diallyl disulfide has been researched along with Stomach Neoplasms in 16 studies
| Timeframe | Studies, this research(%) | All Research% |
|---|---|---|
| pre-1990 | 1 (6.25) | 18.7374 |
| 1990's | 2 (12.50) | 18.2507 |
| 2000's | 5 (31.25) | 29.6817 |
| 2010's | 8 (50.00) | 24.3611 |
| 2020's | 0 (0.00) | 2.80 |
| Authors | Studies |
|---|---|
| Ai, XH; Ding, E; Jiang, H; Ling, H; Liu, F; Su, B; Su, J; Su, Q; Tan, T; Wu, YH; Xia, H; Zeng, X; Zeng, Y | 1 |
| Guo, J; Kong, Y; Su, Q; Tang, H; Tang, Y; Xie, X; Yang, L | 1 |
| Ai, XH; Bo, S; Dai, WX; Hao, J; Hong, X; Hui, H; Hui, L; Li, W; Lin, D; Qi, S; Wu, YH | 1 |
| He, J; Jiang, H; Ling, H; Lu, LF; Su, Q; Xiao, GH | 1 |
| Ai, X; Dai, W; He, H; Jiang, H; Ling, H; Su, B; Su, J; Su, Q; Wu, Y; Xia, H; Zeng, X | 1 |
| Ai, XH; Jiang, H; Ling, H; Liu, F; Ma, YH; Su, B; Su, J; Su, Q; Wu, YH; Xia, H; Yang, BM; Zeng, X; Zeng, Y; Zhang, S; Zhou, ZG | 1 |
| He, J; Ji, XX; Ling, H; Su, Q; Tan, H; Tang, YL; Wen, L; Xia, H; Zhou, JG | 1 |
| Choi, IW; Choi, YH; Choi, YW; Hwang, HJ; Kim, GY; Kim, ND; Park, HS | 1 |
| Ling, H; Liu, Y; Song, Y; Su, Q; Yuan, JP; Zhang, MX | 1 |
| Huang, C; Ling, H; Liu, Y; Song, Y; Su, Q; Tang, RJ; Wang, GH; Yang, YH; Yuan, JP | 1 |
| Dong, L; Liao, QJ; Ling, H; Su, Q; Xiang, SL; Xiao, XL; Zhou, XT | 1 |
| Aquilano, K; Ciriolo, MR; Filomeni, G; Rotilio, G | 1 |
| He, J; Ling, H; Luo, ZY; Song, Y; Su, Q; Tan, H; Yuan, JP; Zeng, X; Zhang, LY; Zhou, XT | 1 |
| Agarwal, R; Chatterjee, ML; Hu, X; Singh, SV; Srivastava, SK; Xia, H; Zaren, HA | 1 |
| Awasthi, S; Awasthi, YC; Benson, PJ; Bleicher, RJ; Hu, X; Singh, SV; Srivastava, SK; Xia, H; Zaren, HA | 1 |
| Barany, G; Sparnins, VL; Wattenberg, LW | 1 |
16 other study(ies) available for diallyl disulfide and Stomach Neoplasms
| Article | Year |
|---|---|
Diallyl disulfide inhibits TGF‑β1‑induced upregulation of Rac1 and β‑catenin in epithelial‑mesenchymal transition and tumor growth of gastric cancer.
Topics: Allyl Compounds; Animals; Antineoplastic Agents; beta Catenin; Cell Line, Tumor; Cell Proliferation; Disulfides; Epithelial-Mesenchymal Transition; Gene Expression Regulation, Neoplastic; Humans; Mice; Neoplasm Invasiveness; rac1 GTP-Binding Protein; Stomach Neoplasms; Transforming Growth Factor beta1; Up-Regulation; Xenograft Model Antitumor Assays | 2018 |
Diallyl disulfide suppresses proliferation and induces apoptosis in human gastric cancer through Wnt-1 signaling pathway by up-regulation of miR-200b and miR-22.
Topics: Adenocarcinoma; Allyl Compounds; Animals; Antineoplastic Agents; Apoptosis; Base Sequence; Cell Line, Tumor; Cell Proliferation; Combined Modality Therapy; Disulfides; Gene Expression Regulation, Neoplastic; Genetic Therapy; Humans; Male; Mice; Mice, Inbred BALB C; Mice, Nude; MicroRNAs; RNA Interference; Stomach Neoplasms; Transcriptome; Tumor Burden; Up-Regulation; Wnt Signaling Pathway; Wnt1 Protein; Xenograft Model Antitumor Assays | 2013 |
Chk1, but not Chk2, is responsible for G2/M phase arrest induced by diallyl disulfide in human gastric cancer BGC823 cells.
Topics: Allyl Compounds; cdc25 Phosphatases; Cell Line, Tumor; Checkpoint Kinase 1; Cyclin B1; Disulfides; Down-Regulation; G2 Phase Cell Cycle Checkpoints; Humans; M Phase Cell Cycle Checkpoints; Phosphorylation; Protein Kinases; RNA, Small Interfering; Signal Transduction; Stomach Neoplasms | 2014 |
Diallyl disulfide selectively causes checkpoint kinase-1 mediated G2/M arrest in human MGC803 gastric cancer cell line.
Topics: Allyl Compounds; Antineoplastic Agents; Cell Line, Tumor; Checkpoint Kinase 1; Checkpoint Kinase 2; Disulfides; G2 Phase Cell Cycle Checkpoints; Gene Expression Regulation, Neoplastic; Gene Knockdown Techniques; Humans; Phosphorylation; Protein Kinases; Stomach Neoplasms | 2014 |
Identification of potential targets for diallyl disulfide in human gastric cancer MGC-803 cells using proteomics approaches.
Topics: Allyl Compounds; Cadherins; Cell Line, Tumor; Cyclin-Dependent Kinases; Disulfides; Down-Regulation; Humans; MAP Kinase Signaling System; Matrix Metalloproteinase 9; Proteome; Proteomics; Proto-Oncogene Proteins c-fos; Receptors, Urokinase Plasminogen Activator; Signal Transduction; Stomach Neoplasms; Tissue Inhibitor of Metalloproteinase-3; Up-Regulation; Urokinase-Type Plasminogen Activator; Vimentin | 2015 |
Diallyl disulfide suppresses epithelial-mesenchymal transition, invasion and proliferation by downregulation of LIMK1 in gastric cancer.
Topics: Allyl Compounds; Animals; Antigens, CD34; Cadherins; Cell Line, Tumor; Cell Movement; Cell Proliferation; Cofilin 1; Disulfides; Down-Regulation; Epithelial-Mesenchymal Transition; Humans; Ki-67 Antigen; Lim Kinases; Lymphatic Metastasis; Male; Matrix Metalloproteinase 9; Mice; Mice, Inbred BALB C; Mice, Nude; Neoplasm Invasiveness; Neoplasm Transplantation; p21-Activated Kinases; rac1 GTP-Binding Protein; rho-Associated Kinases; Stomach Neoplasms; Tissue Inhibitor of Metalloproteinase-3; Transplantation, Heterologous; Vimentin | 2016 |
Growth inhibitory effect and Chk1-dependent signaling involved in G2/M arrest on human gastric cancer cells induced by diallyl disulfide.
Topics: Allyl Compounds; Antineoplastic Agents; Cell Division; Cell Line, Tumor; Checkpoint Kinase 1; Disulfides; G2 Phase; Growth Inhibitors; Humans; Protein Kinases; Signal Transduction; Stomach Neoplasms | 2010 |
Inhibition of matrix metalloproteinase activities and tightening of tight junctions by diallyl disulfide in AGS human gastric carcinoma cells.
Topics: Allyl Compounds; Cell Line, Tumor; Cell Movement; Claudin-3; Claudin-4; Claudins; Disulfides; Dose-Response Relationship, Drug; Garlic; Gene Expression Regulation; Humans; Matrix Metalloproteinase 2; Matrix Metalloproteinase 9; Matrix Metalloproteinase Inhibitors; Plant Extracts; Reverse Transcriptase Polymerase Chain Reaction; RNA, Messenger; Sequence Analysis, RNA; Stomach Neoplasms; Tight Junctions; Tissue Inhibitor of Metalloproteinase-1; Tissue Inhibitor of Metalloproteinase-2 | 2011 |
[Diallyl disulfide-induced G2/M arrest of human gastric cancer MGC803 cells involves activation of p38 MAP kinase pathways].
Topics: Allyl Compounds; Blotting, Western; cdc25 Phosphatases; Cell Cycle Proteins; Cell Division; Cell Line, Tumor; Disulfides; Enzyme Activation; Flow Cytometry; G2 Phase; Humans; Mitogen-Activated Protein Kinases; Mitosis; p38 Mitogen-Activated Protein Kinases; Stomach Neoplasms | 2004 |
Diallyl disulfide-induced G2/M arrest of human gastric cancer MGC803 cells involves activation of p38 MAP kinase pathways.
Topics: Allyl Compounds; Antineoplastic Agents; cdc25 Phosphatases; Cell Cycle Proteins; Cell Division; Cell Line, Tumor; Disulfides; G2 Phase; Humans; MAP Kinase Signaling System; p38 Mitogen-Activated Protein Kinases; Phosphorylation; Stomach Neoplasms | 2004 |
[Antitumor effect of diallyl disulfide on human gastric cancer MGC803 cells xenograft in nude mice].
Topics: Allyl Compounds; Animals; Antineoplastic Agents; Cell Line, Tumor; Disulfides; Humans; Injections, Intraperitoneal; Male; Mice; Mice, Inbred BALB C; Mice, Nude; Neoplasm Transplantation; Proliferating Cell Nuclear Antigen; Stomach Neoplasms | 2005 |
Glutathione-related systems and modulation of extracellular signal-regulated kinases are involved in the resistance of AGS adenocarcinoma gastric cells to diallyl disulfide-induced apoptosis.
Topics: Adenocarcinoma; Allyl Compounds; Antineoplastic Agents; Apoptosis; Cell Division; Cyclin-Dependent Kinase Inhibitor p21; Disulfides; Drug Resistance, Neoplasm; G2 Phase; Glutathione; Glutathione Peroxidase; Humans; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Reactive Oxygen Species; Stomach Neoplasms; Sulfhydryl Compounds; Tumor Cells, Cultured; Tumor Suppressor Protein p53 | 2005 |
Erk is involved in the differentiation induced by diallyl disulfide in the human gastric cancer cell line MGC803.
Topics: Agglutination; Alkaline Phosphatase; Allyl Compounds; Butadienes; Cell Differentiation; Cell Line, Tumor; Cell Survival; Concanavalin A; Disulfides; Humans; Mitogen-Activated Protein Kinase 1; Mitogen-Activated Protein Kinase 3; Nitriles; Phosphoproteins; Phosphorylation; Stomach Neoplasms | 2006 |
Mechanism of differential efficacy of garlic organosulfides in preventing benzo(a)pyrene-induced cancer in mice.
Topics: Allyl Compounds; Animals; Anticarcinogenic Agents; Benzo(a)pyrene; Carcinogens; Cytochrome P-450 CYP1A1; Disulfides; Enzyme Activation; Enzyme Induction; Epoxide Hydrolases; Female; Garlic; Glutathione Transferase; Liver; Liver Neoplasms; Mice; Mice, Inbred A; Microsomes; Microsomes, Liver; Plants, Medicinal; Propane; Stomach; Stomach Neoplasms; Sulfides | 1997 |
Induction of glutathione S-transferase pi as a bioassay for the evaluation of potency of inhibitors of benzo(a)pyrene-induced cancer in a murine model.
Topics: Allyl Compounds; Animals; Anticarcinogenic Agents; Benzo(a)pyrene; Biological Assay; Chromatography, High Pressure Liquid; Disulfides; Enzyme Induction; Female; Garlic; Glutathione S-Transferase pi; Glutathione Transferase; Isoenzymes; Liver; Mice; Mice, Inbred A; Plants, Medicinal; Propane; Regression Analysis; Stomach; Stomach Neoplasms; Sulfides; Treatment Outcome | 1997 |
Inhibition of N-nitrosodiethylamine carcinogenesis in mice by naturally occurring organosulfur compounds and monoterpenes.
Topics: Allium; Allyl Compounds; Animals; Cyclohexane Monoterpenes; Cyclohexenes; Diethylnitrosamine; Disulfides; Female; Limonene; Mice; Monoterpenes; Stomach Neoplasms; Sulfides; Terpenes | 1989 |