diallyl trisulfide has been researched along with Reperfusion Injury in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 3 (75.00) | 24.3611 |
2020's | 1 (25.00) | 2.80 |
Authors | Studies |
---|---|
Gao, B; Su, Y; Sun, H; Sun, Y; Xiang, L; Xu, D; Yang, W; Zhang, H; Zhong, Y; Zou, X | 1 |
Han, J; Li, S; Li, Z; Liu, Y; Tang, X; Wang, H; Xu, Y; Xue, X; Yang, Y; Yu, L; Zhang, J; Zhang, Y | 1 |
Bolevich, S; Bradic, JV; Jakovljevic, VL; Jeremic, JN; Jeremic, NS; Jovicic, NU; Mitrovic, SL; Nikolic Turnic, TR; Srejovic, IM; Tyagi, SC; Zivkovic, VI | 1 |
Guo, C; Liu, Y; Sun, X; Wang, C; Wang, W; Yang, C; Yang, W; Zhang, H | 1 |
4 other study(ies) available for diallyl trisulfide and Reperfusion Injury
Article | Year |
---|---|
Diallyl trisulfide improves spinal cord ischemia-reperfusion injury damage by activating AMPK to stabilize mitochondrial function.
Topics: AMP-Activated Protein Kinases; Animals; Antioxidants; Apoptosis; Mitochondria; Oxidative Stress; Rats; Reperfusion Injury; Spinal Cord | 2023 |
Diallyl trisulfide ameliorates myocardial ischemia-reperfusion injury by reducing oxidative stress and endoplasmic reticulum stress-mediated apoptosis in type 1 diabetic rats: role of SIRT1 activation.
Topics: Allyl Compounds; Animals; Antioxidants; Apoptosis; Caspase 3; Diabetes Mellitus, Experimental; Diabetes Mellitus, Type 1; Disease Models, Animal; Endoplasmic Reticulum Stress; Gene Expression Regulation; Heme Oxygenase-1; Humans; Myocardial Infarction; NF-E2-Related Factor 2; Oxidative Stress; Rats; Reperfusion Injury; Sirtuin 1; Sulfides | 2017 |
The cardioprotective effects of diallyl trisulfide on diabetic rats with ex vivo induced ischemia/reperfusion injury.
Topics: Allyl Compounds; Animals; Apoptosis; Biomarkers; Cardiotonic Agents; Diabetes Mellitus, Experimental; Male; Myocardium; Oxidative Stress; Rats, Wistar; Reperfusion Injury; Sulfides | 2019 |
Controlled release hydrogen sulfide delivery system based on mesoporous silica nanoparticles protects graft endothelium from ischemia-reperfusion injury.
Topics: Allyl Compounds; Animals; Apoptosis; Cell Differentiation; Cell Proliferation; Delayed-Action Preparations; Endothelium, Vascular; Flow Cytometry; Humans; Hydrogen Sulfide; In Situ Nick-End Labeling; Inflammation; Membrane Potential, Mitochondrial; Mice, Inbred BALB C; Nanomedicine; Nanoparticles; Porosity; Protective Agents; Reperfusion Injury; Silicon Dioxide; Sulfides | 2016 |