diallyl trisulfide has been researched along with Angiogenesis, Pathologic in 6 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 0 (0.00) | 18.7374 |
| 1990's | 0 (0.00) | 18.2507 |
| 2000's | 2 (33.33) | 29.6817 |
| 2010's | 3 (50.00) | 24.3611 |
| 2020's | 1 (16.67) | 2.80 |
| Authors | Studies |
|---|---|
| Miao, Y; Peng, J; Wang, M; Wang, Z; Wei, H; Zhou, Y | 1 |
| Liu, N; Niu, L; Qin, J; Tao, Q; Wang, C; Wu, C; Xu, R; Zhang, P | 1 |
| Li, J; Li, Y; Si, M; Yin, H; Zhang, J; Zhang, L | 1 |
| Chung, JG; Hsu, SC; Lai, KC; Lein, JC; Yang, JS; Yu, CC | 1 |
| Chen, Q; Ganapathy, S; Shankar, S; Singh, KP; Srivastava, RK | 1 |
| Arlotti, JA; Bommareddy, A; Dhir, R; Hahm, ER; Marynowski, SW; Potter, DM; Powolny, AA; Singh, SV; Stan, SD; Warin, R; Xiao, D | 1 |
6 other study(ies) available for diallyl trisulfide and Angiogenesis, Pathologic
| Article | Year |
|---|---|
Diallyl Trisulfide Promotes Placental Angiogenesis by Regulating Lipid Metabolism and Alleviating Inflammatory Responses in Obese Pregnant Mice.
Topics: Allyl Compounds; Animals; Female; Humans; Inflammation; Lipid Metabolism; Mammals; Mice; Mice, Obese; Neovascularization, Pathologic; Obesity; Obesity, Maternal; Placenta; Placenta Diseases; Pregnancy; Sulfides | 2022 |
Diallyl trisulfide inhibits proliferation, invasion and angiogenesis of glioma cells by inactivating Wnt/β-catenin signaling.
Topics: Allyl Compounds; Animals; Antineoplastic Agents; Apoptosis; beta Catenin; Cell Line, Tumor; Cell Movement; Cell Proliferation; DNA-Binding Proteins; Gene Expression Regulation, Neoplastic; Glioma; Human Umbilical Vein Endothelial Cells; Humans; Intracellular Signaling Peptides and Proteins; Low Density Lipoprotein Receptor-Related Protein-6; Matrix Metalloproteinase 2; Matrix Metalloproteinase 9; Mice; Mice, Inbred BALB C; Mice, Nude; Neoplasm Invasiveness; Neovascularization, Pathologic; Sulfides; Transcription Factors; Wnt Proteins; Wnt Signaling Pathway | 2017 |
Diallyl trisulfide inhibits proliferation, invasion and angiogenesis of osteosarcoma cells by switching on suppressor microRNAs and inactivating of Notch-1 signaling.
Topics: Allyl Compounds; Antineoplastic Agents; Basic Helix-Loop-Helix Transcription Factors; Cell Movement; Cell Proliferation; Cell Survival; Collagen; Culture Media, Conditioned; Drug Combinations; Gene Expression Regulation, Neoplastic; Homeodomain Proteins; Human Umbilical Vein Endothelial Cells; Humans; Laminin; Matrix Metalloproteinase 2; MicroRNAs; Neoplasm Invasiveness; Neovascularization, Pathologic; Osteosarcoma; Proteoglycans; Receptor, Notch1; RNA, Small Interfering; Signal Transduction; Sulfides; Transcription Factor HES-1; Transfection; Vascular Endothelial Growth Factor A | 2013 |
Diallyl trisulfide inhibits migration, invasion and angiogenesis of human colon cancer HT-29 cells and umbilical vein endothelial cells, and suppresses murine xenograft tumour growth.
Topics: Allyl Compounds; Angiogenesis Inhibitors; Animals; Cell Line; Cell Line, Tumor; Cell Movement; Cell Proliferation; HT29 Cells; Human Umbilical Vein Endothelial Cells; Humans; Mice; Neoplasm Invasiveness; Neovascularization, Pathologic; Sulfides; Xenograft Model Antitumor Assays | 2015 |
Diallyl trisulfide increases the effectiveness of TRAIL and inhibits prostate cancer growth in an orthotopic model: molecular mechanisms.
Topics: Allyl Compounds; Animals; Blotting, Western; Cell Division; Humans; Immunohistochemistry; Male; Mice; Mice, Nude; Neoplasm Metastasis; Neoplasm Transplantation; Neovascularization, Pathologic; Prostatic Neoplasms; Sulfides; TNF-Related Apoptosis-Inducing Ligand | 2008 |
Garlic constituent diallyl trisulfide prevents development of poorly differentiated prostate cancer and pulmonary metastasis multiplicity in TRAMP mice.
Topics: Adenocarcinoma; Allyl Compounds; Animals; Antigens, Viral, Tumor; Apoptosis; Cadherins; Cell Proliferation; Lung Neoplasms; Male; Mice; Mice, Inbred C57BL; Mice, Transgenic; Neovascularization, Pathologic; Proliferating Cell Nuclear Antigen; Prostatic Neoplasms; Sulfides; Synaptophysin | 2008 |