diallyl disulfide has been researched along with Neoplasm Metastasis in 4 studies
Timeframe | Studies, this research(%) | All Research% |
---|---|---|
pre-1990 | 0 (0.00) | 18.7374 |
1990's | 0 (0.00) | 18.2507 |
2000's | 0 (0.00) | 29.6817 |
2010's | 4 (100.00) | 24.3611 |
2020's | 0 (0.00) | 2.80 |
Authors | Studies |
---|---|
Chen, W; Liu, Y; Liu, Z; Lu, Y; Shan, Y; Tao, L; Wang, A; Wei, Z; Wu, Y; Zhu, Z | 1 |
Feng, C; Gao, X; Han, L; Jia, L; Jiao, Y; Jing, F; Ma, Y; Wang, J; Yin, X; Zhang, J; Zhang, Y | 1 |
Chen, B; Liu, P; Liu, X; Tang, H; Xiao, X; Xie, X; Ye, F; Zheng, G | 1 |
Chen, XX; Chen, XY; Li, LD; Liu, XW; Peng, L; Tu, J; Xiong, T; Zhou, ZG | 1 |
4 other study(ies) available for diallyl disulfide and Neoplasm Metastasis
Article | Year |
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Diallyl trisulfides, a natural histone deacetylase inhibitor, attenuate HIF-1α synthesis, and decreases breast cancer metastasis.
Topics: Allyl Compounds; Animals; Breast Neoplasms; Cell Adhesion; Cell Line, Tumor; Disulfides; Female; Gene Expression Regulation, Neoplastic; Gene Regulatory Networks; Histone Deacetylase Inhibitors; Humans; Hypoxia-Inducible Factor 1, alpha Subunit; Mice; Neoplasm Metastasis; Sulfides; Xenograft Model Antitumor Assays | 2017 |
Diallyl disulfide inhibits the metastasis of type Ⅱ esophageal‑gastric junction adenocarcinoma cells via NF-κB and PI3K/AKT signaling pathways in vitro.
Topics: Adenocarcinoma; Allyl Compounds; Anticarcinogenic Agents; Cell Line, Tumor; Cell Movement; Cell Survival; Disulfides; Esophageal Neoplasms; Esophagogastric Junction; Gene Expression Regulation, Neoplastic; Humans; In Vitro Techniques; Matrix Metalloproteinases; Neoplasm Metastasis; NF-kappa B; Phosphatidylinositol 3-Kinases; Proto-Oncogene Proteins c-akt; Signal Transduction; Tissue Inhibitor of Metalloproteinases | 2018 |
Diallyl disulfide suppresses SRC/Ras/ERK signaling-mediated proliferation and metastasis in human breast cancer by up-regulating miR-34a.
Topics: Allyl Compounds; Analysis of Variance; Animals; Blotting, Western; Breast Neoplasms; Cell Line, Tumor; Cell Movement; Cell Proliferation; Disulfides; Female; Gene Expression Regulation; Humans; Immunohistochemistry; Luciferases; Mice; MicroRNAs; Neoplasm Metastasis; Plasmids; RNA Interference; Signal Transduction; Sulfides | 2014 |
[Diallyl disulfide inhibits invasion and metastasis of MCF-7 breast cancer cells in vitro by down-regulating p38 activity].
Topics: Allyl Compounds; Antigens, CD; Breast Neoplasms; Cadherins; Disulfides; Gene Expression Regulation, Neoplastic; Humans; MAP Kinase Signaling System; Matrix Metalloproteinase 9; MCF-7 Cells; Mitogen-Activated Protein Kinase 11; Neoplasm Invasiveness; Neoplasm Metastasis; Transforming Growth Factor beta1; Vimentin | 2016 |