Page last updated: 2024-08-21

dicarbethoxydihydrocollidine and Non-alcoholic Fatty Liver Disease

dicarbethoxydihydrocollidine has been researched along with Non-alcoholic Fatty Liver Disease in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's3 (100.00)24.3611
2020's0 (0.00)2.80

Authors

AuthorsStudies
French, BA; French, SW; Li, J; Liu, H; Tillman, B1
Amstislavskiy, V; Denk, H; Grimm, C; Hache, H; Kashofer, K; Lehrach, H; Osprian, I; Pandey, V; Ralser, M; Starmann, J; Sultan, M; Sültmann, H; Trauner, M; Wierling, C; Yaspo, ML; Zatloukal, K1
Eguchi, A; Feldstein, AE; Font-Burgada, J; Johnson, CD; Karin, M; Povero, D; Wree, A1

Other Studies

3 other study(ies) available for dicarbethoxydihydrocollidine and Non-alcoholic Fatty Liver Disease

ArticleYear
Ufmylation and FATylation pathways are downregulated in human alcoholic and nonalcoholic steatohepatitis, and mice fed DDC, where Mallory-Denk bodies (MDBs) form.
    Experimental and molecular pathology, 2014, Volume: 97, Issue:1

    Topics: Animals; Case-Control Studies; Down-Regulation; Fatty Liver; Gene Expression Regulation; Hepatitis, Alcoholic; Humans; Liver Cirrhosis, Alcoholic; Male; Mallory Bodies; Mice; Mice, Inbred C3H; Non-alcoholic Fatty Liver Disease; Proteins; Pyridines; SNARE Proteins; Ubiquitin-Activating Enzymes; Ubiquitin-Conjugating Enzymes; Ubiquitin-Protein Ligases; Ubiquitins; Vesicular Transport Proteins

2014
Comparative analysis and modeling of the severity of steatohepatitis in DDC-treated mouse strains.
    PloS one, 2014, Volume: 9, Issue:10

    Topics: Animals; Cluster Analysis; Disease Models, Animal; Fatty Liver; Gene Expression Profiling; Gene Expression Regulation; Metabolic Networks and Pathways; Metabolome; Metabolomics; Mice; Non-alcoholic Fatty Liver Disease; Phenotype; Proteomics; Pyridines; Severity of Illness Index; Signal Transduction

2014
Hepatocyte-specific Bid depletion reduces tumor development by suppressing inflammation-related compensatory proliferation.
    Cell death and differentiation, 2015, Volume: 22, Issue:12

    Topics: Animals; BH3 Interacting Domain Death Agonist Protein; Carbon Tetrachloride; Cell Proliferation; Cells, Cultured; Chemical and Drug Induced Liver Injury; Cyclin D1; Diethylnitrosamine; Disease Models, Animal; Hepatocytes; Hydrogen Peroxide; Inflammation; Interleukin-6; Liver; Liver Neoplasms; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Non-alcoholic Fatty Liver Disease; Pyridines; Tumor Necrosis Factor-alpha

2015