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dicarbethoxydihydrocollidine and Liver Neoplasms

dicarbethoxydihydrocollidine has been researched along with Liver Neoplasms in 5 studies

Research

Studies (5)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's1 (20.00)29.6817
2010's3 (60.00)24.3611
2020's1 (20.00)2.80

Authors

AuthorsStudies
Esaki, M; Ishida, T; Miyauchi, Y; Takechi, S1
Baslan, T; Bosbach, B; Chen, CC; Houlihan, SL; Kastenhuber, ER; Lalazar, G; Lowe, SW; Requena, D; Simon, SM; Tian, S; Tschaharganeh, DF; Wilkinson, JE1
Eguchi, A; Feldstein, AE; Font-Burgada, J; Johnson, CD; Karin, M; Povero, D; Wree, A1
Bayan, JA; Birnbaum, MJ; Dang, H; Ding, W; French, BA; French, S; Galicia, VA; He, L; Kanel, G; Rountree, CB; Stiles, BL; Vendryes, C; Wang, KS; Zeng, N1
Bardag-Gorce, F; French, BA; French, SW; Li, J; Oliva, J1

Other Studies

5 other study(ies) available for dicarbethoxydihydrocollidine and Liver Neoplasms

ArticleYear
The effect of dihydropyrazines on lipopolysaccharide-stimulated human hepatoma HepG2 cells via regulating the TLR4-MyD88-mediated NF-κB signaling pathway.
    The Journal of toxicological sciences, 2020, Volume: 45, Issue:7

    Topics: Dicarbethoxydihydrocollidine; Glycation End Products, Advanced; Hep G2 Cells; Humans; Interleukin-1beta; Lipopolysaccharides; Liver Neoplasms; Myeloid Differentiation Factor 88; NF-kappa B; Phosphorylation; Reactive Oxygen Species; Signal Transduction; Toll-Like Receptor 4; Tumor Necrosis Factor-alpha

2020
    Proceedings of the National Academy of Sciences of the United States of America, 2017, 12-12, Volume: 114, Issue:50

    Topics: Adult; Animals; Base Sequence; beta Catenin; Carcinogenesis; Carcinoma, Hepatocellular; Chromosomes, Human, Pair 19; Clustered Regularly Interspaced Short Palindromic Repeats; Cohort Studies; Cyclic AMP-Dependent Protein Kinase Catalytic Subunits; Female; Gene Expression Regulation, Neoplastic; HSP40 Heat-Shock Proteins; Humans; Liver; Liver Neoplasms; Liver Neoplasms, Experimental; Liver Regeneration; Mice; Mice, Inbred C57BL; Oncogene Proteins, Fusion; Pyridines; Sequence Deletion; Young Adult

2017
Hepatocyte-specific Bid depletion reduces tumor development by suppressing inflammation-related compensatory proliferation.
    Cell death and differentiation, 2015, Volume: 22, Issue:12

    Topics: Animals; BH3 Interacting Domain Death Agonist Protein; Carbon Tetrachloride; Cell Proliferation; Cells, Cultured; Chemical and Drug Induced Liver Injury; Cyclin D1; Diethylnitrosamine; Disease Models, Animal; Hepatocytes; Hydrogen Peroxide; Inflammation; Interleukin-6; Liver; Liver Neoplasms; Mice; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Non-alcoholic Fatty Liver Disease; Pyridines; Tumor Necrosis Factor-alpha

2015
Expansion of hepatic tumor progenitor cells in Pten-null mice requires liver injury and is reversed by loss of AKT2.
    Gastroenterology, 2010, Volume: 139, Issue:6

    Topics: Animals; Cell Line; Cell Survival; Cell Transformation, Neoplastic; Chemical and Drug Induced Liver Injury; Gene Expression Regulation, Neoplastic; Intercellular Signaling Peptides and Proteins; Liver Neoplasms; Mice; Mice, Inbred C57BL; Mice, Mutant Strains; Proto-Oncogene Proteins c-akt; PTEN Phosphohydrolase; Pyridines; Signal Transduction; Stem Cells

2010
The regulation of non-coding RNA expression in the liver of mice fed DDC.
    Experimental and molecular pathology, 2009, Volume: 87, Issue:1

    Topics: Animals; Betaine; Biomarkers, Tumor; Diet; Dihydropyridines; Gene Expression Profiling; Gene Expression Regulation; Humans; In Situ Hybridization, Fluorescence; Liver; Liver Neoplasms; Male; Mice; Mice, Inbred C3H; Microarray Analysis; Proteins; RNA, Long Noncoding; RNA, Untranslated; Transcription Factors

2009