known as ADH or VASOPRESSIN) secreted by the NEUROHYPOPHYSIS, impaired KIDNEY response to ADH, and impaired hypothalamic regulation of thirst.
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 1 (50.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 1 (50.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Babina, AV | 1 |
Lavrinenko, VA | 1 |
Shestopalova, LV | 1 |
Ivanova, LN | 1 |
Kato, A | 1 |
Hishida, A | 1 |
Ishibashi, R | 1 |
Nakajima, T | 1 |
Ohura, M | 1 |
Furuya, R | 1 |
Kumagai, H | 1 |
Kimura, M | 1 |
Kaneko, E | 1 |
Trial | Phase | Enrollment | Study Type | Start Date | Status | ||
---|---|---|---|---|---|---|---|
Acute Neuroendocrine Response to Intravenous Infusion of Diclofenac Sodium[NCT05188131] | Phase 4 | 12 participants (Actual) | Interventional | 2021-10-01 | Completed | ||
[information is prepared from clinicaltrials.gov, extracted Sep-2024] |
2 other studies available for diclofenac and Diabetes Insipidus
Article | Year |
---|---|
Morphological characteristics of the inner medullary zone in the kidneys of Brattleboro and Wistar rats during blockade of prostaglandin synthesis.
Topics: Animals; Cell Shape; Diabetes Insipidus; Diclofenac; Epithelium; Extracellular Matrix; Glucuronidase | 2011 |
Nephrogenic diabetes insipidus associated with bilateral ureteral obstruction.
Topics: Adult; Chlorothiazide; Diabetes Insipidus; Diclofenac; Humans; Ileal Neoplasms; Kidney Diseases; Lei | 1994 |