Compounds > diallyl trisulfide
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diallyl trisulfide and Bone Neoplasms

diallyl trisulfide has been researched along with Bone Neoplasms in 3 studies

Research

Studies (3)

TimeframeStudies, this research(%)All Research%
pre-19900 (0.00)18.7374
1990's0 (0.00)18.2507
2000's0 (0.00)29.6817
2010's2 (66.67)24.3611
2020's1 (33.33)2.80

Authors

AuthorsStudies
Feng, S; Gong, F; He, P; Huang, Y; Sheng, B; Tang, L; Wang, Z; Xie, L; Xu, Y1
Cui, J; Diao, Y; Li, J; Wang, Z; Xia, Q1
Li, J; Li, K; Li, X; Sun, N; Tian, J; Wang, H1

Other Studies

3 other study(ies) available for diallyl trisulfide and Bone Neoplasms

ArticleYear
Diallyl trisulfide regulates cell apoptosis and invasion in human osteosarcoma U2OS cells through regulating PI3K/AKT/GSK3β signaling pathway.
    Histology and histopathology, 2020, Volume: 35, Issue:12

    Topics: Allyl Compounds; Animals; Antineoplastic Agents; Apoptosis; Apoptosis Regulatory Proteins; Bone Neoplasms; Cell Cycle Checkpoints; Cell Line, Tumor; Cell Movement; Cell Proliferation; Glycogen Synthase Kinase 3 beta; Humans; Male; Mice, Inbred BALB C; Mice, Nude; Neoplasm Invasiveness; Osteosarcoma; Phosphatidylinositol 3-Kinase; Proto-Oncogene Proteins c-akt; Signal Transduction; Sulfides; Tumor Burden; Xenograft Model Antitumor Assays

2020
Reversion of P-glycoprotein-mediated multidrug resistance by diallyl trisulfide in a human osteosarcoma cell line.
    Oncology reports, 2014, Volume: 31, Issue:6

    Topics: Allyl Compounds; Apoptosis; ATP Binding Cassette Transporter, Subfamily B, Member 1; Bone Neoplasms; Cell Cycle; Cell Line, Tumor; Cell Proliferation; Drug Resistance, Multiple; Drug Resistance, Neoplasm; Gene Expression Regulation, Neoplastic; Humans; NF-kappa B; Osteosarcoma; Sulfides

2014
Diallyl trisulfide induces osteosarcoma cell apoptosis through reactive oxygen species-mediated downregulation of the PI3K/Akt pathway.
    Oncology reports, 2016, Volume: 35, Issue:6

    Topics: Acetylcysteine; Allyl Compounds; Antineoplastic Agents; Antioxidants; Apoptosis; Bone Neoplasms; Cell Line, Tumor; Cell Proliferation; Cell Survival; Chromones; Cyclin D1; Cyclin-Dependent Kinase Inhibitor p21; Cyclin-Dependent Kinase Inhibitor p27; G1 Phase Cell Cycle Checkpoints; Humans; Membrane Potential, Mitochondrial; Mitochondria; Morpholines; Osteosarcoma; Phosphatidylinositol 3-Kinases; Phosphoinositide-3 Kinase Inhibitors; Proto-Oncogene Proteins c-akt; Reactive Oxygen Species; Sulfides

2016