diallyl trisulfide has been researched along with Osteogenic Sarcoma in 5 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 1 (20.00) | 29.6817 |
| 2010's | 3 (60.00) | 24.3611 |
| 2020's | 1 (20.00) | 2.80 |
| Authors | Studies |
|---|---|
| Feng, S; Gong, F; He, P; Huang, Y; Sheng, B; Tang, L; Wang, Z; Xie, L; Xu, Y | 1 |
| Li, J; Li, Y; Si, M; Yin, H; Zhang, J; Zhang, L | 1 |
| Cui, J; Diao, Y; Li, J; Wang, Z; Xia, Q | 1 |
| Li, J; Li, K; Li, X; Sun, N; Tian, J; Wang, H | 1 |
| Diao, DM; Li, JM; Sun, DS; Yang, Q; Zhang, XH; Zhang, YK | 1 |
5 other study(ies) available for diallyl trisulfide and Osteogenic Sarcoma
| Article | Year |
|---|---|
Diallyl trisulfide regulates cell apoptosis and invasion in human osteosarcoma U2OS cells through regulating PI3K/AKT/GSK3β signaling pathway.
Topics: Allyl Compounds; Animals; Antineoplastic Agents; Apoptosis; Apoptosis Regulatory Proteins; Bone Neoplasms; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Movement; Cell Proliferation; Glycogen Synthase Kinase 3 beta; Humans; Male; Mice, Inbred BALB C; Mice, Nude; Neoplasm Invasiveness; Osteosarcoma; Phosphatidylinositol 3-Kinase; Proto-Oncogene Proteins c-akt; Signal Transduction; Sulfides; Tumor Burden; Xenograft Model Antitumor Assays | 2020 |
Diallyl trisulfide inhibits proliferation, invasion and angiogenesis of osteosarcoma cells by switching on suppressor microRNAs and inactivating of Notch-1 signaling.
Topics: Allyl Compounds; Antineoplastic Agents; Basic Helix-Loop-Helix Transcription Factors; Cell Movement; Cell Proliferation; Cell Survival; Collagen; Culture Media, Conditioned; Drug Combinations; Gene Expression Regulation, Neoplastic; Homeodomain Proteins; Human Umbilical Vein Endothelial Cells; Humans; Laminin; Matrix Metalloproteinase 2; MicroRNAs; Neoplasm Invasiveness; Neovascularization, Pathologic; Osteosarcoma; Proteoglycans; Receptor, Notch1; RNA, Small Interfering; Signal Transduction; Sulfides; Transcription Factor HES-1; Transfection; Vascular Endothelial Growth Factor A | 2013 |
Reversion of P-glycoprotein-mediated multidrug resistance by diallyl trisulfide in a human osteosarcoma cell line.
Topics: Allyl Compounds; Apoptosis; ATP Binding Cassette Transporter, Subfamily B, Member 1; Bone Neoplasms; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Drug Resistance, Multiple; Drug Resistance, Neoplasm; Gene Expression Regulation, Neoplastic; Humans; NF-kappa B; Osteosarcoma; Sulfides | 2014 |
Diallyl trisulfide induces osteosarcoma cell apoptosis through reactive oxygen species-mediated downregulation of the PI3K/Akt pathway.
Topics: Acetylcysteine; Allyl Compounds; Antineoplastic Agents; Antioxidants; Apoptosis; Bone Neoplasms; Cell Line, Tumor; Cell Proliferation; Cell Survival; Chromones; Cyclin D1; Cyclin-Dependent Kinase Inhibitor p21; Cyclin-Dependent Kinase Inhibitor p27; G1 Phase Cell Cycle Checkpoints; Humans; Membrane Potential, Mitochondrial; Mitochondria; Morpholines; Osteosarcoma; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Reactive Oxygen Species; Sulfides | 2016 |
A proteomic study on a human osteosarcoma cell line Saos-2 treated with diallyl trisulfide.
Topics: Allyl Compounds; Antineoplastic Agents, Phytogenic; Apoptosis; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Dose-Response Relationship, Drug; Down-Regulation; Electrophoresis, Gel, Two-Dimensional; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Humans; Image Processing, Computer-Assisted; Mass Spectrometry; Osteosarcoma; Proteins; Proteomics; Sulfides; Up-Regulation | 2009 |